SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

The Bond: Our Kinship with Animals, Our Call to Defend Them

2011-04-04T14:06:00.000-07:00

Book sheds new light on Chino beef recall

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07:39 AM PDT on Monday, April 4, 2011

By BEN GOAD Washington Bureau

Special Section: Chino Beef Recall

WASHINGTON - The undercover investigator whose video footage at a Chino slaughterhouse led to the largest beef recall in U.S. history was actually planning to infiltrate an Inland egg production facility but couldn't get hired anywhere.

It was late 2007, and the Humane Society of the United States had hoped to capture images of chickens in cages and the general treatment of animals in industrial agriculture settings. The mission came in advance of the ultimately successful campaign for Prop. 2, a 2008 California ballot initiative to ban the extreme confinement of animals.

Unable to find work at several egg facilities, the investigator instead took work at Westland/Hallmark Meat Co. in Chino.

Wayne Pacelle "It was purely opportunistic and not based on knowledge of questionable practices at the plant," Humane Society President Wayne Pacelle writes in a forthcoming book. "In fact, we'd never even heard of the place before."

Pacelle devotes a large section of his book to the group's investigation at Westland/Hallmark, which yielded footage of workers shocking, beating and dragging cows unable to stand for slaughter -- despite the presence of federal food inspectors and an Agriculture Department veterinarian.

The video provided evidence that downer cows were slaughtered, a violation of the plant's government contract that triggered the recall of 143 million pounds of beef and the closure of what was once the second-largest supplier of beef to the National School Lunch Program.

The investigator, whom Pacelle has never publicly named, concealed a tiny camera underneath his clothes. The camera's battery life was limited, allowing the investigator to record only an hour of film each day, according to Pacelle's account.

"(Co-workers) had no idea the he was a vegan and that the 'meat' in his sandwich was actually a soy riblet he'd bring to work every day," Pacelle writes.

In the book, Pacelle responds to criticism over the timing of the video's release. In the midst of the nationwide recall, lawmakers and officials at the USDA questioned why the Humane Society didn't immediately bring the information to federal authorities.

"It did raise a lot of concern," American Meat Institute spokeswoman Janet Riley recalled. "Industry was not happy about the delay."

While the tapes were compiled in late fall 2007, the Humane Society kept them under wraps until mid-December of that year, when they gave them to the San Bernardino County district attorney's office. They were given to the Agriculture Department on Jan. 30, 2008, about the same time as they posted the footage online.

"Everything had happened under USDA's regulatory authority," Pacelle writes. "I thought it highly unlikely that the department would condemn its own employees' performance and highlight the poor oversight procedures at the plant."

Ultimately, the federal government joined a $150 million Humane Society lawsuit against owners of the now-defunct plant.

The case is slated for trial in March 2013.

Pacelle also credited the investigation with helping to pass Prop. 2.

Though the initiative had nothing to do with slaughter practices, it helped put a spotlight on animal treatment in agricultural plants, he said. He recalled watching the 2008 election results from the same Los Angeles hotel where, coincidentally, he writes, the California Democratic Party was celebrating the victory of President Barack Obama.

"The first county returns I looked for were those of the Inland Empire," he writes. "In the end, Prop. 2 got 62.5 percent of the vote in Riverside and 62 percent in San Bernardino. They proved to be the bellwethers we thought they'd be."

Pacelle's book is titled "The Bond: Our Kinship with Animals, Our Call to Defend Them." It is slated to hit bookstores Tuesday. Comment on this story

http://www.pe.com/localnews/stories/PE_News_Local_D_hallmark04.256c807.html

Greetings,

Considering what the NSLP and the USDA have done in the past, I would welcome any new standard. So I applaud these new standards.

However, feeding those dead stock downer cows to children for over 4 years was something they should never ever be able to forget about. dead stock downer cows are the most high risk animal for mad cow disease, and they knew this when the feeding to our children of this product was taking place. the USDA et al via the NSLP fed our children all across the USA, from state to state, the largest beef recall in history, they fed our children DEAD STOCK DOWNER COWS, the most HIGH RISK CATTLE FOR MAD COW DISEASE, they fed them to our children, and hid the largest beef recall in USA history, hid this recall under the guise of 'animal abuse'. then told the parents not to worry because none of the kids had been sick or died from it to date. CJD can incubate for decades. anyway, just wanted to tell you about this, and thank you for what you are trying to do...

WHO WILL WATCH THE CHILDREN FOR CJD OVER THE NEXT 5 + DECADES ???

Do you actually believe that the USDA et al jumped in on the law suit against Westland/Hallmark, at the time the largest beef recall in USA history, just because a few animals were abused on a video, or to cover their ass, for letting our children, from school district to school district, from state to state, be fed dead stock downer cows.

" In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks "

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

Do you actually believe all these schools recalled this meat because of a few cattle being abused, see list ; FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114

IF url does not work above, go to this link to find out if any of your children and their school were part of this recall ; go to this site ;

http://www.fns.usda.gov/fns/ left hand corner search ;

Hallmark/Westland Meat Packing Co. Beef Recall your should get this ;

http://65.216.150.153/texis/search?pr=FNS

1 through 1 of 1 matching documents, best matches first. sort by date 1: Hallmark - Westland SFA Reporting by State - 3-24-2008.xls Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 The U.S. Department of Agriculture ...

http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114

PLEASE SEE ALSO ;

Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

PLEASE NOTE *

Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME.

snip...

The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

PLEASE be aware, for 4 years, the USDA fed our children all across the Nation dead stock downer cows, the most high risk cattle for BSE aka mad cow disease and other dangerous pathogens. who will watch our children for CJD for the next 5+ decades ???

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

page 114 ;

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf

International Society for Infectious Diseases Web: http://www.isid.org/

please see full text ;

http://transmissiblespongiformencephalopathy.blogspot.com/

To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE. When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.

http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2

Thursday, August 12, 2010

Seven main threats for the future linked to prions

First threat

The TSE road map defining the evolution of European policy for protection against prion diseases is based on a certain numbers of hypotheses some of which may turn out to be erroneous. In particular, a form of BSE (called atypical Bovine Spongiform Encephalopathy), recently identified by systematic testing in aged cattle without clinical signs, may be the origin of classical BSE and thus potentially constitute a reservoir, which may be impossible to eradicate if a sporadic origin is confirmed. ***Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.

Second threat

snip...

http://www.neuroprion.org/en/np-neuroprion.html

http://prionpathy.blogspot.com/2010/08/seven-main-threats-for-future-linked-to.html

http://prionpathy.blogspot.com/

http://prionopathy.blogspot.com/

Saturday, January 29, 2011

Atypical L-Type Bovine Spongiform Encephalopathy (L-BSE) Transmission to Cynomolgus Macaques, a Non-Human Primate

Jpn. J. Infect. Dis., 64 (1), 81-84, 2011

http://transmissiblespongiformencephalopathy.blogspot.com/2011/01/atypical-l-type-bovine-spongiform.html

Wednesday, March 9, 2011

27 U.S. Senators want to force feed Japan Highly Potential North America Mad Cow Beef TSE PRION CJD

March 8, 2011

President Barack Obama The White House

1600 Pennsylvania Avenue, W Washington, DC 20500

Dear President Obama:

http://transmissiblespongiformencephalopathy.blogspot.com/2011/03/27-us-senators-want-to-force-feed-japan.html

Saturday, March 5, 2011

MAD COW ATYPICAL CJD PRION TSE CASES WITH CLASSIFICATIONS PENDING ON THE RISE IN NORTH AMERICA

http://transmissiblespongiformencephalopathy.blogspot.com/2011/03/mad-cow-atypical-cjd-prion-tse-cases.html

Based on a $35 million investment by the Government of Alberta, the Alberta Prion Research Institute (APRI) supports fundamental, applied and multidisciplinary research geared toward providing solutions and models of policy action that can meet the BSE challenge for the beef and food industries, and similar challenges from other prion diseases as well as other diseases in animals and humans related to protein misfolding. APRI supports outcomesoriented research on prevention, mitigation and treatment of these diseases, and is developing the scientific evidence to inform public policy on solutions and best practices. The initiative supports projects that: . Focus on excellent research and innovation . Develop world-class capacity . Help the Alberta livestock industries . May eventually translate into products, services, and public policies . May help Alberta companies develop, for example, new handling practices, surveillance procedures, diagnostic tests, decontamination technologies . Can be applied beyond Alberta's borders-nationally and globally FOR MORE INFORMATION, PLEASE VISIT www.prioninstitute.ca

http://www.prion2011.ca/files/PRION2011_Prospectus_ENG.pdf

COMPARE TO USA PRION FUNDING 2011

"which includes the ___elimination___ of Prion activities ($5,473,000),"

All Other Emerging and Zoonotic Infectious Diseases CDC's FY 2012 request of $52,658,000 for all other emerging and zoonotic infectious disease activities is a decrease of $13,607,000 below the FY 2010 level, which includes the elimination of Prion activities ($5,473,000), a reduction for other cross-cutting infectious disease activities, and administrative savings. These funds support a range of critical emerging and zoonotic infectious disease programs such Lyme Disease, Chronic Fatigue Syndrome, and Special Pathogens, as well as other activities described below.

http://www.cdc.gov/fmo/topic/Budget%20Information/appropriations_budget_form_pdf/FY2012_CDC_CJ_Final.pdf

PRION MAD COW UDPATE NORTH AMERICA 2011

Sunday, April 3, 2011

PRION 2011 NEWWORLD MONTREAL CANADA MAY 16 - 19

http://transmissiblespongiformencephalopathy.blogspot.com/2011/04/prion-2011-newworld-montreal-canada-may.html

Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518

[Docket No. FSIS-2010-0041] Non-Ambulatory Disabled Veal Calves and Other Non-Ambulatory Disabled Livestock at Slaughter; Petitions for Rulemaking

2011-02-09T15:03:00.000-08:00

Subject: [Docket No. FSIS-2010-0041] Non-Ambulatory Disabled Veal Calves and Other Non-Ambulatory Disabled Livestock at Slaughter; Petitions for Rulemaking

Terry S. Singeltary Sr. comment submission on ;

DEPARTMENT OF AGRICULTURE

Food Safety and Inspection Service

9 CFR Part 309

[Docket No. FSIS–2010–0041]

Non-Ambulatory Disabled Veal Calves

Livestock at Slaughter; Petitions for

Rulemaking

AGENCY: Food Safety and Inspection

Service, USDA.

ACTION: Petitions for rulemaking.

SUMMARY: The Food Safety and

Inspection Service (FSIS) is requesting

comments on two petitions for

rulemaking submitted to the Agency

that raise issues associated with the

disposition of non-ambulatory disabled

veal calves and other non-ambulatory

disabled livestock at slaughter. The first

petition, submitted by the Humane

Society of the United States (HSUS),

requests that FSIS repeal a provision in

its ante-mortem inspection regulations

that permits veal calves that are unable

to rise from a recumbent position and

walk because they are tired or cold to

be set apart and held for treatment. Such

calves are permitted to proceed to

slaughter if they are able to rise and

walk after being warmed or rested. The

HSUS has petitioned FSIS to amend the

regulations to require that nonambulatory

disabled veal calves be

condemned and promptly and

humanely euthanized. The second

petition, submitted by Farm Sanctuary,

requests that the Agency amend the

Federal meat inspection regulations to

prohibit the slaughter of nonambulatory

disabled pigs, sheep, goats,

and other amenable livestock. In

addition to requesting comments on the

petitions, the Agency is clarifying its

requirements for condemned nonambulatory

disabled cattle at official

slaughter establishments.

DATES: Comments must be received by

April 8, 2011.

ADDRESSES: FSIS invites interested

persons to submit relevant comments on

the implementation of this proposed

rule. Comments may be submitted by

either of the following methods:

• Federal eRulemaking Portal: This

Web site provides the ability to type

short comments directly into the

comment field on this Web page or

attach a file for lengthier comments. Go

to http://www.regulations.gov. Follow

the online instructions at that site for

submitting comments.

• Mail, including floppy disks or CD–

ROMs, and hand- or courier-delivered

items: Send to Docket Clerk, U.S.

Department of Agriculture (USDA),

FSIS, Room 2–2127, George Washington

Carver Center, 5601 Sunnyside Avenue,

Beltsville, MD 20705–5272.

Instructions: All items submitted by

mail or electronic mail must include the

Agency name and docket number FSIS–

2010–0041. Comments received in

response to this docket will be made

available for public inspection and

posted without change, including any

personal information, to http://www.regulations.gov.

Docket: For access to background

documents or comments received, go to

the FSIS Docket Room at the address

listed above between 8 a.m. and 4:30

p.m., Monday through Friday.

FOR FURTHER INFORMATION CONTACT: Dr.

Daniel Engeljohn, Assistant

Administrator, Office of Policy and

Program Development, FSIS, U.S.

Department of Agriculture, 1400

Independence Avenue, SW.,

Washington, DC 20250–3700, (202) 720–

2709.

SUPPLEMENTARY INFORMATION:

Background

Regulatory Requirements for Non-

Ambulatory Disabled Cattle

snip...

http://edocket.access.gpo.gov/2011/pdf/2011-2504.pdf

I believe for the following reasons, that ALL downer non-ambulatory disabled livestock at slaughter should all be condemned. A diseased and or disabled animal (possibly disabled due to disease, and if no test is done, the risk factor is real) should never, ever, regardless of age, should be allowed into the food and or feed chain. They should be incinerated. ...

kind regards,

Terry S. Singeltary Sr.

CONSUMPTION OF VEAL AND VENISON AND SIGNIFICANT INCREASE CJD

Sat Apr 28, 2007 07:10 68.238.100.254

greetings,

i have been working on something for a while 'the big lie', the following data to be added in, but i thought due to what i think the importance of this is, i thought i would go ahead and put this out now for those that might be interested. ........tss

CHANGING SCIENCE TO FIT YOUR INDUSTRY NEEDS COVER-UP IN FULL MODE NOW

POLICY - RESTRICTED

CREUTZFELDT-JAKOB DISEASE: 3RD ANNUAL REPORT OF THE UK SURVEILLANCE UNIT

1. This submission, which has been agreed with colleagues in HEF(M). alerts PS(L) to the contents of the forthcoming annual report of the CJD Surveillance Unit and presents options for publication. It also highlights concern over the presentation of results which could be misrepresented by the media and others as evidence of a lilnk between CJD and the consumption of veal. ...

RECOMMENDATION

2. PS(L) is invited to agree the recommendation at para 13.

PROBLEM

7. The main findings in the case-control study were STATISTICALLY SIGNIFICANT ASSOCIATIONS BETWEEN CONSUMPTION OF VEAL OR VENISON AND THE DEVELOPMENT OF CJD (INCREASED RISKS OF 2-13x). There was also evidence of a dose-response relationship between dietary exposure and development of the disease. (Last year's findings showed an apparent association between eating black pudding and risk of CJD which was neither statistically significant nor biologically plausible - interestingly, this has not been (replicated was marked out with something i cannot read), and then this complete sentence was marked through to be replaced ;

THIS YEAR'S FINDINGS SHOW A NUMBER OF ASSOCIATIONS BUT THE STRONGEST IS FOR VEAL.

IP PS(L) wishes to probe this further we think it best to explain the matter VERBALLY. The problem is how to present the findings in this year's annual report in a way which avoids unnecessary public alarm and limits the scope for media scare stores. (or the facts...TSS)

This is of considerable concern given recent development. In particular Ministers will be particularly concerned about the European dimension given the recent troubles with the Germans.

9. DH doctors advise - and we understand Dr Wills agrees - that the association the study found between the developments of CJD and veal consumption cannot be regarded as demonstrating a causal relationship or give any reason to change the advice that eating beef and veal is safe. IF PS(L) wishes to probe this further we think it best to explain the matter verbally. The problem is how to present the findings in this year's annual report in a way which avoids unnecessary public alarm and limits the scope for media scare stories.

Next steps ...

snip... full text ;

http://collections.europarchive.org/tna/20080103020408/http://www.bseinquiry.gov.uk/files/yb/1994/07/00001001.pdf

i can't believe some of these same ............people are still allowed to be in the decission making positions they are in. ........OH WAIT DUMMY, YES YOU CAN, it's called $$$

BRITISH DEER FARMERS ASSOCIATION

OCTOBER 1994

Dear Mr Elmhirst,

CREUTZFELDT-JAKOB DISEASE (CJD) SURVEILLANCE UNIT REPORT

Thank you for your recent letter concerning the publication of the third annual report from the CJD Surveillance Unit. I am sorry that you are dissatisfied with the way in which this report was published.

The Surveillance Unit is completely independent outside body and the Department of Health is committed to publishing their reports as soon as they become available. In the circumstances it is not the practice to circulate the report for comment since the findings of the report would not be amended. In future we can ensure that the British Deer Farmers Association receives a copy of the report in advance of publication.

snip...

The statistical results regarding the consumption of venison was put into perspective in the body of the report and was NOT MENTIONED AT ALL IN THE PRESS RELEASE. Media attention regarding this report was low key but gave a realistic presentation of the statistical findings of the Unit. This approach to publication was successful in that consumption of VENISON was highlighted only by the media i.e. in the News at one television programme.

I believe that a further statement about the report, or indeed statistical links between CJD and consumption of Venison, would increase, and quite possibly GIVE DAMAGING CREDENCE, to the whole issue. From the low key media reports of which I am aware it seems unlikely that venison consumption will suffer adversely, if at all.

http://web.archive.org/web/20030511010117/http://www.bseinquiry.gov.uk/files/yb/1994/10/00003001.pdf

see buttered and watered down report here that caters to industry instead of human safety. this is what happen when you have industry run the government.
see where this ;

PROBLEM

7. The main findings in the case-control study were STATISTICALLY SIGNIFICANT ASSOCIATIONS BETWEEN CONSUMPTION OF VEAL OR VENISON AND THE DEVELOPMENT OF CJD (INCREASED RISKS OF 2-13x). There was also evidence of a dose-response relationship between dietary exposure and development of the disease. (Last year's findings showed an apparent association between eating black pudding and risk of CJD which was neither statistically significant nor biologically plausible - interestingly, this has not been (replicated was marked out with something i cannot read), and then this complete sentence was marked through to be replaced ;

THIS YEAR'S FINDINGS SHOW A NUMBER OF ASSOCIATIONS BUT THE STRONGEST IS FOR VEAL. ...snip..end

went to this ;

CJD9/10022

October 1994

Mr R.N. Elmhirst
Chairman
British Deer Farmers Association
Holly Lodge
Spencers Lane
BerksWell
Coventry
CV7 7BZ

Dear Mr Elmhirst,

CREUTZFELDT-JAKOB DISEASE (CJD) SURVEILLANCE UNIT REPORT

Thank you for your recent letter concerning the publication of the third annual report from the CJD Surveillance Unit. I am sorry that you are dissatisfied with the way in which this report was published.

The Surveillance Unit is a completely independant outside body and the Department of Health is committed to publishing their reports as soon as they become available. In the circumstances it is not the practice to circulate the report for comment since the findings of the report would not be amended. In future we can ensure that the British Deer Farmers Association receives a copy of the report in advance of publication.

The Chief Medical Officer has undertaken to keep the public fully informed of the results of any research in respect of CJD. This report was entirely the work of the unit and was produced completely independantly of the the Department.

The statistical results reqarding the consumption of venison was put into perspective in the body of the report and was not mentioned at all in the press release. Media attention regarding this report was low key but gave a realistic presentation of the statistical findings of the Unit. This approach to publication was successful in that consumption of venison was highlighted only once by the media ie. in the News at one television proqramme.

I believe that a further statement about the report, or indeed statistical links between CJD and consumption of venison, would increase, and quite possibly give damaging credence, to the whole issue. From the low key media reports of which I am aware it seems unlikely that venison consumption will suffer adversely, if at all. ...end

http://web.archive.org/web/20030511010117/http://www.bseinquiry.gov.uk/files/yb/1994/10/00003001.pdf

see watered down report here ;

http://web.archive.org/web/20030511211625/http://www.bseinquiry.gov.uk/files/yb/1994/10/00004001.pdf

The first L-BSE case was reported in a young Holstein steer (23 months old; BSE/JP8).

snip...

To date, 27 cases of L-BSE and 24 cases of H-BSE have been reported worldwide (16), thus meaning that the prevalence of atypical BSE is considerably lower than that of C-BSE. However, recent studies showed that L-BSE is easily transmissible to transgenic mice expressing human (17,18) or bovine (19,20) prion protein, as well as to non-human primates (21), with shorter incubation periods than for the transmission of C-BSE to these animals. The virulent nature of L-BSE has stimulated new concern for human public health since the transmission of C-BSE to humans resulted in variant Creutzfeldt-Jakob disease (vCJD) (4-7), a new emergent prion disease.

http://www.nih.go.jp/JJID/64/81.pdf

These results suggest that L-type BSE is more virulent strain to primates comparing to cBSE.

http://www.prion2010.org/bilder/prion_2010_program_latest_w_posters_4_.pdf?139&PHPSESSID=a30a38202cfec579000b77af81be3099

Monday, May 12, 2008

BSE YOUNGEST AGE STATISTICS UNDER 30 MONTHS

http://bseyoungestage.blogspot.com/

Wednesday, March 31, 2010

Atypical BSE in Cattle

To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE. When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.

This study will contribute to a correct definition of specified risk material (SRM) in atypical BSE. The incumbent of this position will develop new and transfer existing, ultra-sensitive methods for the detection of atypical BSE in tissue of experimentally infected cattle.

http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2

snip...

please see all seven threats listed in the USA, and more...FULL TEXT ;

Thursday, August 12, 2010

Seven main threats for the future linked to prions

First threat

The TSE road map defining the evolution of European policy for protection against prion diseases is based on a certain numbers of hypotheses some of which may turn out to be erroneous. In particular, a form of BSE (called atypical Bovine Spongiform Encephalopathy), recently identified by systematic testing in aged cattle without clinical signs, may be the origin of classical BSE and thus potentially constitute a reservoir, which may be impossible to eradicate if a sporadic origin is confirmed. Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.

Second threat

snip...

http://www.neuroprion.org/en/np-neuroprion.html

http://prionpathy.blogspot.com/2010/08/seven-main-threats-for-future-linked-to.html

http://prionpathy.blogspot.com/

P26 TRANSMISSION OF ATYPICAL BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN HUMANIZED MOUSE MODELS

Liuting Qing1, Fusong Chen1, Michael Payne1, Wenquan Zou1, Cristina Casalone2, Martin Groschup3, Miroslaw Polak4, Maria Caramelli2, Pierluigi Gambetti1, Juergen Richt5*, and Qingzhong Kong1 1Department of Pathology, Case Western Reserve University, Cleveland, OH 44106, USA; 2CEA, Istituto Zooprofilattico Sperimentale, Italy; 3Friedrich-Loeffler-Institut, Germany; 4National Veterinary Research Institute, Poland; 5Kansas State University, Diagnostic Medicine/Pathobiology Department, Manhattan, KS 66506, USA. *Previous address: USDA National Animal Disease Center, Ames, IA 50010, USA

Classical BSE is a world-wide prion disease in cattle, and the classical BSE strain (BSE-C) has led to over 200 cases of clinical human infection (variant CJD). Two atypical BSE strains, BSE-L (also named BASE) and BSE-H, have been discovered in three continents since 2004. The first case of naturally occurring BSE with mutated bovine PrP gene (termed BSE-M) was also found in 2006 in the USA. The transmissibility and phenotypes of these atypical BSE strains/isolates in humans were unknown. We have inoculated humanized transgenic mice with classical and atypical BSE strains (BSE-C, BSE-L, BSE-H) and the BSE-M isolate. We have found that the atypical BSE-L strain is much more virulent than the classical BSE-C. The atypical BSE-H strain is also transmissible in the humanized transgenic mice with distinct phenotype, but no transmission has been observed for the BSE-M isolate so far.

III International Symposium on THE NEW PRION BIOLOGY: BASIC SCIENCE, DIAGNOSIS AND THERAPY 2 - 4 APRIL 2009, VENEZIA (ITALY)

http://www.istitutoveneto.it/prion_09/Abstracts_09.pdf

Monday, October 19, 2009

Atypical BSE, BSE, and other human and animal TSE in North America Update October 19, 2009

snip...

I ask Professor Kong ;

Thursday, December 04, 2008 3:37 PM Subject: RE: re--Chronic Wating Disease (CWD) and Bovine Spongiform Encephalopathies (BSE): Public Health Risk Assessment

''IS the h-BSE more virulent than typical BSE as well, or the same as cBSE, or less virulent than cBSE? just curious.....''

Professor Kong reply ;

.....snip

''As to the H-BSE, we do not have sufficient data to say one way or another, but we have found that H-BSE can infect humans. I hope we could publish these data once the study is complete.

Thanks for your interest.''

Best regards,

Qingzhong Kong, PhD Associate Professor Department of Pathology Case Western Reserve University Cleveland, OH 44106 USA

Rare BSE mutation raises concerns over risks to public health

SIR — Atypical forms (known as H- and L-type) of bovine spongiform encephalopathy (BSE) have recently appeared in several European countries as well as in Japan, Canada and the United States. This raises the unwelcome possibility that variant Creutzfeldt–Jakob disease (vCJD) could increase in the human population. Of the atypical BSE cases tested so far, a mutation in the prion protein gene (PRNP) has been detected in just one, a cow in Alabama with BSE; her healthy calf also carried the mutation (J. A. Richt and S. M. Hall PLoS Pathog. 4, e1000156; 2008). This raises the possibility that the disease could occasionally be genetic in origin. Indeed, the report of the UK BSE Inquiry in 2000 suggested that the UK epidemic had most likely originated from such a mutation and argued against the scrapierelated assumption. Such rare potential pathogenic PRNP mutations could occur in countries at present considered to be free of BSE, such as Australia and New Zealand. So it is important to maintain strict surveillance for BSE in cattle, with rigorous enforcement of the ruminant feed ban (many countries still feed ruminant proteins to pigs). Removal of specified risk material, such as brain and spinal cord, from cattle at slaughter prevents infected material from entering the human food chain. Routine genetic screening of cattle for PRNP mutations, which is now available, could provide additional data on the risk to the public. Because the point mutation identified in the Alabama animals is identical to that responsible for the commonest type of familial (genetic) CJD in humans, it is possible that the resulting infective prion protein might cross the bovine–human species barrier more easily. Patients with vCJD continue to be identified. The fact that this is happening less often should not lead to relaxation of the controls necessary to prevent future outbreaks. Malcolm A. Ferguson-Smith Cambridge University Department of Veterinary Medicine, Madingley Road, Cambridge CB3 0ES, UK e-mail: maf12@cam.ac.uk Jürgen A. Richt College of Veterinary Medicine, Kansas State University, K224B Mosier Hall, Manhattan, Kansas 66506-5601, USA

NATUREVol 45726 February 2009

http://www.nature.com/nature/journal/v457/n7233/full/4571079b.html

Our findings demonstrate that cervid PrPSc, upon strain adaptation by serial passages in vitro or in cervid transgenic mice, is capable of converting human PrPC to produce PrPSc with unique biochemical properties, likely representing a new human prion strain. The newly generated CWD-huPrPSc material has been inoculated into transgenic mice expressing human PrP to study infectivity and disease phenotype and this data will be published elsewhere.

http://www.jbc.org/content/early/2011/01/04/jbc.M110.198465.long

Tuesday, January 25, 2011

Generation of a new form of human PrPSc in vitro by inter-species transmission from cervids prions

http://chronic-wasting-disease.blogspot.com/2011/01/generation-of-new-form-of-human-prpsc.html

WHAT ABOUT CWD AND LIVESTOCK, especially, second passage ;

Title: Experimental Second Passage of Chronic Wasting Disease (Cwd(mule Deer)) Agent to Cattle

Authors

Hamir, Amirali Kunkle, Robert Miller, Janice - ARS RETIRED Greenlee, Justin Richt, Juergen

Submitted to: Journal of Comparative Pathology Publication Type: Peer Reviewed Journal Publication Acceptance Date: July 25, 2005 Publication Date: January 1, 2006 Citation: Hamir, A.N., Kunkle, R.A., Miller, J.M., Greenlee, J.J., Richt, J.A. 2006. Experimental second passage of chronic wasting disease (CWD(mule deer)) agent to cattle. Journal of Comparative Pathology. 134(1):63-69.

Interpretive Summary: To compare the findings of experimental first and second passage of chronic wasting disease (CWD) in cattle, 6 calves were inoculated into the brain with CWD-mule deer agent previously (first) passaged in cattle. Two other uninoculated calves served as controls. Beginning 10-12 months post inoculation (PI), all inoculates lost appetite and weight. Five animals subsequently developed clinical signs of central nervous system (CNS) abnormality. By 16.5 months PI, all cattle had been euthanized because of poor prognosis. None of the animals showed microscopic lesions of spongiform encephalopathy (SE) but the CWD agent was detected in their CNS tissues by 2 laboratory techniques (IHC and WB). These findings demonstrate that inoculated cattle amplify CWD agent but also develop clinical CNS signs without manifestation of microscopic lesions of SE. This situation has also been shown to occur following inoculation of cattle with another TSE agent, namely, sheep scrapie. The current study confirms previous work that indicates that the diagnostic tests currently used for confirmation of bovine spongiform encephalopathy (BSE) in the U.S. would detect CWD in cattle, should it occur naturally. Furthermore, it raises the possibility of distinguishing CWD from BSE in cattle due to the absence of microscopic lesions and a unique multifocal distribution of PrPres, as demonstrated by IHC, which in this study, appears to be more sensitive than the WB. Technical Abstract: To compare clinicopathological findings of first and second passage of chronic wasting disease (CWD) in cattle, a group of calves (n=6) were intracerebrally inoculated with CWD-mule deer agent previously (first) passaged in cattle. Two other uninoculated calves served as controls. Beginning 10-12 months post inoculation (PI), all inoculates lost appetite and lost weight. Five animals subsequently developed clinical signs of central nervous system (CNS) abnormality. By 16.5 months PI, all cattle had been euthanized because of poor prognosis. None of the animals showed microscopic lesions of spongiform encephalopathy (SE) but PrPres was detected in their CNS tissues by immunohistochemistry (IHC) and Western blot (WB) techniques. These findings demonstrate that intracerebrally inoculated cattle not only amplify CWD PrPres but also develop clinical CNS signs without manifestation of morphologic lesions of SE. This situation has also been shown to occur following inoculation of cattle with another TSE agent, scrapie. The current study confirms previous work that indicates the diagnostic techniques currently used for confirmation of bovine spongiform encephalopathy (BSE) in the U.S. would detect CWD in cattle, should it occur naturally. Furthermore, it raises the possibility of distinguishing CWD from BSE in cattle due to the absence of neuropathologic lesions and a unique multifocal distribution of PrPres, as demonstrated by IHC, which in this study, appears to be more sensitive than the WB.

http://www.ars.usda.gov/research/publications/publications.htm?SEQ_NO_115=178318

Wednesday, January 19, 2011

EFSA BIOHAZ Scientific Opinion on the revision of the quantitative risk assessment (QRA) of the BSE risk posed by processed animal proteins (PAPs)

EFSA Journal 2011;9(1):1947

http://transmissiblespongiformencephalopathy.blogspot.com/2011/01/efsa-biohaz-scientific-opinion-on.html

Monday, January 17, 2011

MAD COW Update on Feed Enforcement Activities to Limit the Spread of BSE January 13, 2011

January 2011

http://transmissiblespongiformencephalopathy.blogspot.com/2011/01/mad-cow-update-on-feed-enforcement.html

Thursday, November 18, 2010

Increased susceptibility of human-PrP transgenic mice to bovine spongiform encephalopathy following passage in sheep

http://bse-atypical.blogspot.com/2010/11/increased-susceptibility-of-human-prp.html

Tuesday, January 18, 2011

Agent strain variation in human prion disease: insights from a molecular and pathological review of the National Institutes of Health series of experimentally transmitted disease

http://transmissiblespongiformencephalopathy.blogspot.com/2011/01/agent-strain-variation-in-human-prion.html

Thursday, December 23, 2010

Molecular Typing of Protease-Resistant Prion Protein in Transmissible Spongiform Encephalopathies of Small Ruminants, France, 2002–2009 Volume 17, Number 1–January 2011

http://transmissiblespongiformencephalopathy.blogspot.com/2010/12/molecular-typing-of-protease-resistant.html

Wednesday, January 19, 2011

EFSA and ECDC review scientific evidence on possible links between TSEs in animals and humans Webnachricht 19 Januar 2011

http://transmissiblespongiformencephalopathy.blogspot.com/2011/01/efsa-and-ecdc-review-scientific.html

Friday, January 21, 2011

Strain-Specific Barriers against Bovine Prions in Hamsters

http://transmissiblespongiformencephalopathy.blogspot.com/2011/01/strain-specific-barriers-against-bovine.html

Saturday, December 18, 2010

OIE Global Conference on Wildlife Animal Health and Biodiversity - Preparing for the Future (TSE AND PRIONS) Paris (France), 23-25 February 2011

http://transmissiblespongiformencephalopathy.blogspot.com/2010/12/oie-global-conference-on-wildlife.html

Monday, November 30, 2009

USDA AND OIE COLLABORATE TO EXCLUDE ATYPICAL SCRAPIE NOR-98 ANIMAL HEALTH CODE

http://nor-98.blogspot.com/2009/11/usda-and-oie-collaborate-to-exclude.html

http://bseusa.blogspot.com/2010/04/usda-and-oie-out-of-touch-with-risk.html

Tuesday, November 02, 2010

BSE - ATYPICAL LESION DISTRIBUTION (RBSE 92-21367) statutory (obex only) diagnostic criteria CVL 1992

http://bse-atypical.blogspot.com/2010/11/bse-atypical-lesion-distribution-rbse.html

Friday, February 04, 2011

NMLB and USDA allow scrapie prion infected mutton to enter food chain on the Navajo Reservation in New Mexico

----- Original Message -----

From: Terry S. Singeltary Sr.

To: President.BenShelly

Cc: sroanhorse ; opvp.nelson ; alaughing; georgehardeen; pressoffice

Sent: Thursday, February 03, 2011 12:15 PM

Subject: NMLB and USDA allow scrapie prion infected mutton to enter food chain on the Navajo Reservation in New Mexico

Greetings Honorable People of the Great Navajo Nation, and the Honorable President Ben Shelly,

I send this to you with great concern. ...

http://scrapie-usa.blogspot.com/2011/02/nmlb-and-usda-allow-scrapie-prion.html

USA

5 Includes 16 cases in which the diagnosis is pending, and 18 inconclusive cases;

6 Includes 21 (19 from 2010) cases with type determination pending in which the diagnosis of vCJD has been excluded.

2010

PLEASE NOTE REFERENCE LINES 5. AND 6.

Monday, August 9, 2010

National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010) Year Total Referrals2 Prion Disease Sporadic Familial Iatrogenic vCJD

1996 & earlier 51 33 28 5 0 0

1997 114 68 59 9 0 0

1998 88 52 44 7 1 0

1999 120 72 64 8 0 0

2000 146 103 89 14 0 0

2001 209 119 109 10 0 0

2002 248 149 125 22 2 0

2003 274 176 137 39 0 0

2004 325 186 164 21 0 1(3)

2005 344 194 157 36 1 0

2006 383 197 166 29 0 2(4)

2007 377 214 187 27 0 0

2008 394 231 204 25 0 0

2009 425 259 216 43 0 0

2010 204 124 85 20 0 0

TOTAL 3702(5) 2177(6) 1834 315 4 3

1 Listed based on the year of death or, if not available, on year of referral;

2 Cases with suspected prion disease for which brain tissue and/or blood (in familial cases) were submitted;

3 Disease acquired in the United Kingdom;

4 Disease was acquired in the United Kingdom in one case and in Saudi Arabia in the other case;

5 Includes 16 cases in which the diagnosis is pending, and 18 inconclusive cases;

6 Includes 21 (19 from 2010) cases with type determination pending in which the diagnosis of vCJD has been excluded.

http://www.cjdsurveillance.com/pdf/case-table.pdf

Monday, August 9, 2010

National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010)

(please watch and listen to the video and the scientist speaking about atypical BSE and sporadic CJD and listen to Professor Aguzzi)

http://prionunitusaupdate2008.blogspot.com/2010/08/national-prion-disease-pathology.html

Saturday, June 13, 2009

Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States 2003 revisited 2009

http://cjdusa.blogspot.com/2009/06/monitoring-occurrence-of-emerging-forms.html

Saturday, January 2, 2010

Human Prion Diseases in the United States January 1, 2010 ***FINAL***

http://prionunitusaupdate2008.blogspot.com/2010/01/human-prion-diseases-in-united-states.html

my comments to PLosone here ;

http://www.plosone.org/annotation/listThread.action?inReplyTo=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd&root=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd

14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf

Tuesday, December 14, 2010

Infection control of CJD, vCJD and other human prion diseases in healthcare and community settings part 4, Annex A1, Annex J, UPDATE DECEMBER 2010

http://creutzfeldt-jakob-disease.blogspot.com/2010/12/infection-control-of-cjd-vcjd-and-other.html

HOW many of you recieved a written CJD Questionnaire asking real questions pertaining to route and source (and there are many here in North America) ?

IS every case getting a cjd questionnaire asking real questions ???

Friday, November 30, 2007

CJD QUESTIONNAIRE USA CWRU AND CJD FOUNDATION USA PRION UNIT

http://cjdquestionnaire.blogspot.com/2007/11/cjd-questionnaire.html

DEAD STOCK DOWNER COWS I.E. Non-Ambulatory, are the most high risk animal for mad cow type disease i.e. TSE, the prion disease.

DID your children eat them at school ?

WHO WILL WATCH THE CHILDREN FOR CJD OVER THE NEXT 5 + DECADES ???

Do you actually believe that the USDA et al jumped in on the law suit against Westland/Hallmark, at the time the largest beef recall in USA history, just because a few animals were abused on a video, or to cover their ass, for letting our children, from school district to school district, from state to state, be fed dead stock downer cows.

>>> In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks... <<<

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

Do you actually believe all these schools recalled this meat because of a few cattle being abused, see list ; FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114

IF url does not work above, go to this link to find out if any of your children and their school were part of this recall ; go to this site ;

http://www.fns.usda.gov/fns/ left hand corner search ; Hallmark/Westland Meat Packing Co. Beef Recall your should get this ;

http://65.216.150.153/texis/search?pr=FNS

1 through 1 of 1 matching documents, best matches first. sort by date 1: Hallmark - Westland SFA Reporting by State - 3-24-2008.xls Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 The U.S. Department of Agriculture ...

http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114

PLEASE SEE ALSO ; Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkWestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

PLEASE NOTE * Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME.

snip...

The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

PLEASE be aware, for 4 years, the USDA fed our children all across the Nation dead stock downer cows, the most high risk cattle for BSE aka mad cow disease and other dangerous pathogens. who will watch our children for CJD for the next 5+ decades ???

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

never surrender, never forget. ...

TSS

Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518 flounder9@verizon.net

Meatingplace.com bans Singeltary for praising First Lady Michelle Obama's Healthy Nutritional NSLP efforts

2010-10-13T13:25:00.000-07:00

Meatingplace.com bans Singeltary for praising First Lady Michelle Obama's Healthy Nutritional NSLP efforts

Greetings,

I have been posting for years to meatingplace.com on the issue of some beef producers, the USDA et al, and their blantant disregard for Transmissible Spongiform Encephalopathy. Today, i suppose was the last straw for them. They just could not handle any more of the truth. I wish i had downloaded the stupid reply i had replied to, just another rediculous comment blaming President Obama on everything. But they seemed upset about First Lady Michelle Obama and her effort in trying to teach our children and their parents about a healthier diet. evidently most were not having any part of a healthy diet for our Children, and I pointed out some of the past to one commentor. OF course you would think that most parents and consumers would NOT want their children eating dead stock downer cows. but fact is, they did for 4 plus years under the Bush administration. IT was not until it became law, and the bill was finally finalized under the Obama admistration that this ban on deadstock downer cows finally was finalized and became law. I simply commented and praised the Obama administration for this, and was THEN banned from any other free comments. now i must pay to comment as a consumer, after years and years of not paying. something smells $$$

article in question was ;

''USDA psyching kids into eating healthier lunches''.

http://www.meatingplace.com/ArticleLocator/ArticleRedirector.aspx?code=2s19036&rType=3

MY COMMENT ;

Ya know, some folks really crack me up, they would blame everything down to their dirty shorts on President Obama, instead of stopping and looking in the mirror. I guess some WOULD rather have our children all across the Nation feast on deadstock downer cows for over 4 years, anything for a buck right folks $ oh, that's right, that happened under the Bush administration, it was under the Obama administration that this heinous practices was banned finally. I applaud First Lady Michelle Obama's effort in trying to teach our children and their parents about a healthier diet. Have you looked at some of these young teens lately ? I am surprised some of the far right goons have not pounced on her for that, like most all the right was all up in arms about President Obama speaking to the school children. But these same right seem to forget, it was the school house, speaking to children, is exactly where President Bush was when the twin towers were hit. So, grow a pair America, fixing what got so screwed up over 8 years is not going to come over night, and it ain't going to be cheap. did any of you really believe that we could climb out of the hole we were in and it not cost us anything ? I watched the Jamie Olive movement, when he was in America, asking our school children about different foods and such. I was dumbfounded when just about a complete class room of "grade-schoolers, when shown a basket of vegetables - and they can't identify any of them. Not even the potato. Yes, it's sensational, reality television, but it's underscoring an abhorrent truth in America - not only don't people know where their food comes from, they don't even know what it is". Sadly, this was all fact. so was the following ;

WHO WILL WATCH THE CHILDREN FOR CJD OVER THE NEXT 5 + DECADES ???

PLEASE SEE ALSO ;

Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef.

As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

Thursday, October 07, 2010

Experimental Transmission of H-type Bovine Spongiform Encephalopathy to Bovinized Transgenic Mice

http://bse-atypical.blogspot.com/2010/10/experimental-transmission-of-h-type.html

Monday, August 9, 2010

National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010)

(please watch and listen to the video and the scientist speaking about atypical BSE and sporadic CJD and listen to Professor Aguzzi)

SEE where sporadic cjd in the USA went from 59 cases in 1997, to 216 cases in 2009. a steady increase since 1997. ...TSS

National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010)

Year Total Referrals2 Prion Disease Sporadic Familial Iatrogenic vCJD

1997 114 68 59 9 0 0

to

2009 425 259 216 43 0 0

http://www.cjdsurveillance.com/pdf/case-table.pdf

see full text ;

http://prionunitusaupdate2008.blogspot.com/2010/08/national-prion-disease-pathology.html

END

THEN, after someone made a reply to me, i was going to kindly reply back with more facts, but could not post anymore or access my free account was shut down, all i get now is this ;

Time to upgrade your membership...

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my how the truth hurts sometimes. ...TSS

CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER

>>> Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. <<<

Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas

Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas.

She left 6 Kids and a Husband.The Purpose of this web is to give information in Spanish to the Hispanic community, and to all the community who want's information about this terrible disease.-

Physician Discharge

Summary, Parkland Hospital, Dallas Texas

Admit Date: 12/29/2009

Discharge Date: 1/20/2010

Attending Provider: Greenberg, Benjamin Morris;

General Neurology Team: General Neurology Team

Linda was a Hispanic female with no past medical history presents with 14 months of incresing/progressive altered mental status, generalized weakness, inability to walk, loss of appetite, inability to speak, tremor and bowel/blader incontinence. She was, in her usual state of health up until February, 2009, when her husbans notes that she began forgetting things like names and short term memories. He also noticed mild/vague personality changes such as increased aggression. In March, she was involved in a hit and run MVA,although she was not injured. The police tracked her down and ticketed her. At that time, her son deployed to Iraq with the Army and her husband assumed her mentation changes were due to stress over these two events. Also in March, she began to have weakness in her legs, making it difficult to walk. Over the next few months, her mentation and personality changes worsened, getting to a point where she could no longer recognized her children. She was eating less and less. She was losing more weight. In the last 2-3 months, she reached the point where she could not walk without an assist, then 1 month ago, she stopped talking, only making grunting/aggressive sounds when anyone came near her. She also became both bowel and bladder incontinent, having to wear diapers. Her '"tremor'" and body jerks worsened and her hands assumed a sort of permanent grip position, leading her family to put tennis balls in her hands to protect her fingers. The husband says that they have lived in Nebraska for the past 21 years. They had seen a doctor there during the summer time who prescribed her Seroquel and Lexapro, Thinking these were sx of a mood disorder. However, the medications did not help and she continued to deteriorate clinically.

Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. The husband says that he does not know any fellow workers with a similar illness. He also says that she did not have any preceeding illness or travel.

http://www.recordandoalinda.com/index.php?option=com_content&view=article&id=19:cjd-english-info&catid=9:cjd-ingles&Itemid=8

Terry S. Singeltary Sr. has added the following comment:

According to the World Health Organisation, the future public health threat of vCJD in the UK and Europe and potentially the rest of the world is of concern and currently unquantifiable. However, the possibility of a significant and geographically diverse vCJD epidemic occurring over the next few decades cannot be dismissed .

The key word here is diverse. What does diverse mean?

If USA scrapie transmitted to USA bovine does not produce pathology as the UK c-BSE, then why would CJD from there look like UK vCJD?"

SEE FULL TEXT ;

http://www.promedmail.org/pls/apex/f?p=2400:1001:568933508083034::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1000,82101

.57 The experiment which might have determined whether BSE and scrapie were caused by the same agent (ie, the feeding of natural scrapie to cattle) was never undertaken in the UK. It was, however, performed in the USA in 1979, when it was shown that cattle inoculated with the scrapie agent endemic in the flock of Suffolk sheep at the United States Department of Agriculture in Mission, Texas, developed a TSE quite unlike BSE. 32 The findings of the initial transmission, though not of the clinical or neurohistological examination, were communicated in October 1988 to Dr Watson, Director of the CVL, following a visit by Dr Wrathall, one of the project leaders in the Pathology Department of the CVL, to the United States Department of Agriculture. 33 The results were not published at this point, since the attempted transmission to mice from the experimental cow brain had been inconclusive. The results of the clinical and histological differences between scrapie-affected sheep and cattle were published in 1995. Similar studies in which cattle were inoculated intracerebrally with scrapie inocula derived from a number of scrapie-affected sheep of different breeds and from different States, were carried out at the US National Animal Disease Centre. 34 The results, published in 1994, showed that this source of scrapie agent, though pathogenic for cattle, did not produce the same clinical signs of brain lesions characteristic of BSE.

32 Clark, W., Hourrigan, J. and Hadlow, W. (1995) Encephalopathy in Cattle Experimentally Infected with the Scrapie Agent, American Journal of Veterinary Research, 56, 606-12

33 YB88/10.00/1.1

http://web.archive.org/web/20040823105233/www.bseinquiry.gov.uk/files/yb/1988/10/00001001.pdf

THIS is one month recall page, where 10 MILLION POUNDS OF BANNED MAD COW FEED WENT OUT INTO COMMERCE, TO BE FED OUT. very little of the product that reaches commerce is ever returned via recall, very, very little. this was 2007, TEN YEARS AFTER THE AUGUST 4, 1997, PARTIAL AND VOLUNTARY MAD COW FEED BAN IN THE USA, that was nothing but ink on paper. i have listed the tonnage of mad cow feed that was in ALABAMA in one of the links too, this is where the infamous g-h-BSEalabama case was, a genetic relation matching the new sporadic CJD in the USA. seems this saga just keeps getting better and better.......$$$

10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. BLOOD LACED MBM IN COMMERCE USA 2007

Date: March 21, 2007 at 2:27 pm PST

RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II

___________________________________

PRODUCT

Bulk cattle feed made with recalled Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007

CODE

Cattle feed delivered between 01/12/2007 and 01/26/2007

RECALLING FIRM/MANUFACTURER

Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.

Firm initiated recall is ongoing.

REASON

Blood meal used to make cattle feed was recalled because it was cross- contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement.

VOLUME OF PRODUCT IN COMMERCE

42,090 lbs.

DISTRIBUTION

WI

___________________________________

PRODUCT

Custom dairy premix products: MNM ALL PURPOSE Pellet, HILLSIDE/CDL Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal, TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML W/SMARTA, Recall # V-025-2007

CODE

The firm does not utilize a code - only shipping documentation with commodity and weights identified.

RECALLING FIRM/MANUFACTURER

Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007. Firm initiated recall is complete.

REASON

Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement.

VOLUME OF PRODUCT IN COMMERCE

9,997,976 lbs.

DISTRIBUTION

ID and NV

END OF ENFORCEMENT REPORT FOR MARCH 21, 2007

http://www.fda.gov/Safety/Recalls/EnforcementReports/2007/ucm120446.htm

Tuesday, September 14, 2010

Feed Safety and BSE/Ruminant Feed Ban Support Project (U18)

http://madcowfeed.blogspot.com/2010/09/feed-safety-and-bseruminant-feed-ban.html

Saturday, October 2, 2010

BSE surveillance front and centre: CFIA and USA

http://madcowtesting.blogspot.com/2010/10/bse-surveillance-front-and-centre-cfia.html

Sunday, October 3, 2010

Scrapie, Nor-98 atypical Scrapie, and BSE in sheep and goats North America, who's looking ?

http://nor-98.blogspot.com/2010/10/scrapie-nor-98-atypical-scrapie-and-bse.html

let's take a closer look at this new prionpathy or prionopathy, and then let's look at the g-h-BSEalabama mad cow.

This new prionopathy in humans? the genetic makeup is IDENTICAL to the g-h-BSEalabama mad cow, the only _documented_ mad cow in the world to date like this, ......wait, it get's better. this new prionpathy is killing young and old humans, with LONG DURATION from onset of symptoms to death, and the symptoms are very similar to nvCJD victims, OH, and the plaques are very similar in some cases too, bbbut, it's not related to the g-h-BSEalabama cow, WAIT NOW, it gets even better, the new human prionpathy that they claim is a genetic TSE, has no relation to any gene mutation in that family. daaa, ya think it could be related to that mad cow with the same genetic make-up ??? there were literally tons and tons of banned mad cow protein in Alabama in commerce, and none of it transmitted to cows, and the cows to humans there from ??? r i g h t $$$

ALABAMA MAD COW g-h-BSEalabama

In this study, we identified a novel mutation in the bovine prion protein gene (Prnp), called E211K, of a confirmed BSE positive cow from Alabama, United States of America. This mutation is identical to the E200K pathogenic mutation found in humans with a genetic form of CJD. This finding represents the first report of a confirmed case of BSE with a potential pathogenic mutation within the bovine Prnp gene. We hypothesize that the bovine Prnp E211K mutation most likely has caused BSE in "the approximately 10-year-old cow" carrying the E221K mutation.

http://www.plospathogens.org/article/info%3Adoi%2F10.1371%2Fjournal.ppat.1000156

http://www.plospathogens.org/article/fetchObjectAttachment.action?uri=info%3Adoi%2F10.1371%2Fjournal.ppat.1000156&representation=PDF

Saturday, August 14, 2010

BSE Case Associated with Prion Protein Gene Mutation (g-h-BSEalabama) and VPSPr PRIONPATHY

(see mad cow feed in COMMERCE IN ALABAMA...TSS)

http://prionpathy.blogspot.com/2010/08/bse-case-associated-with-prion-protein.html

THIS is why this question is so important to me, IS h-BSE more or less virulent than the c-BSE ?

Wednesday, July 28, 2010

re-Freedom of Information Act Project Number 3625-32000-086-05, Study of Atypical BSE UPDATE July 28, 2010

http://bse-atypical.blogspot.com/2010/07/re-freedom-of-information-act-project.html

SEE where sporadic cjd in the USA went from 59 cases in 1997, to 216 cases in 2009. a steady increase since 1997. ...TSS

Monday, August 9, 2010

National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010)

Year Total Referrals2 Prion Disease Sporadic Familial Iatrogenic vCJD

1997 114 68 59 9 0 0

to

2009 425 259 216 43 0 0

http://www.cjdsurveillance.com/pdf/case-table.pdf

PLEASE SEE FULL TEXT HERE ;

Monday, August 9, 2010

National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010)

(please watch and listen to the video and the scientist speaking about atypical BSE and sporadic CJD and listen to Professor Aguzzi)

http://prionunitusaupdate2008.blogspot.com/2010/08/national-prion-disease-pathology.html

Tuesday, August 03, 2010

Variably protease-sensitive prionopathy: A new sporadic disease of the prion protein

http://creutzfeldt-jakob-disease.blogspot.com/2010/08/variably-protease-sensitive-prionopathy.html

Monday, August 9, 2010

Variably protease-sensitive prionopathy: A new sporadic disease of the prion protein or just more PRIONBALONEY ?

http://prionunitusaupdate2008.blogspot.com/2010/08/variably-protease-sensitive-prionopathy.html

***+++***

Thursday, July 10, 2008

A Novel Human Disease with Abnormal Prion Protein Sensitive to Protease update July 10, 2008 Friday, June 20, 2008

http://cjdmadcowbaseoct2007.blogspot.com/2008/07/novel-human-disease-with-abnormal-prion.html

BRITISH MEDICAL JOURNAL

Re: vCJD in the USA * BSE in U.S. 15 November 1999

Terry S Singeltary

snip...

It's their move, it's CHECK, but once CHECKMATE has been called, how many thousands or millions, will be at risk or infected or even dead. You can't play around with these TSE's. I cannot stress that enough. They are only looking at body bags, and the fact the count is so low. But, then you have to look at the fact it is not a reportable disease in most states, mis-diagnosis, no autopsies performed. The fact that their one-in-a- million theory is a crude survey done about 5 years ago, that's a joke, under the above circumstances. A bad joke indeed........

snip...

http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406

BRITISH MEDICAL JOURNAL

U.S. Scientist should be concerned with a CJD epidemic in the U.S., as well...

2 January 2000

Terry S Singeltary

In reading your short article about 'Scientist warn of CJD epidemic' news in brief Jan. 1, 2000. I find the findings in the PNAS old news, made famous again. Why is the U.S. still sitting on their butts, ignoring the facts? We have the beginning of a CJD epidemic in the U.S., and the U.S. Gov. is doing everything in it's power to conceal it.

The exact same recipe for B.S.E. existed in the U.S. for years and years. In reading over the Qualitative Analysis of BSE Risk Factors-1, this is a 25 page report by the USDA:APHIS:VS. It could have been done in one page. The first page, fourth paragraph says it all;

"Similarities exist in the two countries usage of continuous rendering technology and the lack of usage of solvents, however, large differences still remain with other risk factors which greatly reduce the potential risk at the national level."

Then, the next 24 pages tries to down-play the high risks of B.S.E. in the U.S., with nothing more than the cattle to sheep ratio count, and the geographical locations of herds and flocks. That's all the evidence they can come up with, in the next 24 pages.

Something else I find odd, page 16;

"In the United Kingdom there is much concern for a specific continuous rendering technology which uses lower temperatures and accounts for 25 percent of total output. This technology was _originally_ designed and imported from the United States. However, the specific application in the production process is _believed_ to be different in the two countries."

A few more factors to consider, page 15;

snip...see full text ;

http://www.bmj.com/cgi/eletters/320/7226/8/b#6117

Diagnosis and Reporting of Creutzfeldt-Jakob Disease

Singeltary, Sr et al. JAMA.2001; 285: 733-734.

Diagnosis and Reporting of Creutzfeldt-Jakob Disease

Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

To the Editor: In their Research Letter, Dr Gibbons and colleagues1 reported that the annual US death rate due to Creutzfeldt-Jakob disease (CJD) has been stable since 1985. These estimates, however, are based only on reported cases, and do not include misdiagnosed or preclinical cases. It seems to me that misdiagnosis alone would drastically change these figures. An unknown number of persons with a diagnosis of Alzheimer disease in fact may have CJD, although only a small number of these patients receive the postmortem examination necessary to make this diagnosis. Furthermore, only a few states have made CJD reportable. Human and animal transmissible spongiform encephalopathies should be reportable nationwide and internationally.

Terry S. Singeltary, Sr Bacliff, Tex

1. Gibbons RV, Holman RC, Belay ED, Schonberger LB. Creutzfeldt-Jakob disease in the United States: 1979-1998. JAMA. 2000;284:2322-2323. FREE FULL TEXT

http://jama.ama-assn.org/cgi/content/extract/285/6/733?maxtoshow=&hits=10&RESULTFORMAT=&fulltext=singeltary&searchid=1&FIRSTINDEX=0&resourcetype=HWCIT

http://jama.ama-assn.org/cgi/content/full/285/6/733

JOURNAL OF NEUROLOGY

MARCH 26, 2003

RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob

disease in the United States

Email Terry S. Singeltary:

flounder@wt.net

I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which are sporadic are all spontaneous, without route/source. We have many TSEs in the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey by intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly expose others. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic CJDs in the USA are type 2 PrPSc?

http://www.neurology.org/cgi/eletters/60/2/176#535

THE PATHOLOGICAL PROTEIN Hardcover, 304 pages plus photos and illustrations. ISBN 0-387-95508-9

June 2003

BY Philip Yam

CHAPTER 14 LAYING ODDS

Answering critics like Terry Singeltary, who feels that the U.S. under- counts CJD, Schonberger conceded that the current surveillance system has errors but stated that most of the errors will be confined to the older population.

http://www.thepathologicalprotein.com/

doi:10.1016/S1473-3099(03)00715-1 Copyright © 2003 Published by Elsevier Ltd. Newsdesk

Tracking spongiform encephalopathies in North America

Xavier Bosch

Available online 29 July 2003.

Volume 3, Issue 8, August 2003, Page 463

"My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my mom to hvCJD (Heidenhain variant CJD) and have been searching for answers ever since. What I have found is that we have not been told the truth. CWD in deer and elk is a small portion of a much bigger problem." 49-year-old Singeltary is one of a number of people who have remained largely unsatisfied after being told that a close relative died from a rapidly progressive dementia compatible with spontaneous Creutzfeldt-Jakob ...

............................

http://www.thelancet.com/journals/laninf/article/PIIS1473309903007151/fulltext

http://download.thelancet.com/pdfs/journals/1473-3099/PIIS1473309903007151.pdf

2006

There is a growing number of human CJD cases, and they were presented last week in San Francisco by Luigi Gambatti(?) from his CJD surveillance collection.

He estimates that it may be up to 14 or 15 persons which display selectively SPRPSC and practically no detected RPRPSC proteins.

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/1006-4240t1.htm

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/2006-4240t1.pdf

2008 - 2010

The statistical incidence of CJD cases in the United States has been revised to reflect that there is one case per 9000 in adults age 55 and older. Eighty-five percent of the cases are sporadic, meaning there is no known cause at present.

http://www.cjdfoundation.org/fact.html

CJD USA RISING, with UNKNOWN PHENOTYPE ;

5 Includes 41 cases in which the diagnosis is pending, and 17 inconclusive cases; 6 Includes 46 cases with type determination pending in which the diagnosis of vCJD has been excluded.

http://www.cjdsurveillance.com/pdf/case-table.pdf

Friday, November 30, 2007

CJD QUESTIONNAIRE USA CWRU AND CJD FOUNDATION

http://cjdquestionnaire.blogspot.com/

Meeting of the Transmissible Spongiform Encephalopathies Committee On June 12, 2009 (TRANSCRIPT)

http://www.fda.gov/downloads/AdvisoryCommittees/CommitteesMeetingMaterials/BloodVaccinesandOtherBiologics/TransmissibleSpongiformEncephalopathiesAdvisoryCommittee/UCM171810.pdf

Meeting of the Transmissible Spongiform Encephalopathies Committee On June 12, 2009 (Singeltary submission)

http://tseac.blogspot.com/2009/05/meeting-of-transmissible-spongiform.html

Saturday, June 13, 2009

Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States 2003 revisited 2009

http://cjdusa.blogspot.com/2009/06/monitoring-occurrence-of-emerging-forms.html

Friday, February 05, 2010

New Variant Creutzfelt Jakob Disease case reports United States 2010 A Review

http://vcjd.blogspot.com/2010/02/new-variant-creutzfelt-jakob-disease.html

Saturday, January 2, 2010

Human Prion Diseases in the United States January 1, 2010 ***FINAL***

http://prionunitusaupdate2008.blogspot.com/2010/01/human-prion-diseases-in-united-states.html

my comments to PLosone here ;

http://www.plosone.org/annotation/listThread.action?inReplyTo=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd&root=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd

Tuesday, September 14, 2010

Transmissible Spongiform Encephalopathies Advisory Committee; Notice of Meeting October 28 and 29, 2010 (COMMENT SUBMISSION)

http://tseac.blogspot.com/2010/09/transmissible-spongiform_14.html

14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

page 114 ;

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf

layperson

Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518
flounder9@verizon.net

USDA Encourages Schools to Take the HealthierUS School Challenge to Help Improve the Nutrition of School Children Nationwide

2010-08-24T11:33:00.000-07:00

Release No. 0419.10 Contact: USDA Office of Communications (202) 720-4623

USDA Encourages Schools to Take the Healthier

US School Challenge to Help Improve the Nutrition of School Children Nationwide New Steps Taken To Make Challenge Easier and More Successful

POTEET, Texas, Aug. 23, 2010 – Agriculture Secretary Tom Vilsack today encouraged schools throughout the country to participate in the HealthierUS School Challenge (HUSSC), an initiative that helps improve the health and nutrition of children. USDA created the HUSSC to recognize schools that maintain healthy school environments by improving the quality of meals and increasing physical activity and nutrition education. The HUSSC is also a key component of First Lady Michelle Obama's Let's Move! campaign to end childhood obesity within a generation.

"Even as Congress considers legislation to make historic improvements to school meals, there are also steps that moms and dads and others in the community can take to improve the health and nutrition of the meals their kids eat each day," said Vilsack. "While many schools have made healthful changes to school meals, more can be accomplished. The Challenge encourages schools to take voluntary extra steps toward improving the nutrition of all foods offered in schools, enhancing the opportunity for children to be physically active during the school day and providing nutrition education to help them learn how to make healthy lifestyle decisions."

In February 2010, USDA and First Lady Michelle Obama called on stakeholders to double the number of HUSSC schools in a year and add 1,000 schools per year for two years after that. Currently 794 HUSSC awards have been made to schools. USDA's Food and Nutrition Service (FNS) is making it easier and more appealing for schools to take the HUSSC including:

Providing schools monetary incentives for earning HUSSC certification. FNS certifies schools as Gold of Distinction, Gold, Silver or Bronze status. HUSSC schools receive $2,000 for Gold Award of Distinction, $1,500 for Gold, $1,000 for Silver and $500 for Bronze; Offering an online HUSSC toolkit to provide schools a step by step guide on how to navigate the application process; and Providing schools with a range of educational and technical assistance materials that promote consumption of fruits and vegetables and other key aspects of the Dietary Guidelines – including a Menu Planner for Healthy School Meals. According to an Institute of Medicine Report released in October 2009, school-age children eat too many discretionary calories, too few fruits and vegetables, particularly dark green and dark orange fruits and vegetables, too few whole grains and low-fat dairy products and too many solid fats and sugars. Over the past two decades, USDA has worked to eliminate or reduce levels of fat, sodium and sugar in foods it makes available to schools and other outlets.

"If you look at the health and nutrition of our nation's children, you can see we need immediate action," said Under Secretary for Food, Nutrition and Consumer Services Kevin Concannon. "Obesity is growing faster than any other public health issue in the United States. The HUSSC raises the bar and helps schools improve the quality of school meals and increase the participation in school meal programs. We can accomplish a number of these goals through rapid passage of the Child Nutrition Reauthorization now before Congress."

Improving USDA's child nutrition programs are a top priority of the Obama Administration. Congress is currently considering legislation to bolster the Child Nutrition Act, which authorizes the National School Lunch, School Breakfast, and Summer Food Service Programs. These programs serve nearly 32 million children each school day and work in concert to form a national safety net against hunger. Improving the Child Nutrition Act is the legislative centerpiece of First Lady Michelle Obama's Let's Move! campaign and highlighted in the White House report Solving the Problem of Childhood Obesity Within a Generation released Tuesday, May 11. By passing strong reauthorization legislation, the Administration hopes to reduce hunger, promote access, and improve the overall health and nutrition of children throughout the country.

USDA's Food and Nutrition Service (FNS) oversees the administration of 15 nutrition assistance programs, including the child nutrition programs, which touch the lives of one in four Americans over the course of a year. More information about FNS and its nutrition assistance programs is available at www.fns.usda.gov.

--------------------------------------------------------------------------------

USDA is an equal opportunity provider, employer and lender. To file a complaint of discrimination, write: USDA, Director, Office of Civil Rights, 1400 Independence Ave., SW, Washington, DC 20250-9410 or call (800) 795-3272 (voice), or (202) 720-6382 (TDD).

http://www.usda.gov/wps/portal/usda/!ut/p/c4/04_SB8K8xLLM9MSSzPy8xBz9CP0os_gAC9-wMJ8QY0MDpxBDA09nXw9DFxcXQ-cAA_2CbEdFAEUOjoE!/?contentidonly=true&contentid=2010%2F08%2F0419.xml

Greetings,

I would like to thank Madame First Lady Michelle Obama, and her husband the President, for standing up to big ag and poisoned food from big ag, from the most high risk cattle i.e. dead stock downers, going to our children all across the Nation, and going the extra step to make the food for our children safer and more nutritious.

Thanks !

Considering what the NSLP and the USDA have done in the past, I would welcome any new standard. So I applaud these new standards.

However, feeding those dead stock downer cows to children for over 4 years was something they should never ever be able to forget about. dead stock downer cows are the most high risk animal for mad cow disease, and they knew this when the feeding to our children of this product was taking place. the USDA et al via the NSLP fed our children all across the USA, from state to state, the largest beef recall in history, they fed our children DEAD STOCK DOWNER COWS, the most HIGH RISK CATTLE FOR MAD COW DISEASE, they fed them to our children, and hid the largest beef recall in USA history, hid this recall under the guise of 'animal abuse'. then told the parents not to worry because none of the kids had been sick or died from it to date. CJD can incubate for decades. anyway, just wanted to tell you about this, and thank you for what you are trying to do...

WHO WILL WATCH THE CHILDREN FOR CJD OVER THE NEXT 5 + DECADES ???

Do you actually believe that the USDA et al jumped in on the law suit against Westland/Hallmark, at the time the largest beef recall in USA history, just because a few animals were abused on a video, or to cover their ass, for letting our children, from school district to school district, from state to state, be fed dead stock downer cows.

>>> In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks... <<<

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

Do you actually believe all these schools recalled this meat because of a few cattle being abused, see list ; FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114

IF url does not work above, go to this link to find out if any of your children and their school were part of this recall ; go to this site ;

http://www.fns.usda.gov/fns/

left hand corner search ;

Hallmark/Westland Meat Packing Co. Beef Recall

you should get this ;

http://65.216.150.153/texis/search?pr=FNS

1 through 1 of 1 matching documents, best matches first. sort by date 1: Hallmark - Westland SFA Reporting by State - 3-24-2008.xls Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 The U.S. Department of Agriculture ...

http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114

PLEASE SEE ALSO ;

Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

PLEASE NOTE *

Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME.

snip...

The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

PLEASE be aware, for 4 years, the USDA fed our children all across the Nation dead stock downer cows, the most high risk cattle for BSE aka mad cow disease and other dangerous pathogens. who will watch our children for CJD for the next 5+ decades ???

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

2010

ALABAMA MAD COW g-h-BSEalabama

In this study, we identified a novel mutation in the bovine prion protein gene (Prnp), called E211K, of a confirmed BSE positive cow from Alabama, United States of America. This mutation is identical to the E200K pathogenic mutation found in humans with a genetic form of CJD. This finding represents the first report of a confirmed case of BSE with a potential pathogenic mutation within the bovine Prnp gene. We hypothesize that the bovine Prnp E211K mutation most likely has caused BSE in "the approximately 10-year-old cow" carrying the E221K mutation.

http://www.plospathogens.org/article/info%3Adoi%2F10.1371%2Fjournal.ppat.1000156

http://www.plospathogens.org/article/fetchObjectAttachment.action?uri=info%3Adoi%2F10.1371%2Fjournal.ppat.1000156&representation=PDF

Saturday, August 14, 2010

BSE Case Associated with Prion Protein Gene Mutation (g-h-BSEalabama) and VPSPr PRIONPATHY

(see mad cow feed in COMMERCE IN ALABAMA...TSS)

http://prionpathy.blogspot.com/2010/08/bse-case-associated-with-prion-protein.html

g-h-BSE-alabama E211K mad cows USA how many would that be annually ???

if our ciphering is correct (?), that would be about 35 g-h-BSE-alabama E211K mad cows going into the food chain a year.

an incidence of less than 1 in 2000.

let's see, that's 500 such per million.

or 50,000 cows per 100 million (US herd size).

even at less than 1 in a million, with 35 million slaughtered, that's 35 infected cows going into the food chain each year.

hmmm, friendly fire there from ???

Wednesday, July 28, 2010

re-Freedom of Information Act Project Number 3625-32000-086-05, Study of Atypical BSE UPDATE July 28, 2010

http://bse-atypical.blogspot.com/2010/07/re-freedom-of-information-act-project.html

Tuesday, August 03, 2010

Variably protease-sensitive prionopathy: A new sporadic disease of the prion protein

http://creutzfeldt-jakob-disease.blogspot.com/2010/08/variably-protease-sensitive-prionopathy.html

Monday, August 9, 2010

Variably protease-sensitive prionopathy: A new sporadic disease of the prion protein or just more PRIONBALONEY ?

http://prionunitusaupdate2008.blogspot.com/2010/08/variably-protease-sensitive-prionopathy.html

Thursday, August 12, 2010

Seven main threats for the future linked to prions

http://prionpathy.blogspot.com/2010/08/seven-main-threats-for-future-linked-to.html

http://prionpathy.blogspot.com/

14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf

Wednesday, March 31, 2010

Atypical BSE in Cattle

http://bse-atypical.blogspot.com/2010/03/atypical-bse-in-cattle-position-post.html

Wednesday, August 18, 2010

Incidence of CJD Deaths Reported by CJD-SS in Canada as of July 31, 2010

http://creutzfeldt-jakob-disease.blogspot.com/2010/08/incidence-of-cjd-deaths-reported-by-cjd.html

Monday, August 9, 2010

National Prion Disease Pathology Surveillance Center Cases Examined (July 31, 2010)

(please watch and listen to the video and the scientist speaking about atypical BSE and sporadic CJD and listen to Professor Aguzzi)

http://prionunitusaupdate2008.blogspot.com/2010/08/national-prion-disease-pathology.html

WE MUST NEVER FORGET !

Sunday, May 18, 2008

BSE, CJD, and Baby foods (the great debate 1999 to 2005)

http://bseinquiry.blogspot.com/2008/05/bse-cjd-and-baby-foods-great-debate.html

TSS

MAD COW SLURRY FED TO SCHOOL CHILDREN AND OTHERS Sat, 27 Oct 2001

2010-05-23T19:15:00.000-07:00

Subject: MAD COW SLURRY FED TO SCHOOL CHILDREN AND OTHERS !

From: "Terry S. Singeltary Sr."

Reply-To: Bovine Spongiform Encephalopathy

Date: Sat, 27 Oct 2001 20:08:20 -0700

Content-Type: text/plain

Parts/Attachments: text/plain (114 lines)

Reply

######## Bovine Spongiform Encephalopathy #########

October 28 2001 BRITAIN

© Breeding trouble: companies that used slurry scraped from the bones of cattle, which has been linked to vCJD, the human form of mad cow disease, have now been named Photograph: Stone Leading food groups used 'mad cow' danger meats

Mark Macaskill

SOME of Britain's best-known food producers used mechanically recovered meat - the slaughterhouse slurry blamed for spreading the human form of mad cow disease - and supplied it to unwitting consumers.

The companies - including Bowyers, Batchelors and Dalepak - used the now-outlawed product in pies, burgers and sausages sold to thousands of schools, hospitals, corner shops and supermarkets.

People who ate such products are thought to be at greater risk of developing the fatal brain disease vCJD, the human form of BSE. This is because mechanically recovered meat (MRM) from cows is scraped from the bone and is likely to include tissue from the nervous system.

Publication of the company names will embarrass the Food Standards Agency (FSA), which has been criticised for its failure to track down the firms responsible. In August, scientists tasked by the government with investigating the spread of vCJD, complained that, despite repeated demands, the agency had still to come up with the names, six years after the request was first filed.

Despite the FSA's difficulties, The Sunday Times has been able to identify products that contained MRM by tracking forward from the companies that bought the machinery needed to produce it.

These include products from two firms - Bowyers and Dalepak - both now owned by Northern Foods, the food conglomerate chaired by Lord Haskins, the prime minister's adviser on rural affairs.

MRM was banned for use in food in 1995, when the links between BSE and vCJD began to emerge. At least 107 people have died of vCJD and a further six have the disease. The worst- case scenario in official forecasts is for 40,000 people in Britain to be infected.

Batchelors, the soup brand, confirmed last week that bovine MRM was used in "non-branded" soups and other products until 1992. Bovine MRM was also used by Bowyers, the sausage manufacturer, and Dalepak, which made the Ross range of burgers, steaks and grills, Northern Foods confirmed.

A company that produced and supplied MRM was Scotbeef, one of the UK's largest beef producers. Last week it admitted producing MRM from 1980 until 1984 and selling it on to smaller wholesalers.

MRM became popular in the mid-1970s, when machines that recovered residual flesh from carcasses became available. Bones were dropped into a chamber and "squeezed" by a hydraulic ram. The slurry was then forced through tiny holes that filtered out any large particles. The product was then used to "pad out" a range of foods.

Two MRM machines, the Protecon and the Hydro, were made in the Netherlands. A number of slaughterhouses and meat manufacturers bought them, including Scotbeef, Midland Meat Packers Ltd, one of Britain's largest abattoirs, and Chard Meat Company, which has since gone into liquidation.

Another company, Perimax, with plants in Scotland and England, was set up in 1976 specifically to produce MRM from pig, cattle and poultry carcasses.

Most MRM was produced from poultry and pigs, but a significant amount - estimated at 5% - came from cattle.

Last week Northern Foods admitted that Bowyers used bovine MRM in its economy sausages before it took the company over in 1984. It also revealed that Dalepak had used bovine MRM. "It has always been our policy not to use mechanically recovered meat. When we acquired Bowyers and Dalepak, the company moved swiftly to remove MRM, but this would have taken a few months to complete," said a spokesman for Northern Foods last week.

Midland Meat Packers, one of the largest producers of bovine MRM, and Newtech Ltd, based in Wisbech, Cambridgeshire, which distributed the Hydro machine, declined to comment.

An FSA spokesman said: "So far, our only contact with the meat industry has been through the representing bodies, but all manufacturers will be contacted when our research gets under way."

http://www.sunday-times.co.uk/news/

TSS

########### http://mailhost.rz.uni-karlsruhe.de/warc/bse-l.html ############

2009

WHO WILL WATCH THE CHILDREN FOR CJD OVER THE NEXT 5 + DECADES ???

Do you actually believe that the USDA et al jumped in on the law suit against Westland/Hallmark, at the time the largest beef recall in USA history, just because a few animals were abused on a video, or to cover their ass, for letting our children, from school district to school district, from state to state, be fed dead stock downer cows.

>>> In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks... <<<

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

Do you actually believe all these schools recalled this meat because of a few cattle being abused, see list ; FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114

IF url does not work above, go to this link to find out if any of your children and their school were part of this recall ; go to this site ;

http://www.fns.usda.gov/fns/

left hand corner search ; Hallmark/Westland Meat Packing Co. Beef Recall your should get this ;

http://65.216.150.153/texis/search?pr=FNS

1 through 1 of 1 matching documents, best matches first. sort by date 1: Hallmark - Westland SFA Reporting by State - 3-24-2008.xls Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 The U.S. Department of Agriculture ...

http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114

PLEASE SEE ALSO ;

Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

PLEASE NOTE *

Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME.

snip...

The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

PLEASE be aware, for 4 years, the USDA fed our children all across the Nation dead stock downer cows, the most high risk cattle for BSE aka mad cow disease and other dangerous pathogens. who will watch our children for CJD for the next 5+ decades ???

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

page 114 ;

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf

International Society for Infectious Diseases Web: http://www.isid.org/

please see full text ;

http://transmissiblespongiformencephalopathy.blogspot.com/

To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE. When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.

http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2

please see full text ;

Wednesday, March 31, 2010

Atypical BSE in Cattle

http://bse-atypical.blogspot.com/2010/03/atypical-bse-in-cattle-position-post.html

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html

Monday, October 19, 2009

Atypical BSE, BSE, and other human and animal TSE in North America Update October 2009

http://bse-atypical.blogspot.com/2009/10/atypical-bse-bse-and-other-human-and.html

CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER

>>> Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. <<<

Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas

Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas.She left 6 Kids and a Husband.The Purpose of this web is to give information in Spanish to the Hispanic community, and to all the community who want's information about this terrible disease.-

Physician Discharge Summary, Parkland Hospital, Dallas Texas

Admit Date: 12/29/2009

Discharge Date: 1/20/2010

Attending Provider: Greenberg, Benjamin Morris;

General Neurology Team: General Neurology Team Linda was a Hispanic female with no past medical history presents with 14 months of incresing/progressive altered mental status, generalized weakness, inability to walk, loss of appetite, inability to speak, tremor and bowel/blader incontinence. She was, in her usual state of health up until February, 2009, when her husbans notes that she began forgetting things like names and short term memories. He also noticed mild/vague personality changes such as increased aggression. In March, she was involved in a hit and run MVA,although she was not injured. The police tracked her down and ticketed her. At that time, her son deployed to Iraq with the Army and her husband assumed her mentation changes were due to stress over these two events. Also in March, she began to have weakness in her legs, making it difficult to walk. Over the next few months, her mentation and personality changes worsened, getting to a point where she could no longer recognized her children. She was eating less and less. She was losing more weight. In the last 2-3 months, she reached the point where she could not walk without an assist, then 1 month ago, she stopped talking, only making grunting/aggressive sounds when anyone came near her. She also became both bowel and bladder incontinent, having to wear diapers. Her '"tremor'" and body jerks worsened and her hands assumed a sort of permanent grip position, leading her family to put tennis balls in her hands to protect her fingers. The husband says that they have lived in Nebraska for the past 21 years. They had seen a doctor there during the summer time who prescribed her Seroquel and Lexapro, Thinking these were sx of a mood disorder. However, the medications did not help and she continued to deteriorate clinically. Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. The husband says that he does not know any fellow workers with a similar illness. He also says that she did not have any preceeding illness or travel.

http://www.recordandoalinda.com/index.php?option=com_content&view=article&id=19:cjd-english-info&catid=9:cjd-ingles&Itemid=8

>>> Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. <<<

please see full text ;

Monday, March 29, 2010 Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas

http://creutzfeldt-jakob-disease.blogspot.com/2010/03/irma-linda-andablo-cjd-victim-she-died.html

CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER

http://cjdtexas.blogspot.com/2010/03/cjd-texas-38-year-old-female-worked.html

USA sporadic CJD cases rising ;

There is a growing number of human CJD cases, and they were presented last week in San Francisco by Luigi Gambatti(?) from his CJD surveillance collection. He estimates that it may be up to 14 or 15 persons which display selectively SPRPSC and practically no detected RPRPSC proteins.

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/1006-4240t1.htm

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/2006-4240t1.pdf

CJD USA RISING, with UNKNOWN PHENOTYPE ; 5 Includes 41 cases in which the diagnosis is pending, and 17 inconclusive cases; 6 Includes 46 cases with type determination pending in which the diagnosis of vCJD has been excluded.

http://www.cjdsurveillance.com/pdf/case-table.pdf

Friday, February 05, 2010

New Variant Creutzfelt Jakob Disease case reports United States 2010 A Review

http://vcjd.blogspot.com/2010/02/new-variant-creutzfelt-jakob-disease.html

Saturday, January 2, 2010 Human Prion Diseases in the United States January 1, 2010 ***FINAL***

http://prionunitusaupdate2008.blogspot.com/2010/01/human-prion-diseases-in-united-states.html

Archive Number 20100405.1091 Published Date 05-APR-2010 Subject PRO/AH/EDR> Prion disease update 1010 (04)

snip...

[Terry S. Singeltary Sr. has added the following comment:

"According to the World Health Organisation, the future public health threat of vCJD in the UK and Europe and potentially the rest of the world is of concern and currently unquantifiable. However, the possibility of a significant and geographically diverse vCJD epidemic occurring over the next few decades cannot be dismissed.

The key word here is diverse. What does diverse mean? If USA scrapie transmitted to USA bovine does not produce pathology as the UK c-BSE, then why would CJD from there look like UK vCJD?"

http://www.promedmail.org/pls/apex/f?p=2400:1001:568933508083034::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1000,82101

Tuesday, March 2, 2010

Animal Proteins Prohibited in Ruminant Feed/Adulterated/Misbranded Rangen Inc 2/11/10 USA

http://madcowfeed.blogspot.com/2010/03/animal-proteins-prohibited-in-ruminant.html

Monday, March 1, 2010

ANIMAL PROTEIN I.E. MAD COW FEED IN COMMERCE A REVIEW 2010

http://madcowfeed.blogspot.com/2010/03/animal-protien-ie-mad-cow-feed-in.html

TSS

USDA SETS NEW STANDARDS FOR GROUND BEEF PURCHASES

2010-05-14T08:38:00.000-07:00

Release No. 0267.10

Contact: Michael Jarvis (202)720-8998 Michael.Jarvis@ams.usda.gov Billy Cox (202)720-8998 Billy.cox@ams.usda.gov

USDA SETS NEW STANDARDS FOR GROUND BEEF PURCHASES

Washington, May 14, 2010 -- Agriculture Secretary Tom Vilsack today announced that USDA had completed tough new food safety standards for ground beef purchased by the Agricultural Marketing Service (AMS) for Federal food and nutrition assistance programs including school lunches.

"The health of our school children is a top priority at USDA, and today we are moving ahead quickly with plans announced earlier this year to ensure that food provided to nutrition programs is as safe and nutritious as possible," Vilsack said. "The new standards announced today ensure our purchases are in line with major private-sector buyers of ground beef and are part of our continued effort to employ the best scientific knowledge to increase the safety of our nutritional programs."

This past February, Vilsack announced a series of initiatives to improve the safety of food purchased for school lunch and nutrition assistance programs, and standards announced today marks the completion of one of those initiatives. The standards are the result of a joint review by the USDA's Food Safety and Inspection Service (FSIS) and Agricultural Research Service (ARS) that has been ongoing since the February announcement.

The new requirements will be applicable to AMS ground beef contracts awarded on or after July 1, 2010.

In addition to continuing a zero tolerance for E. coli O157:H7 and Salmonella, the new AMS standards will: (1) tighten microbiological testing protocols; (2) tighten the microbiological upper specification and critical limits; (3) increase microbiological sampling frequency for finished products to every 15 minutes; and, (4) institute additional rejection criteria for source trimmings used to manufacture AMS purchased ground beef. AMS will also consider any vendor classified by FSIS as having a long term poor safety record as an ineligible vendor until a complete cause-and-effect analysis is completed.

The National Academy of Sciences (NAS) has begun its review of AMS ground beef purchase requirements. NAS brings together experts in all areas of science to provide an independent review of the program. The review is focused on three major areas: 1) a thorough evaluation of the scientific validity of current technical requirements and methods; 2) to benchmark those processes and methods against recognized industry leading programs, which supply product directly to consumers through retail sales or food service operations; and 3) provide recommendations to AMS on how to perform future periodic evaluations against industry recognized best practices.

--------------------------------------------------------------------------------

USDA is an equal opportunity provider, employer and lender. To file a complaint of discrimination, write: USDA, Director, Office of Civil Rights, 1400 Independence Ave., SW, Washington, DC 20250-9410 or call (800) 795-3272 (voice), or (202) 720-6382 (TDD).

http://www.usda.gov/wps/portal/usda/!ut/p/c4/04_SB8K8xLLM9MSSzPy8xBz9CP0os_gAC9-wMJ8QY0MDpxBDA09nXw9DFxcXQ-cAA_2CbEdFAEUOjoE!/?contentidonly=true&contentid=2010%2F05%2F0267.xml

Greetings,

Considering what the NSLP and the USDA have done in the past, I would welcome any new standard. So I applaud these new standards.

However, feeding those dead stock downer cows to children for over 4 years was something they should never ever be able to forget about. dead stock downer cows are the most high risk animal for mad cow disease, and they knew this when the feeding to our children of this product was taking place. the USDA et al via the NSLP fed our children all across the USA, from state to state, the largest beef recall in history, they fed our children DEAD STOCK DOWNER COWS, the most HIGH RISK CATTLE FOR MAD COW DISEASE, they fed them to our children, and hid the largest beef recall in USA history, hid this recall under the guise of 'animal abuse'. then told the parents not to worry because none of the kids had been sick or died from it to date. CJD can incubate for decades. anyway, just wanted to tell you about this, and thank you for what you are trying to do...

WHO WILL WATCH THE CHILDREN FOR CJD OVER THE NEXT 5 + DECADES ???

Do you actually believe that the USDA et al jumped in on the law suit against Westland/Hallmark, at the time the largest beef recall in USA history, just because a few animals were abused on a video, or to cover their ass, for letting our children, from school district to school district, from state to state, be fed dead stock downer cows.

>>> In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks... <<<

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

Do you actually believe all these schools recalled this meat because of a few cattle being abused,

see list ; FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114

IF url does not work above, go to this link to find out if any of your children and their school were part of this recall ; go to this site ;

http://www.fns.usda.gov/fns/

left hand corner search ; Hallmark/Westland Meat Packing Co. Beef Recall your should get this ;

http://65.216.150.153/texis/search?pr=FNS

1 through 1 of 1 matching documents, best matches first. sort by date 1: Hallmark - Westland SFA Reporting by State - 3-24-2008.xls Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 The U.S. Department of Agriculture ...

http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=Hallmark/Westland+Meat+Packing+Co.+Beef+Recall&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4bc8d6e114

PLEASE SEE ALSO ;

Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

PLEASE NOTE *

Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME.

snip...

The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

PLEASE be aware, for 4 years, the USDA fed our children all across the Nation dead stock downer cows, the most high risk cattle for BSE aka mad cow disease and other dangerous pathogens. who will watch our children for CJD for the next 5+ decades ???

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

page 114 ;

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf

International Society for Infectious Diseases Web: http://www.isid.org/

please see full text ;

http://transmissiblespongiformencephalopathy.blogspot.com/

To date the OIE/WAHO assumes that the human and animal health standards set out in the BSE chapter for classical BSE (C-Type) applies to all forms of BSE which include the H-type and L-type atypical forms. This assumption is scientifically not completely justified and accumulating evidence suggests that this may in fact not be the case. Molecular characterization and the spatial distribution pattern of histopathologic lesions and immunohistochemistry (IHC) signals are used to identify and characterize atypical BSE. Both the L-type and H-type atypical cases display significant differences in the conformation and spatial accumulation of the disease associated prion protein (PrPSc) in brains of afflicted cattle. Transmission studies in bovine transgenic and wild type mouse models support that the atypical BSE types might be unique strains because they have different incubation times and lesion profiles when compared to C-type BSE. When L-type BSE was inoculated into ovine transgenic mice and Syrian hamster the resulting molecular fingerprint had changed, either in the first or a subsequent passage, from L-type into C-type BSE. In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.

http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2

please see full text ;

Wednesday, March 31, 2010

Atypical BSE in Cattle

http://bse-atypical.blogspot.com/2010/03/atypical-bse-in-cattle-position-post.html

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html

Monday, October 19, 2009

Atypical BSE, BSE, and other human and animal TSE in North America Update October 2009

http://bse-atypical.blogspot.com/2009/10/atypical-bse-bse-and-other-human-and.html

CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER

>>> Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. <<<

Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas.She left 6 Kids and a Husband.The Purpose of this web is to give information in Spanish to the Hispanic community, and to all the community who want's information about this terrible disease.-

Physician Discharge Summary, Parkland Hospital, Dallas Texas Admit Date: 12/29/2009

Discharge Date: 1/20/2010

Attending Provider: Greenberg, Benjamin Morris; General Neurology Team: General Neurology Team

Linda was a Hispanic female with no past medical history presents with 14 months of incresing/progressive altered mental status, generalized weakness, inability to walk, loss of appetite, inability to speak, tremor and bowel/blader incontinence.She was, in her usual state of health up until February, 2009, when her husbans notes that she began forgetting things like names and short term memories. He also noticed mild/vague personality changes such as increased aggression. In March, she was involved in a hit and run MVA,although she was not injured. The police tracked her down and ticketed her. At that time, her son deployed to Iraq with the Army and her husband assumed her mentation changes were due to stress over these two events. Also in March, she began to have weakness in her legs, making it difficult to walk. Over the next few months, her mentation and personality changes worsened, getting to a point where she could no longer recognized her children. She was eating less and less. She was losing more weight. In the last 2-3 months, she reached the point where she could not walk without an assist, then 1 month ago, she stopped talking, only making grunting/aggressive sounds when anyone came near her. She also became both bowel and bladder incontinent, having to wear diapers. Her '"tremor'" and body jerks worsened and her hands assumed a sort of permanent grip position, leading her family to put tennis balls in her hands to protect her fingers. The husband says that they have lived in Nebraska for the past 21 years. They had seen a doctor there during the summer time who prescribed her Seroquel and Lexapro, Thinking these were sx of a mood disorder. However, the medications did not help and she continued to deteriorate clinically. Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. The husband says that he does not know any fellow workers with a similar illness. He also says that she did not have any preceeding illness or travel.

http://www.recordandoalinda.com/index.php?option=com_content&view=article&id=19:cjd-english-info&catid=9:cjd-ingles&Itemid=8

>>> Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. <<<

please see full text ; Monday, March 29, 2010

Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas

http://creutzfeldt-jakob-disease.blogspot.com/2010/03/irma-linda-andablo-cjd-victim-she-died.html

CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER

http://cjdtexas.blogspot.com/2010/03/cjd-texas-38-year-old-female-worked.html

USA sporadic CJD cases rising ; There is a growing number of human CJD cases, and they were presented last week in San Francisco by Luigi Gambatti(?) from his CJD surveillance collection. He estimates that it may be up to 14 or 15 persons which display selectively SPRPSC and practically no detected RPRPSC proteins.

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/1006-4240t1.htm

http://www.fda.gov/ohrms/dockets/ac/06/transcripts/2006-4240t1.pdf

CJD USA RISING, with UNKNOWN PHENOTYPE ; 5 Includes 41 cases in which the diagnosis is pending, and 17 inconclusive cases; 6 Includes 46 cases with type determination pending in which the diagnosis of vCJD has been excluded.

http://www.cjdsurveillance.com/pdf/case-table.pdf

Friday, February 05, 2010

New Variant Creutzfelt Jakob Disease case reports United States 2010 A Review

http://vcjd.blogspot.com/2010/02/new-variant-creutzfelt-jakob-disease.html

Saturday, January 2, 2010 Human Prion Diseases in the United States January 1, 2010 ***FINAL***

http://prionunitusaupdate2008.blogspot.com/2010/01/human-prion-diseases-in-united-states.html

Archive Number 20100405.1091 Published Date 05-APR-2010 Subject PRO/AH/EDR> Prion disease update 1010 (04)

snip...

[Terry S. Singeltary Sr. has added the following comment:

"According to the World Health Organisation, the future public health threat of vCJD in the UK and Europe and potentially the rest of the world is of concern and currently unquantifiable. However, the possibility of a significant and geographically diverse vCJD epidemic occurring over the next few decades cannot be dismissed.

The key word here is diverse. What does diverse mean? If USA scrapie transmitted to USA bovine does not produce pathology as the UK c-BSE, then why would CJD from there look like UK vCJD?"

http://www.promedmail.org/pls/apex/f?p=2400:1001:568933508083034::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1000,82101

see mad cow feed ban violations ;

Monday, March 8, 2010 UPDATE 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009 Greetings,

I got a follow on this in the mail this past Saturday in the mail. thought some might be interested in the following ;

DEPARTMENT OF HEALTH & HUMAN SERVICES Public Health Service Food and Drug Administration Rockville MD 20857

Terry Singeltary P.O. box 42. Bacliff, TX USA 77518

Dear Requestor

In reply refer to: F2009-7430

http://madcowfeed.blogspot.com/2010/03/update-429128-lbs-feed-for-ruminant.html

Tuesday, March 2, 2010

Animal Proteins Prohibited in Ruminant Feed/Adulterated/Misbranded Rangen Inc 2/11/10 USA

http://madcowfeed.blogspot.com/2010/03/animal-proteins-prohibited-in-ruminant.html

Monday, March 1, 2010

ANIMAL PROTEIN I.E. MAD COW FEED IN COMMERCE A REVIEW 2010

http://madcowfeed.blogspot.com/2010/03/animal-protien-ie-mad-cow-feed-in.html

TEST ! TEST ! TEST !

TEST all food producing animals for humans and animal for all strains of Transmissible Spongiform Encephalopathy, and they will come $$$

Friday, August 29, 2008

CREEKSTONE VS USDA COURT OF APPEALS, BUSH SAYS, NO WAY, NO HOW

http://madcowtesting.blogspot.com/2008/08/creekstone-vs-usda-court-of-appeals.html

BUT, THE TESTING MUST BE DONE CORRECTLY ;

Owner and Corporation Plead Guilty to Defrauding Bovine Spongiform Encephalopathy (BSE) Surveillance Program

An Arizona meat processing company and its owner pled guilty in February 2007 to charges of theft of Government funds, mail fraud, and wire fraud. The owner and his company defrauded the BSE Surveillance Program when they falsified BSE Surveillance Data Collection Forms and then submitted payment requests to USDA for the services. In addition to the targeted sample population (those cattle that were more than 30 months old or had other risk factors for BSE), the owner submitted to USDA, or caused to be submitted, BSE obex (brain stem) samples from healthy USDA-inspected cattle. As a result, the owner fraudulently received approximately $390,000. Sentencing is scheduled for May 2007.

snip...

Topics that will be covered in ongoing or planned reviews under Goal 1 include:

soundness of BSE maintenance sampling (APHIS),

implementation of Performance-Based Inspection System enhancements for specified risk material (SRM) violations and improved inspection controls over SRMs (FSIS and APHIS),

snip...

The findings and recommendations from these efforts will be covered in future semiannual reports as the relevant audits and investigations are completed.

4 USDA OIG SEMIANNUAL REPORT TO CONGRESS FY 2007 1st Half

http://www.usda.gov/oig/webdocs/sarc070619.pdf

snip... please see full text ;

http://bse-atypical.blogspot.com/2008/06/mad-cows-and-computer-models-us.html

----- Original Message -----

From: Williams, Monica

To: flounder9@verizon.net

Cc: Williams, Monica

Sent: Wednesday, May 12, 2010 7:21 AM

Subject: Freedom of Information Act Request

Terry Singeltary Sr.:

This is to acknowledge receipt of your May 10, 2010, Freedom of Information Act (FOIA) request concerning the results for Research Project Number: 3625-32000-086-05, Study of Atypical BSE at the Virus and Prion Research Unit. Your request was received in this office on May 11, 2010, and assigned FOIA No. 10-93, with a response date of June 9, 2010.

If you have any questions, concerning the status of your request, please contact this office at 301-504-1640 or by e-mail at reefoia@ars.usda.gov.

Monica Williams

FOIA Office, REE USDA

5601 Sunnyside Avenue

Room 1-2226C, Mail Stop 5128

Beltsville, MD 20705-5128

reefoia@ars.usda.gov

Telephone: 301-504-1640

Facsimile: 301-504-1647

From: Terry S. Singeltary Sr. [mailto:flounder9@verizon.net]

Sent: Wednesday, May 05, 2010 5:29 PM

To: USDAfoia@da.usda.gov

Cc: phyllis.fong@oig.usda.gov; FOIASTAFF@oig.usda.gov; Public Affairs; Tips, HHS

Subject: F.O.I.A. re-Publication Request Project Number: 3625-32000-086-05 Study of Atypical Bse

Greetings OIE USDA FOIA et al;

I have made numerous attempts at finding the results of the following study with absolutely no luck or reply.

So I suppose I must make a F.O.I.A. request on the following ;

re-F.O.I.A. re-Publication Request Project Number: 3625-32000-086-05 Study of Atypical Bse

----- Original Message -----

From: flounder9@verizon.net

To: webmaster@ars.usda.gov

Cc: flounder9@verizon.net

Sent: Tuesday, May 04, 2010 6:43 AM

Subject: From ARS Web Site: PUBLICATION REQUEST

Message:

Greetings,

Publication Request Project Number: 3625-32000-086-05 Study of Atypical Bse

the study below said that the end date was Sep 14, 2009.

can you now tell me please what those results were, and if it will be necessary to change SRM removal due to any different tissue infectivity distribution ?

could you please supply me with the results of this study ?

Research Project: Study of Atypical Bse Location: Virus and Prion Research Unit Project Number: 3625-32000-086-05

Project Type: Specific Cooperative Agreement Start Date: Sep 15, 2004 End Date: Sep 14, 2009

Objective: The objective of this cooperative research project with Dr. Maria Caramelli from the Italian BSE Reference Laboratory in Turin, Italy, is to conduct comparative studies with the U.S. bovine spongiform encephalopathy (BSE) isolate and the atypical BSE isolates identified in Italy. The studies will cover the following areas: 1. Evaluation of present diagnostics tools used in the U.S. for the detection of atypical BSE cases. 2. Molecular comparison of the U.S. BSE isolate and other typical BSE isolates with atypical BSE cases. 3. Studies on transmissibility and tissue distribution of atypical BSE isolates in cattle and other species. Approach: This project will be done as a Specific Cooperative Agreement with the Italian BSE Reference Laboratory, Istituto Zooprofilattico Sperimentale del Piemonte, in Turin, Italy. It is essential for the U.S. BSE surveillance program to analyze the effectiveness of the U.S diagnostic tools for detection of atypical cases of BSE. Molecular comparisons of the U.S. BSE isolate with atypical BSE isolates will provide further characterization of the U.S. BSE isolate. Transmission studies are already underway using brain homogenates from atypical BSE cases into mice, cattle and sheep. It will be critical to see whether the atypical BSE isolates behave similarly to typical BSE isolates in terms of transmissibility and disease pathogenesis. If transmission occurs, tissue distribution comparisons will be made between cattle infected with the atypical BSE isolate and the U.S. BSE isolate. Differences in tissue distribution could require new regulations regarding specific risk material (SRM) removal.

http://www.ushrl.saa.ars.usda.gov/research/projects/projects.htm?accn_no=408490

Greetings again O.I.G. et al ;

I find it disturbing that we are still floundering with public health in 2010 on this issue, especially since we know that ;

>>> non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures. <<<

BUT yet the USDA et al is still floundering on these results, and still base trade on these OIE standards that in all reality, put the cart before the horse in regards to the legal trading of these atypical BSE and scrapie strains, before any transmission studies were done that would prove the same thing that happened in the U.K. with c-BSE and nvCJD, would not happen again.

http://www.prionetcanada.ca/detail.aspx?menu=5&dt=293380&app=93&cat1=387&tp=20&lk=no&cat2

14th International Congress on Infectious Diseases H-type and L-type Atypical BSE

January 2010 (special pre-congress edition)

18.173 page 189

Experimental Challenge of Cattle with H-type and L-type Atypical BSE

A. Buschmann1, U. Ziegler1, M. Keller1, R. Rogers2, B. Hills3, M.H. Groschup1. 1Friedrich-Loeffler-Institut, Greifswald-Insel Riems, Germany, 2Health Canada, Bureau of Microbial Hazards, Health Products & Food Branch, Ottawa, Canada, 3Health Canada, Transmissible Spongiform Encephalopathy Secretariat, Ottawa, Canada

Background: After the detection of two novel BSE forms designated H-type and L-type atypical BSE the question of the pathogenesis and the agent distribution of these two types in cattle was fully open. From initial studies of the brain pathology, it was already known that the anatomical distribution of L-type BSE differs from that of the classical type where the obex region in the brainstem always displays the highest PrPSc concentrations. In contrast in L-type BSE cases, the thalamus and frontal cortex regions showed the highest levels of the pathological prion protein, while the obex region was only weakly involved.

Methods:We performed intracranial inoculations of cattle (five and six per group) using 10%brainstemhomogenates of the two German H- and L-type atypical BSE isolates. The animals were inoculated under narcosis and then kept in a free-ranging stable under appropriate biosafety conditions.

At least one animal per group was killed and sectioned in the preclinical stage and the remaining animals were kept until they developed clinical symptoms. The animals were examined for behavioural changes every four weeks throughout the experiment following a protocol that had been established during earlier BSE pathogenesis studies with classical BSE.

Results and Discussion:

All animals of both groups developed clinical symptoms and had to be euthanized within 16 months. The clinical picture differed from that of classical BSE, as the earliest signs of illness were loss of body weight and depression. However, the animals later developed hind limb ataxia and hyperesthesia predominantly and the head. Analysis of brain samples from these animals confirmed the BSE infection and the atypical Western blot profile was maintained in all animals. Samples from these animals are now being examined in order to be able to describe the pathogenesis and agent distribution for these novel BSE types.

Conclusions: A pilot study using a commercially avaialble BSE rapid test ELISA revealed an essential restriction of PrPSc to the central nervous system for both atypical BSE forms. A much more detailed analysis for PrPSc and infectivity is still ongoing.

http://www.isid.org/14th_icid/

http://ww2.isid.org/Downloads/IMED2009_AbstrAuth.pdf

http://www.isid.org/publications/ICID_Archive.shtml

14th ICID International Scientific Exchange Brochure - Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America update October 2009

T. Singeltary

Bacliff, TX, USA

Background: An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods: 12 years independent research of available data

Results: I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion: I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

see page 114 ;

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf

Sent: Friday, April 16, 2010 11:38 AM

Subject: PRO-MED ATYPICAL SCRAPIE

Background ----------- "Retrospective studies have identified cases predating the initial identification of this form of scrapie, and epidemiological studies have indicated that it does not conform to the behaviour of an infectious disease, giving rise to the hypothesis that it represents spontaneous disease. However, atypical scrapie isolates have been shown to be infectious experimentally, through intracerebral inoculation in transgenic mice and sheep. [Many of the neurological diseases can be transmitted by intracerebral inoculation, which causes this moderator to approach intracerebral studies as a tool for study, but not necessarily as a direct indication of transmissibility of natural diseases. - Mod.TG] "The 1st successful challenge of a sheep with 'field' atypical scrapie from an homologous donor sheep was reported in 2007.

"Results -------- "This study demonstrates that atypical scrapie has distinct clinical, pathological, and biochemical characteristics which are maintained on transmission and sub-passage, and which are distinct from other strains of transmissible spongiform encephalopathies in the same host genotype.

"Conclusions ------------ Atypical scrapie is consistently transmissible within AHQ homozygous sheep, and the disease phenotype is preserved on sub-passage." Lastly, this moderator wishes to thank Terry Singletary for some of his behind the scenes work of providing citations and references for this posting. - Mod.TG]

The HealthMap/ProMED-mail interactive map of Australia is available at . - Sr.Tech.Ed.MJ]

http://www.promedmail.org/pls/otn/f?p=2400:1001:962575216785367::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1000,81729

Archive Number 20100405.1091 Published Date 05-APR-2010

Subject PRO/AH/EDR> Prion disease update 1010 (04)

snip...

[Terry S. Singeltary Sr. has added the following comment:

"According to the World Health Organisation, the future public health threat of vCJD in the UK and Europe and potentially the rest of the world is of concern and currently unquantifiable. However, the possibility of a significant and geographically diverse vCJD epidemic occurring over the next few decades cannot be dismissed.

The key word here is diverse. What does diverse mean? If USA scrapie transmitted to USA bovine does not produce pathology as the UK c-BSE, then why would CJD from there look like UK vCJD?"

http://www.promedmail.org/pls/apex/f?p=2400:1001:568933508083034::NO::F2400_P1001_BACK_PAGE,F2400_P1001_PUB_MAIL_ID:1000,82101

Monday, March 29, 2010

CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER

>>> Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. <<<

http://recordandoalinda.com/index.php?option=com_content&view=article&id=19&Itemid=8

http://www.recordandoalinda.com/index.php?option=com_content&view=article&id=2:frontpage&catid=1:frontpage

Wednesday, April 28, 2010

BSE, Scrapie, CWD, REPORT OF THE MEETING OF THE OIE TERRESTRIAL ANIMAL HEALTH STANDARDS COMMISSION Paris, 8-12 February 2010

REPORT OF THE MEETING OF THE OIE TERRESTRIAL ANIMAL

http://usdameatexport.blogspot.com/2010/04/bse-scrapie-cwd-report-of-meeting-of.html

Thank You!

Kindest Regards,

terry

Terry S. Singeltary Sr.

P.O. Box 42

Bacliff, Texas USA 77518

Whistleblower Testifies Against USDA

2010-03-06T09:27:00.000-08:00

March 5, 2010

Whistleblower Testifies Against USDA

Dr. Dean Wyatt says the USDA retaliated against him for trying to enforce the Humane Methods of Slaughter Act

“Food integrity and humane handling whistleblowers should not have to rely on an undercover video investigation in order for USDA supervisors to take their disclosures seriously.”—Dr. Wyatt

In testimony before a House subcommittee hearing on March 4, Dr. Dean Wyatt, public health veterinarian for the Food Safety and Inspection Service (FSIS) of the USDA, expressed frustration about the USDA’s lack of support for inspectors who are “just trying to do their job” to protect animal welfare and food safety.

In his testimony, Dr. Wyatt said USDA officials not only overturned his recommendations, siding with slaughter plants in violation of federal law, but also personally retaliated against him for trying to enforce the law. Dr. Wyatt, who has been with the USDA for 18 years, was reprimanded and threatened with termination by his supervisors after reporting repeated violations of the USDA’s Humane Methods of Slaughter Act (HMSA).

Wyatt spoke about suspending operations at the Bushway Packing slaughter plant in Vermont on three occasions for improper handling of animals; each time district officials for the Agency reopened the plant and allowed it to continue without addressing humane handling concerns. Violations included dragging a “downed” calf unable to stand off a truck by the hind leg and dragging a days-old calf through a holding pen. In another instance, Wyatt described an Agency official changing the wording in a written report to suggest an animal had been “dropped” rather than thrown off of a truck.

After an undercover video investigation was conducted by The HSUS at the Bushway Packing plant, the USDA closed the plant for violations of the HMSA. A criminal investigation into conduct at the slaughterhouse is underway.

Thursday’s hearing coincided with the release by the Government Accountability Office (GAO) of a report that encouraged stronger enforcement of the HMSA. Specific recommendations included establishing “clear and specific” criteria for suspending plant operations; improving standards for compliance; and taking a closer look at noncompliance reports.

Under the HMSA, FSIS inspectors are charged with overseeing humane handling of animals and with conducting fecal contamination checks to prevent contaminated meat from entering the food supply. Animals too sick to stand or walk are prohibited from entering the food supply due to the potential threat of mad cow disease.

In 2008, an HSUS investigation into the processing of downed cows at the Hallmark Meat Packing slaughter plant in Chino, Calif. led the USDA to close the plant and recall 143 million pounds of beef. The Chino plant was the second largest supplier of beef to the nation’s school lunch program.

Stanley Painter, representing the union of government food inspection workers (National Joint Council of Food Inspection Local Unions), testified that he concurred with Dr. Wyatt’s assessment that the USDA did not provide adequate support or training for inspectors at the nation’s slaughter plants.

Speaking before the subcommittee, Wayne Pacelle, president and CEO of The Humane Society of the United States, said he was heartened by the new Administration’s willingness to take humane concerns seriously. He went on to say that slaughter plant violations are chronic: “Every time we have looked, we have found problems.”

Pacelle recommended that the USDA create its own mobile investigations unit and enforce a policy of “zero percent tolerance” for abuses and inhumane handling, citing the billions of animals and hundreds of millions of consumers who rely on the USDA for proper enforcement of the HMSA.

Pacelle also called on the USDA to close the loophole on downer calves. The Obama Administration closed the loophole on downer cows following The HSUS’s Hallmark investigation, but a loophole that applies to young animals still exists.

In his closing remarks, subcommittee chairman Dennis Kucinich thanked Dr. Wyatt for his public service and his courage to blow the whistle on the USDA, as well as The HSUS for sharing the results of its undercover investigations. Citing its conflicts of interest, Kucinich encouraged the USDA to do more to protect consumers and promised to continue to press for changes that will lead to greater consumer confidence in the USDA.

http://humanesociety.org/news/news/2010/whistleblower_reports_USDA_failures.html

Humane Methods of Slaughter Act: Actions Are Needed to Strengthen Enforcement.

GAO-10-203, February 19

http://www.gao.gov/cgi-bin/getrpt?GAO-10-203

Highlights -

http://www.gao.gov/highlights/d10203high.pdf

OTHER WRITTEN PRODUCT

Humane Methods of Slaughter Act: USDA Inspectors' Views on Enforcement (GAO-10-244SP), February 2010, an E-supplement to GAO-10-203.

GAO-10-244SP, February 19

http://www.gao.gov/cgi-bin/getrpt?GAO-10-244SP

TESTIMONY

Humane Methods of Slaughter Act: Weaknesses in USDA Enforcement.

GAO-10-487T, March 4

http://www.gao.gov/cgi-bin/getrpt?GAO-10-487T

TAFS

How are cattle stunned?

In recent times cattle would be stunned by means of a gun, from which a bolt was driven through the skull and into the brain. This renders the animal unconscious. In some abattoirs, penetrative stunning would be followed by “pithing”. This involves passing a flexible rod through the hole in the skull, and down the spinal canal. It is, or was, done primarily to protect the operators responsible for bleeding the cattle. It did so by destroying spinal reflexes controlling the front limbs, and prevented reflex kicking. In larger abattoirs with greater mechanisation of procedures, more rapid stunning and bleeding operations frequently make this an unnecessary procedure. Smaller abattoirs, with greater dependence on manual labour, and slower speeds of operation, may still find pithing to be necessary. Pithing has actually been banned in Europe.

A variant of this method was used in some countries. In this method the flexible rod was replaced by the injection of air through the bolt and stun hole into the skull. This effectively pithed the animal. This is now illegal in the European Union.

An alternative form of stunning, called percussion stunning, applies the stun to the outside of the skull without penetrating the skull.

In some abattoirs electrical stunning is used. In other words the cattle may be electrocuted.

How can stunning increase risk to consumers from BSE?

There is some research evidence that suggests that penetrative stunning, with or without pithing, can cause some brain tissue to enter the blood stream. Although such a risk was demonstrated many years ago by artificially contaminating the stun bolt or pithing rod with bacteria, recent studies have specifically demonstrated trace amounts of brain material in the blood of some animals immediately following stunning(2-5, 8, 9, 10, 12, 21, 24).

The injection of air through the stun bolt has the potential to significantly increase the risk. Occasionally, samples of brain material, visible to the naked eye, have been identified lodged in tissues that receive blood after passing through the heart (usually lodged in lung or occasionally liver)(14, 15, 20, 27).

In these circumstances, the methods of stunning have the potential if used on BSE-infected animals, to drive some infectivity into the blood stream. The extent of contamination of the rest of the carcase by this means has not been demonstrated. As mentioned above, with the exception of extremely small particles of brain, most would become lodged in organs such as the lung.

Penetrative stunning also has the potential to expose the meat of the head (and indeed operators) to brain tissue that exudes from the hole, either on the pithing rod or subsequently after removal of the head.

Carriage of heads to specialist plants where the meat from the head is removed (boning-out plants) can compound the likelihood of contamination if several heads are in contact. Following removal of the head, leakage of cerebro-spinal fluid, which bathes the brain and has not been shown to be infectious in BSE cases, has the theoretical potential to cause further cross-contamination.

TAFS

Is it only the stunning process that represents a risk?

No. In the majority of abattoirs it is normal practice to split the carcase in order make handling easier, and to supply traditional cuts of meats to the retail and manufacturing trade. This is also required to ensure that the whole carcase can be inspected to ensure that it is fit for human consumption. Splitting normally involves sawing the spinal column in half along its length. This also frequently cuts through the spinal cord. If the animal is infected with BSE, the spinal cord must be assumed to be as infectious as the brain(7, 11, 13, 16, 17, 19, 21, 22).

It is therefore inevitable that a certain amount of spinal cord tissue is spread along the cut surface of the spinal column by the saw. In addition there will be a certain amount of spray contamination of the surface of the carcase, but research into this aspect is ongoing(6, 11, 18, 25, 28).

14. Garland, T, Bauer, N. & Bailey, M.Jr. (1996). Brain emboli in the lungs of cattle after stunning (letter). Lancet. 348: 610.

15. Garland, Tam. (1996). Brain emboli in the lungs of cattle (reply to K C Taylor). Lancet. 348: 749.

20. Munro R. (1997). Neural tissue embolism in cattle. Veterinary Record. 140, N20, 536.

21. Prendergast, D.M., Sheridan, J.J., Daly, D.J., McDowell, D.A. & Blair, I.S. (2003). Dissemination of central nervous system tissue from the brain and spinal cord of cattle after captive bolt stunning and carcass splitting. Meat Science. 65:1201-1209.

27. Taylor, K.C. (1996). Brain emboli in the lungs of cattle. Lancet. 348: 749

http://www.tseandfoodsafety.org/position_papers/TAFS_POSITION_PAPER_ON%20SLAUGHTER%202009.pdf

WHY is it that some of you folks are more worried about animal abuse (which i do not condone), but yet you will allow your children, all across the Nation, be fed the most high risk cattle for mad cow disease and other dangerous pathogens via the NSLP. For over 4 years, dead stock downer cows were fed to our children all across the Nation. But yet it seems that most are more concerned about how the animals are treated before slaughter. What about our children ? WE apparently just turn our heads on a disease that is in THE USA, has been, still is, sCJD rising, this is disease is of long incubation, but yet once clinical, it is 100% fatal. who will watch our children over the next 5 decades for CJD, or does anyone care ?

The PrP TSE mad cow agent in humans can incubate up to 50+ years in some cases, in other cases, not so long. so, come back in 50+ years and confirm this. junk science, industry friendly regulations, and or just not complying with existing regulations have been rampant over the past 12 years I have been paying attention, it has been the norm. maybe sound science will prevail in the end, maybe not. but feeding children diseased and sick cows via the NSLP was flat wrong, and anyone that thinks the largest beef recall there from, was just because a few animals were abused, well, they too are just flat wrong as well. ...

WHO WILL WATCH THE CHILDREN FOR CJD OVER THE NEXT 5 + DECADES ???

Do you actually believe that the USDA et al jumped in on the law suit against Westland/Hallmark, at the time the largest beef recall in USA history, just because a few animals were abused on a video, or to cover their ass, for letting our children, from school district to school district, from state to state, be fed dead stock downer cows.

>>>In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks... <<<

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

Wednesday, December 16, 2009

Congress to Sample School Lunches

http://downercattle.blogspot.com/2009/12/congress-to-sample-school-lunches.html

Do you actually believe all these schools recalled this meat because of a few cattle being abused,

see list ; FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

IF url does not work above, go to this link to find out if any of your children and their school were part of this recall ; go to this site ;

http://www.fns.usda.gov/fns/

left hand corner search ; Hallmark/Westland Meat Packing Co. Beef Recall your should get this ;

http://65.216.150.153/texis/search?pr=FNS

1 through 1 of 1 matching documents, best matches first. sort by date 1: Hallmark - Westland SFA Reporting by State - 3-24-2008.xls Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 The U.S. Department of Agriculture ...

http://www.fns.usda.gov/...ety/Hallmark-Westland_byState.pdf

PLEASE SEE ALSO ;

Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME. snip... The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

CJD USA RISING, with UNKNOWN PHENOTYPE ;

5 Includes 41 cases in which the diagnosis is pending, and 17 inconclusive cases; 6 Includes 46 cases with type determination pending in which the diagnosis of vCJD has been excluded.

http://www.cjdsurveillance.com/pdf/case-table.pdf

Saturday, January 2, 2010

Human Prion Diseases in the United States January 1, 2010 ***FINAL***

http://prionunitusaupdate2008.blogspot.com/2010/01/human-prion-diseases-in-united-states.html

my comments to PLosone here ;

http://www.plosone.org/annotation/listThread.action?inReplyTo=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd&root=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd

Tuesday, March 2, 2010

Animal Proteins Prohibited in Ruminant Feed/Adulterated/Misbranded Rangen Inc 2/11/10 USA

http://madcowfeed.blogspot.com/2010/03/animal-proteins-prohibited-in-ruminant.html

Monday, March 1, 2010

ANIMAL PROTEIN I.E. MAD COW FEED IN COMMERCE A REVIEW 2010

http://madcowfeed.blogspot.com/2010/03/animal-protien-ie-mad-cow-feed-in.html

14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf

see page 114

International Society for Infectious Diseases Web: http://www.isid.org

Saturday, January 2, 2010

Human Prion Diseases in the United States January 1, 2010 ***FINAL***

http://prionunitusaupdate2008.blogspot.com/2010/01/human-prion-diseases-in-united-states.html

my comments to PLosone here ;

http://www.plosone.org/annotation/listThread.action?inReplyTo=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd&root=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd

Friday, February 05, 2010

New Variant Creutzfelt Jakob Disease case reports United States 2010 A Review

http://vcjd.blogspot.com/2010/02/new-variant-creutzfelt-jakob-disease.html

Sunday, February 14, 2010

[Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine Spongiform Encephalopathy (BSE)

http://bseusa.blogspot.com/2010/02/docket-no-fsis-2006-0011-fsis-harvard.html

Wednesday, February 24, 2010

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America 14th

ICID International Scientific Exchange Brochure -

http://transmissiblespongiformencephalopathy.blogspot.com/2010/02/transmissible-spongiform-encephalopathy.html

TSE

http://transmissiblespongiformencephalopathy.blogspot.com/

Wednesday, March 3, 2010

NOR-98 ATYPICAL SCRAPIE USA 4 CASES DETECTED JANUARY 2010

http://nor-98.blogspot.com/2010/03/nor-98-atypical-scrapie-usa-4-cases.html

Friday, March 5, 2010

Fatal Transmissible Amyloid Encephalopathy: A New Type of Prion Disease Associated with Lack of Prion Protein Membrane Anchoring

http://betaamyloidcjd.blogspot.com/2010/03/fatal-transmissible-amyloid.html

TSS

School Food Safety Program Based on Hazard Analysis and Critical Control Point Principles (HACCP); Approval of Information Collection Request

2010-02-24T13:38:00.000-08:00

Sent: Wednesday, February 24, 2010 11:32 AM
Subject: School Food Safety Program Based on Hazard Analysis and Critical Control Point Principles (HACCP); Approval of Information Collection Request

[Federal Register: February 24, 2010 (Volume 75, Number 36)]
[Rules and Regulations]
[Page 8239]
From the Federal Register Online via GPO Access [wais.access.gpo.gov]
[DOCID:fr24fe10-1]

========================================================================
Rules and Regulations
Federal Register
________________________________________________________________________

This section of the FEDERAL REGISTER contains regulatory documents
having general applicability and legal effect, most of which are keyed
to and codified in the Code of Federal Regulations, which is published
under 50 titles pursuant to 44 U.S.C. 1510.

The Code of Federal Regulations is sold by the Superintendent of Documents.
Prices of new books are listed in the first FEDERAL REGISTER issue of each
week.

========================================================================

[[Page 8239]]

DEPARTMENT OF AGRICULTURE

Food and Nutrition Service

7 CFR Parts 210 and 220

[FNS-2008-0033]
RIN 0584-AD65

School Food Safety Program Based on Hazard Analysis and Critical
Control Point Principles (HACCP); Approval of Information Collection
Request

AGENCY: Food and Nutrition Service, USDA.

ACTION: Final rule; approval of information collection request.

-----------------------------------------------------------------------

SUMMARY: The final rule entitled School Food Safety Program Based on Hazard Analysis and Critical Control Point Principles (HACCP) was published on December 15, 2009, which implemented a legislative provision requiring school food authorities participating in the National School Lunch Program (NSLP) or the School Breakfast Program (SBP) to develop a school food safety program for the preparation and service of school meals served to children. The Office of Management and Budget (OMB) cleared the associated information collection requirements (ICR) on November 2, 2009. This document announces approval of the ICR.

DATES: The ICR associated with the final rule published in the Federal Register on December 15, 2009, at 74 FR 66213, was approved by OMB on December 30, 2009, under OMB Control Number 0584-0550.

FOR FURTHER INFORMATION CONTACT: Lynn Rodgers-Kuperman, Chief, Program Analysis and Monitoring Branch, Child Nutrition Division, Food and Nutrition Service, USDA, 3101 Park Center Drive, Room 640, Alexandria, Virginia 22302, (703) 305-2600, or Lynn.Rogers@fns.usda.gov.

SUPPLEMENTARY INFORMATION: The December 15, 2009 (74 FR 66213), final rule implemented a legislative provision which requires school food authorities participating in the National School Lunch Program (NSLP) or the School Breakfast Program (SBP) to develop a school food safety program for the preparation and service of school meals served to children. The school food safety program must be based on the (HACCP) system established by the Secretary of Agriculture. The food safety program enables schools to take systematic action to prevent or minimize the risk of food-borne illness among children participating in the NSLP and SBP. The information collection requirements were approved by OMB on December 30, 2009.

Dated: February 4, 2010.
Julia Paradis,
Administrator, Food and Nutrition Service.
[FR Doc. 2010-3476 Filed 2-23-10; 8:45 am]
BILLING CODE 3410-30-P

http://edocket.access.gpo.gov/2010/2010-3476.htm

Foodborne Disease Outbreaks in United States Schools: Discussion

Discussion

Between 1973 and 1997, >600 foodborne disease outbreaks in schools were reported to CDC. These outbreaks resulted in nearly 50 000 illnesses, >1500 hospitalizations and 1 death. This represents ~5% of all foodborne disease outbreaks and 12% of all outbreak-associated cases reported to CDC. The three most commonly identified etiologic agents were Salmonella, S. aureus and C. perfringens. The percentage of outbreaks of known etiology due to Salmonella serotype Enteritidis increased over the surveillance period, whereas the percentage caused by S. aureus decreased. Other notable trends include an 8-fold decrease in the percentage of outbreaks due to turkey, a 3-fold increase in outbreaks linked to salads and a decline in the number of milk-associated outbreaks. Whether these changes reflect variation in the relative safety of these items, a change in dietary habits or both cannot be determined with certainty from these data. Nevertheless some of the changes may be attributed to fewer tours of milk dairies and raw milk tastings and a large increase in the number of salads consumed.

The majority of outbreaks with known vehicles were caused by foods prepared on school premises. Therefore prevention efforts should focus on school-based interventions. Practices identified as contributing to outbreaks in schools include improper refrigeration, prolonged handling and inadequate reheating of cooked foods.[7] Following established food safety guidelines on food preparation, handling, storage and service can greatly reduce the risk of foodborne disease outbreaks.[13-15] All meat and poultry should be thoroughly cooked, cooked foods not used immediately should be rapidly chilled to refrigeration temperature (<40°f)>160°F), that the temperature of egg-containing products be carefully monitored and that devices used to mix or prepare raw eggs be regularly disassembled, cleaned and sanitized. Pasteurized eggs should be used for all meals requiring large quantities of pooled eggs and are the best choice for all recipes containing eggs.

Several outbreaks in schools have been attributed to contamination of food by food-handlers who worked while ill[6, 11] or had poor personal hygiene.[17, 18] In our review of reported foodborne outbreaks in school, 57% of outbreaks were attributed to likely contamination by a food-handler. The adoption of a work policy that includes paid leave for food handlers with gastroenteritis would probably increase compliance with illness-related work exclusion policies. Training and certifying all food handlers in school cafeterias in specific techniques, such as good personal hygiene, adequate hand washing, proper cooling and reheating of foods and methods of preventing cross-contamination between cooked and raw foods, would also likely reduce the incidence of foodborne disease outbreaks.[19]

In addition to promoting proper food handling and hygiene practices among school employees, it is important that schools purchase foodstuffs that have been produced safely. Purchase contracts for meat, poultry and eggs often have not stipulated food safety criteria.[20] Requiring such foods to be produced under Hazard Analysis and Critical Control Point (HACCP) or egg quality assurance plans that meet microbiologic performance requirements would be an important addition to school food safety. During school year 2000 the US Department of Agriculture (USDA), which annually purchases >100 million pounds of beef products for the National School Lunch Program and other federal food and nutrition programs, began requiring that ground beef purchased through the program test negative for both E. coli O157:H7 and Salmonella.[21] However, USDA directly provides only a small percentage of food served in schools. School food authorities purchase 83% of the food served in school lunch programs and all of the food served in school breakfast programs.[20] Additional efforts to assure the purchase of safe food stuffs may be an important step, although the extent to which this would increase the risk of foodborne outbreaks in schools is not clear. Recent US declines in Salmonella and Campylobacter are credited to the mandated HACCP rule for meat and poultry. The changes in incidence of foodborne infections have occurred in the context of the introduction of the HACCP regulations for meat and poultry in processing plants, increased attention to egg and fresh produce safety, industry efforts, food safety education, increased regulation of imported food and other prevention measures.[22]

The risk of outbreaks caused by bacterial and parasitic pathogens could be further reduced through the broader application of irradiation pasteurization of solid foods using low-dose gamma rays, radiographs or electron beams.[23-25] Although widespread use of this technology has been hampered by the perception of consumer concerns,[26] the use of irradiation to pasteurize food has been endorsed by the World Health Organization, the US Department of Health and Human Services, the USDA, the American Medical Association and the American Public Health Association.[26-29] Irradiation pasteurization of meat and poultry used in schools would be an important further step in reducing outbreaks caused by to bacterial pathogens such as E. coli O157:H7 and Salmonella. Because viral foodborne pathogens such as hepatitis A and Norwalk-like viruses are more radioresistant, irradiation pasteurization is less likely to prevent outbreaks due to these pathogens.

The limitations of our report should be recognized. The number of foodborne outbreaks reported by this passive surveillance system represents only a small proportion of those that occur. Foodborne outbreaks caused by an etiologic agent with a short incubation period (e.g. bacterial toxins) are more likely to be recognized as common source outbreaks than are diseases with longer incubation periods (e.g. hepatitis A). Furthermore our report likely underestimates the proportion of viral gastroenteritis outbreaks in schools, because stool and serum testing of specimens for viral pathogens (e.g. Norwalk-like viruses) is not widely available, making confirmation of these outbreaks more difficult. Outbreaks caused by organisms that are not routinely screened for by laboratories or that require special media for detection, such as E. coli O157:H7, Norwalk-like viruses, Vibrio species, Yersinia, C. perfringens and Campylobacter may be underrepresented. In addition testing of some organisms (e.g. E. coli O157:H7, Norwalk-like viruses, enzyme immunoassay for Giardia and toxin testing) became available during the course of the time period covered in this review. Information on the size and other characteristics of affected schools was not available. Other limitations include lack of information about how factors contributing to outbreak were determined and lack of information on the specific school setting (e.g. elementary school, high school or university). A recently published primer directed to primary care physicians, who are more likely to see the index case of a potential food-related disease outbreak, is a teaching tool for primary care physicians about foodborne illness and to remind them of their important role in recognizing suspicious symptoms, disease clusters and etiologic agents and reporting cases of foodborne illness to public health authorities.[30]

Relative to the number of meals served in America's schools, the number of reported outbreaks of foodborne illness may appear relatively small. Nevertheless the cumulative disease burden on this vulnerable population is considerable. Adequate training of school staff and the integration of food safety criteria into purchase contracts are measures that can be immediately employed to reduce the burden of foodborne disease among school children. Investigations of foodborne disease outbreaks in schools and continued outbreak surveillance are needed to identify trends in disease frequency, to detect the emergence of new causes of foodborne illness and to ensure the highest standards of food safety for school children in America.

Foodborne disease websites: www.cdc.gov/foodnet, http://www.foodsafety.gov/, http://www.cfsan.fda.gov/,
www.cdc.gov/ncidod/dbmd/diseaseinfo/foodborneinfections
and http://www.cdc.gov/ncidod/dbmd/outbreak/default.htm.

http://www.medscape.com/viewarticle/439841_4

Q: You've been very vocal about your belief that HACCP [Hazard Analysis and Critical Control Points] systems employed by the meat industry are 'a hoax.' If you were writing the meat safety rules, what would they look like?

A: Number one, HACCP was advertised as being science-based, and it is not. HACCP was designed by Pillsbury 20-30 years ago. They were making fully-cooked, ready-to-eat food for the astronaut program and it had to be guaranteed safe. Well, those were highly-processed, fully-cooked ready-to-eat-foods--the pathogens would be cooked out--they all had a real "kill step."

Well, the USDA saw the HACCP program and thought, 'Gee, that sounds really good. Lets apply that meat inspection.' The problem is the vast majority of what we process in meat plants is not fully-cooked, ready-to-eat, it is raw.

The USDA shouldn't use the term HACCP unless the products they are working with are fully cooked.

Number two, when the agency required the industry to implement HACCP, the agency said that under the program the USDA's role would be hands-off. That is an absolute disaster.

The USDA cannot be hands-off.

Under the transition to HACCP, the USDA knowingly acquiesced its authority back to the industry. It's an absolute disaster waiting to happen.

The natural long-term consequences of the agency adopting a hands-off, non-involvement role is ongoing outbreaks and recurring recalls all the time now.

HACCP cannot work in the raw meat industry.

I really think that meat inspection should be moved from the USDA, and that a separate agency should be created to perform inspection of not only meat and poultry but also produce, which is currently assigned to the FDA.

http://www.foodsafetynews.com/2009/11/qa-with-meatpacking-maverick-munsell/index.html?p=2

A: I say it's embarassing, because it should be embarassing to the USDA that, in spite of their "science-based" meat inspection program we have all these ongoing outbreaks and recalls. It should also be embarrassing to our industry.

It's been 11 year since the biggest packers implemented the HACCP program. You'd think that by this point the program would be maturing and paying dividends, but in fact it's going the opposite direction.

We've got to finally realize that the consuming pubic are going to see through this--this façade. At times I wonder if the word "embarrassing" is not the right word. The overarching, more important concern is food safety and sick consumers and people who are dying.

People are dying. A lot of people are getting sick, this should be an embarrassment to this industry, and to the USDA.

http://www.foodsafetynews.com/2009/11/qa-with-meatpacking-maverick-munsell/

From: TSS

Subject: Food Safety and Inspection Service Assessment of the Equivalence of the Canadian Inspection System Report No: 24601-05-HY OIG

Date: January 12, 2006 at 7:22 am PST

Audit Report

Food Safety and Inspection Service

Assessment of the Equivalence of the

Canadian Inspection System

Report No. 24601-05-Hy December 2005

snip...

Executive Summary

Food Safety and Inspection Service Assessment of the Equivalence of the Canadian Inspection System (Audit Report No. 24601-05-Hy)

Results in Brief We evaluated the Food Safety and Inspection Service’s (FSIS) assessment of the equivalence of the Canadian inspection system for meat and poultry products. In a November 6, 2003, memorandum, the FSIS Administrator and the Under Secretary for Food Safety identified serious concerns with the Canadian inspection system. They noted in the memorandum that these concerns had the potential for compromising public health. We found FSIS did not timely address these serious concerns. For example, in July 2003, FSIS identified that Canadian inspection officials were not enforcing certain pathogen reduction and Hazard Analysis and Critical Control Point (HACCP) system regulations. These same types of concerns were identified again in June 2005, almost 2 years later.

Timely actions were not taken because FSIS does not have protocols or guidelines for evaluating deficiencies in a country’s inspection system that could jeopardize a country’s overall equivalence determination. In addition, FSIS did not institute compensating controls (e.g., increased port-of-entry testing) to ensure that public health was not compromised while deficiencies were present. Over 4.4 billion pounds of Canadian processed product entered U.S. commerce from January 1, 2003 through May 31, 2005. In FSIS’ information system, the products were categorized as cuts and trimmings of raw product as well as products with additional processing from pork, veal, beef, poultry, and lamb. These products were produced and allowed to be exported to the United States even though FSIS officials questioned the equivalence of the Canadian inspection system.

FSIS regulations1 require foreign inspection systems to provide standards equivalent to those of the United States. These requirements include the implementation of sanitation controls and HACCP requirements. Sanitation controls cover all aspects of facility and equipment sanitation, the prevention of actual or potential instances of product cross-contamination, good personal hygiene practices, and good product handling and storage practices. All plants must develop, adopt, and implement a HACCP plan for each of their processes. Under HACCP, plants identify critical control points during their processes where hazards such as microbial contamination can occur, establish controls to prevent or reduce those hazards, and maintain records documenting that controls are working as intended.

In July 2003, as part of an onsite review, FSIS identified serious concerns with the Canadian inspection system. These concerns included the

1 Title 9, Code of Federal Regulations (C.F.R.) § 327.2 (a) (2) and 9 C.F.R. § 381.196 (a) (2), January 1, 2005 edition.

USDA/OIG-Audit No. 24601-05-Hy Page ii

insufficient implementation of sanitation controls and HACCP requirements by establishments and the lack of enforcement in these areas by Canadian inspection officials. Based on these concerns, FSIS proposed an enforcement review in 2004. (Enforcement reviews can lead to a determination that a country’s system is not equivalent to U.S. standards and, thus, not eligible to export to the United States). The proposed 2004 enforcement review was not conducted and FSIS officials did not reassess Canada’s implementation and enforcement of sanitation controls and HAACP requirements until almost 2 years later. When FSIS officials finally returned to Canada in May 2005, they continued to find the same types of deficiencies they had found in 2003. FSIS should analyze the deficiencies identified in the 2003 and 2005 reviews to determine whether immediate actions are needed to address concerns regarding public health and if additional enforcement measures are needed.

FSIS’ analysis of the regulations governing the Canadian inspection system identified two areas which may not be equivalent to the United States inspection system. FSIS found that Canadian policy allowed less than daily inspection coverage in processing establishments. By contrast, FSIS has a long established history of requiring the presence of an inspector in a U.S. processing establishment at least once per shift per day. FSIS also identified differences in the testing performed for Listeria monocytogenes. Canadian inspection officials require establishments to perform risk-based environmental sampling, as opposed to the finished product sampling required by FSIS.

In a management alert to the FSIS Administrator in July 2005, we reported that FSIS had not taken timely action to resolve the agency’s June 2003 finding that Canada does not require daily inspection coverage at processing establishments that export product to the United States. In addition, FSIS’ actions regarding Canadian processing establishments were not consistent with how the agency treated similarly situated countries. When FSIS identified less than daily inspection in establishments in Australia in June 2004, and in Belgium in July 2003, the establishments were immediately delisted and no longer allowed to export product to the United States. According to FSIS officials, Australia and Belgium did not pursue an equivalence determination, which was pursued by Canada. In response to our recommendations, FSIS agreed to initiate a number of actions to ensure that an equivalence determination was made regarding daily inspection coverage. However, FSIS asserted that a final decision could not be made until 2007. In the interim, FSIS agreed to implement measures that the agency believes will ensure there is no increased risk to the public health in the United States. These measures included doubling the sampling of Canadian shipments and increasing the presence of Canadian inspection officials in processing establishments exporting to the United States.

USDA/OIG-Audit No. 24601-05-Hy Page iii

Recommendation

In Brief FSIS needs to develop and implement protocols for postponing or canceling a scheduled enforcement review and for determining which equivalence deficiencies would call into question a country’s overall equivalence to U.S. standards. In addition, FSIS should analyze the deficiencies identified in the 2003 and 2005 reviews of the Canadian inspection system to determine whether additional actions are needed to address concerns regarding public health. Finally, FSIS needs to develop an action plan for determining whether the Canadian inspection system control for Listeria monocytogenes in ready-to-eat products is equivalent to that of the United States.

Agency Response

FSIS agreed with the report’s recommendations. We have incorporated the agency’s response in the Findings and Recommendations section of this report, along with the OIG position. The response is included as Exhibit A.

OIG Position

Based on the response, we were able to reach management decision on the report’s five recommendations.

snip...

however, these reviews did not focus on the differences identified in 2003. These two reviews primarily evaluated the implementation of FSIS’ requirements related to BSE. In December 2004, FSIS officials performed a review of 15 Canadian establishments that slaughtered cattle and calves for export to the United States. This review found that Canadian establishments implemented FSIS’ requirements for BSE and controlled the use of hormone implants in calves. In February 2005, FSIS officials visited two Canadian beef slaughter establishments and three establishments that processed this product. This review found that

USDA/OIG-Audit No. 24601-05-Hy Page 8

7 Specified risk materials are prohibited from use for human food. The materials include the brain, skull, eyes, trigeminal ganglia, spinal cord, vertebral column (excluding the vertebrae of the tail, the traverse processes of the thoracic and lumbar vertebrae, and the wings of the sacrum), dorsal root ganglia of cattle 30 months of age and older, tonsils, and distal ileum of the small intestine of all cattle.

Canadian inspection officials adequately implemented FSIS’ rules regarding BSE and specified risk materials.7

• Serious Concerns Continue in 2005. In May 2005, FSIS initiated a more thorough examination of the Canadian inspection system. FSIS visited 35 establishments, which included 3 meat slaughter establishments, 21 meat and poultry processing establishments, and 11 meat and poultry establishments that had both slaughter and processing operations. FSIS also evaluated operations for residue and microbiological testing at 12 laboratories. The review was completed in June 2005, and FSIS officials continued to find a number of deficiencies that call into question the equivalence of the Canadian inspection system. As in 2003, the deficiencies included the insufficient implementation of sanitation controls and HACCP requirements by establishments and the lack of enforcement in these areas by Canadian inspection officials. FSIS officials noted, but did not report, less than daily inspection at 17 processing establishments.

- In 21 of the 35 establishments, FSIS officials found that the Canadian inspection system did not have adequate sanitation controls. FSIS continued to find that Canadian establishments did not ensure sanitation controls were adequately implemented or evaluated for effectiveness. In addition, the establishments did not take corrective actions when sanitation controls failed to prevent direct product contamination or adulteration and did not maintain daily records of these activities.

- FSIS officials found that Canadian inspection officials did not implement certain HACCP requirements in 19 of the 35 establishments. FSIS again found that Canadian establishments were deficient in validating their HACCP plans, documenting corrective actions, and reassessing the adequacy of the plans.

- As part of the review of specific establishments, FSIS again evaluated whether Canadian inspection officials adequately enforced FSIS requirements. FSIS officials found that the Canadian inspection system did not have adequate controls to ensure FSIS requirements were enforced. FSIS officials identified deficiencies in the areas of sanitation controls and HACCP requirements that had not been previously noted by Canadian inspection officials. This condition occurred in 29 of the 35 establishments visited by FSIS officials.

USDA/OIG-Audit No. 24601-05-Hy Page 9

In 2003, FSIS identified concerns which caused the agency to question the equivalence of the Canadian inspection system and to express concern about U.S. public health. The same types of concerns were identified in the review completed in June 2005. FSIS should analyze the deficiencies identified in the 2003 and 2005 reviews to determine whether immediate actions are needed to address concerns regarding public health and if additional enforcement measures are needed.

• Less Than Daily Inspection in Processing Establishments. ...

snip...

http://www.usda.gov/oig/webdocs/24601-05-HY.pdf

Greetings,

question please, i do no understand the part that ;

This review found that

USDA/OIG-Audit No. 24601-05-Hy Page 8

>>> Canadian inspection officials adequately implemented FSIS’ rules regarding BSE and specified risk materials.7<<<

To: fdadockets@oc.fda.gov

Cc: ggraber@cvm.fda.gov; Linda.Grassie@fda.gov; BSE-L

Subject: Docket No. 2003N-0312 Animal Feed Safety System [TSS SUBMISSION TO DOCKET 2003N-0312]

Greetings FDA, my name is Terry S. Singeltary Sr., i lost my mother to hvCJD (Heidenhain Variant Creutzfeldt Jakob Disease). i would kindly like to comment on the proposed HACCP method of detecting and or preventing TSEs in the human/animal feed supply. it seems to me by implementing something that was designed for Astronauts instead of cattle, something that the GAO has already stated is terribly flawed (HACCP), i find it very disturbing to continue to insist on refusing to use rapid TSE TESTING in sufficient numbers to find TSEs, as with other Countries that they too once thought they were BSE free. for example, it took Italy 1 MILLION rapid TSE tests since 2001 to find 102 cases of BSE. THE USA has only tested 48,000 cattle in the 14 years of surveillance. there is documented proof that indeed the USA cattle have been infected with a TSE for decades, but the FDA/USDA and other USA Gov. agencies continue to conveniently ignore these findings. YOU must not ignore what Richard Marsh found. Plus, you must not ignore Asante/Collinge new findings that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest _sporadic_ CJD. The USA has been feeding ruminant by-products back to cattle, deer, elk and sheep for decades, and TSEs in these species have been recycled for feed for decades in the USA. The rendering process here in the USA will not kill this agent. to implement any HACCP over massive rapid TSE testing is only prolonging the inevitable, and will only allow the agent to spread further. it is simply a band-aid approach to something that needs a tourniquet... 3. Meat and Poultry: Better USDA Oversight and Enforcement of Safety Rules Needed to Reduce Risk of Foodborne Illnesses. GAO-02-902, August 30. FSIS Is Not Ensuring that Plants' HACCP Plans Meet Regulatory Requirements

snip...

According to FSIS's food safety systems correlation reviews, inspectors are not consistently identifying and documenting failures of plants' HACCP plans to meet regulatory requirements. Furthermore, FSIS does not expect its inspectors to determine whether HACCP plans are based on sound science--the cornerstone of an effective plan. While in-depth verification reviews examine the scientific aspects of HACCP plans, they have been conducted in very few plants, and consumer safety officers hired to review the scientific soundness of HACCP plans may take several years to assess the plans at all plants. Moreover, inspectors in 55 percent of the 5,000 plants nationwide did not document any HACCP violations during fiscal year 2001. When we brought this information to the attention of FSIS officials, they were surprised that so many plants had no HACCP violations for an entire year.

snip...

2. USDA believes that the title of the report is misleading. We disagree. We believe the title accurately reflects the concerns detailed throughout the body of the report.

snip...

http://www.gao.gov/cgi-bin/getrpt?GAO-02-902

http://www.gao.gov/new.items/rc00255.pdf

FDA acknowledges that it has not yet identified and inspected all firms subject to the ban” pg. 3 ;

http://www.gao.gov/new.items/d02183.pdf

The report concludes that “federal actions do not sufficiently ensure that all BSE-infected animals or products are kept out or that if BSE were found it would be detected promptly and not spread to other cattle through animal feed or enter the human food chain” italics added pg. 3 ;

http://www.gao.gov/new.items/d02183.pdf

and why does everybody conveniently ignore these findings;

Asante/Collinge et al, that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest _sporadic_ CJD;

http://www.fda.gov/ohrms/dockets/ac/03/slides/3923s1_OPH.htm

To be published in the Proceedings of the Fourth International Scientific Congress in Fur Animal Production. Toronto, Canada, August 21-28, 1988

Evidence That Transmissible Mink Encephalopathy Results from Feeding Infected Cattle

R.F. Marsh* and G.R. Hartsough •Department of Veterinary Science, University of Wisconsin-Madison, Madison, Wisconsin 53706; and ^Emba/Creat Lakes Ranch Service, Thiensville, Wisconsin 53092

ABSTRACT

Epidemiologic investigation of a new incidence of transmissible mink encephalopathy (TME) in Stetsonville, Wisconsin suggests that the disease may have resulted from feeding infected cattle to mink. This observation is supported by the transmission of a TME-like disease to experimentally inoculated cattle, and by the recent report of a new bovine spongiform encephalopathy in England. INTRODUCTION ....snip...end...TSS

http://www.fda.gov/ohrms/dockets/dockets/03n0312/03N-0312_emc-000001.txt

NSLP USDA CERTIFIED DEAD STOCK DOWNER COW SCHOOL LUNCH PROGRAM

>>>Question #1: How many kids have died from a pathogen outbreak involving ground beef that is dervied from the NSLP? Answer: ZERO<<<

>In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks<

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

Wednesday, December 16, 2009 Congress to Sample School Lunches

http://downercattle.blogspot.com/2009/12/congress-to-sample-school-lunches.html

Do you actually believe all these schools recalled this meat because of a few cattle being abused,

see list ;

FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

IF url does not work above, go to this link to find out if any of your children and their school were part of this recall ;

go to this site ;

http://www.fns.usda.gov/fns/

left hand corner search ; Hallmark/Westland Meat Packing Co. Beef Recall your should get this ;

http://65.216.150.153/texis/search?pr=FNS

1 through 1 of 1 matching documents, best matches first. sort by date 1: Hallmark - Westland SFA Reporting by State - 3-24-2008.xls Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 The U.S. Department of Agriculture ...

http://www.fns.usda.gov/...ety/Hallmark-Westland_byState.pdf

PLEASE SEE ALSO ;

Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME. snip... The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

PLEASE be aware, for 4 years, the USDA fed our children all across the Nation dead stock downer cows, the most high risk cattle for BSE aka mad cow disease and other dangerous pathogens. who will watch our children for CJD for the next 5+ decades ???

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

please see full text here ;

Tuesday, November 17, 2009

SEAC EFFECT OF AGE ON THE PATHOGENESIS OF TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES SEAC 103/2

http://downercattle.blogspot.com/2009/11/seac-effect-of-age-on-pathogenesis-of.html

Saturday, January 2, 2010

Human Prion Diseases in the United States January 1, 2010 ***FINAL***

http://prionunitusaupdate2008.blogspot.com/2010/01/human-prion-diseases-in-united-states.html

my comments to PLosone here ;

http://www.plosone.org/annotation/listThread.action?inReplyTo=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd&root=info%3Adoi%2F10.1371%2Fannotation%2F04ce2b24-613d-46e6-9802-4131e2bfa6fd

Friday, February 05, 2010

New Variant Creutzfelt Jakob Disease case reports United States 2010 A Review

http://vcjd.blogspot.com/2010/02/new-variant-creutzfelt-jakob-disease.html

Sunday, February 14, 2010

[Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine Spongiform Encephalopathy (BSE)

http://bseusa.blogspot.com/2010/02/docket-no-fsis-2006-0011-fsis-harvard.html

Wednesday, February 24, 2010

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America
14th ICID International Scientific Exchange Brochure -

14th ICID International Scientific Exchange Brochure -

Final Abstract Number: ISE.114

Session: International Scientific Exchange

Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America

update October 2009

T. Singeltary

Bacliff, TX, USA

Background:

An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.

Methods:

12 years independent research of available data

Results:

I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.

Conclusion:

I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.

http://ww2.isid.org/Downloads/14th_ICID_ISE_Abstracts.pdf

International Society for Infectious Diseases Web: http://www.isid.org/

http://transmissiblespongiformencephalopathy.blogspot.com/2010/02/transmissible-spongiform-encephalopathy.html

Transmissible Spongiform Encephalopathy

http://transmissiblespongiformencephalopathy.blogspot.com/

TSS

Congress to Sample School Lunches

2009-12-16T13:51:00.000-08:00

Congress to Sample School Lunches

12/16/2009 9:41:00 AM

WASHINGTON—The U.S. Department of Agriculture (USDA) will serve a sampling of traditional school lunch menu items to members of Congress next week to illustrate improvements its has made in the nutritional quality and taste of the $1.2 billion in school commodity foods and as well as win support to boost funding to continue reworking menus, reported the Washington Post.

http://www.foodproductdesign.com/news/2009/12/congress-to-sample-school-lunches.aspx

USDA will offer lawmakers a sampling of school lunches Agency seeks boost in funding to continue reworking menus

By Jane Black Washington Post Staff Writer Friday, December 11, 2009

Chicken fajita strips, sliced ham and canned green beans: That's what's for lunch one day next week for some lawmakers and congressional staffers, courtesy of the U.S. Department of Agriculture. The menu offers the same products, known as commodity foods, that the agency provides every day to public schools across the nation.

The goal of next week's tasting is to show lawmakers the improvements the department has made in the nutritional quality -- and taste -- of the $1.2 billion in school commodity foods and to win support to fund further improvements. With one-third of American children overweight or obese, the USDA has been working to cut salt and fat and provide more fruits and vegetables.

"These guys are moving in the right direction," said Tony Geraci, food service director for Baltimore City public schools and a pioneer for healthful foods in schools. "Is it fixed? Hell, no. But at least now we're having conversations about this. Before, it was straight-up stonewalling."

The tasting is also an attempt to rehabilitate the reputation of the commodity foods program, which provides 15 to 20 percent of the food served in U.S. school cafeterias. Officially called USDA Foods, the program has long been perceived as a conflict of interest in the department's mission: to support American farmers and ranchers while overseeing nutrition programs for low-income families and schoolchildren.

Is the program a way to distribute meats, cheeses and other commodities that couldn't find a buyer on the open market? Or is the department really making choices based on public health?

Improving the quality of food provided free to schools is important at a time when school budgets are being squeezed. President Obama has proposed an additional $1 billion for child nutrition programs, including school lunch, in his 2010 budget.

But in the face of a projected federal deficit of $1.3 trillion , even the strongest supporters of school-lunch reform say that Congress is unlikely to approve a substantial funding increase when it takes up the issue next year.

To prepare for the Capitol Hill debut next week, the USDA offered samples to Secretary Tom Vilsack, who tried more than a dozen products, including canned green beans, apple slices and hamburger patties.

On paper, anyway, the green beans looked good. They are formulated to meet USDA specifications and have 64 percent less sodium than commercially available canned beans. For the 2010 school year, the agency has mandated that canned vegetables have no more than 140 milligrams of sodium per serving, 71 percent fewer than in the Food and Drug Administration's "healthy" standard.

The hamburger patties, developed for a pilot program last year to help fight childhood obesity, were 95 percent lean. The most similar commercial beef patty available is 92 percent lean.

The USDA offers more than 180 fresh and processed foods to schools, up from 54 in 1981. The products are provided to schools free, based on the number of students eligible for government assistance. Schools buy the rest of their ingredients from commercial suppliers.

School food directors say the quality of available commodities is excellent -- if schools choose wisely. The USDA offers high-quality dried fruits, nuts, brown rice, legumes and unprocessed meat, among other things.

Last month, the USDA announced that as part of the bonus commodity program, which is part of the commodity foods program and allows the agency to buy surplus food to help support prices for farmers, it would make available $33 million worth of apples, tart cherries and dried plums to schools and other programs. Some of the cherries will be processed into cherry-apple juice, with no artificial colors, flavors or sweeteners, for schools.

At least one challenge remains: persuading schools to embrace the more healthful options. Many schools lack kitchens and are at most capable of reheating prepared items. And many school food service directors do not have nutrition or culinary training.

They also know that they can sell more trays of greasy pizza and french fries to students than fruits and vegetables, a tactic that helps keep tight budgets in line. As food service director Geraci said: "If you have 20,000 lunch ladies that just want to open up a box of chicken nuggets, they're going to keep making them."

To encourage more healthful choices, the USDA is awaiting help from Congress when it takes up the Child Nutrition and WIC Reauthorization Act next year. As part of the legislation, lawmakers are considering a measure that would allow the department to set strict standards for all food sold in schools, including vending machine fare. They are also considering how much new money to allocate to the $12 billion annual program.

Vilsack said he hopes Congress will make more money available.

"The nutritional value of these foods is going to be a little bit more expensive," said Vilsack, who said that he expects costs will rise as more students are granted access to free or reduced-price meals. "We've been making progress on the food safety side and on the nutrition side. But to take the next steps, it's going to require more resources."

http://www.washingtonpost.com/wp-dyn/content/article/2009/12/10/AR2009121001956.html

>>>To prepare for the Capitol Hill debut next week, the USDA offered samples to Secretary Tom Vilsack, <<<

>>>who tried more than a dozen products, including canned green beans, apple slices and hamburger patties. <<<

Greetings,

Holy mad cow, this will be just like those token slaughter house inspections and such they do for Japan, Korea, and the other trading partners. Tell them way in advance, and then serve em up the best. Give me a break. I wonder if some of those Congressman/woman were served up some dead stock downer cows, the most high risk cattle for mad cow disease, and other dangerous pathogens, like our children were for 4+ years, via the NSLP ? This should have NEVER happened. No, we must not let them forget. They refuse to speak of it, and I can't forget. It must not happen again. Hopefully, all this is not just hot air coming out of Washington, and indeed Congress plans on cleaning house. I have seen this hot air before, time and time again, and in the end, nothing but hot air. Case in point, the mad cow feed ban of August 4, 1997, and the Surveillance for BSE and other TSE. Your only kidding yourself if you don't think these children were not exposed to high risk dead stock downers. it should never happen again. but they hid it under the guise, (the beef recall), the largest ever (at that time), they mask this beef recall as 'animal abuse'. I don't get it. How was this beef tainted by animal abuse? I don't condone animal abuse, but what about child abuse ? and who will watch our children for the next 5+ decades for CJD ? with our CJD human TSE surveillance system in the USA, even if they are detected (?), they will go down as sporadic, or some odd phenotype that is spontaneous or some odd TSE from nothing $

The PrP TSE mad cow agent in humans can incubate up to 50+ years in some cases, in other cases, not so long. so, come back in 50+ years and confirm this. junk science, industry friendly regulations, and or just not complying with existing regulations have been rampant over the past 12 years I have been paying attention, it has been the norm. maybe sound science will prevail in the end, maybe not. but feeding children diseased and sick cows via the NSLP was flat wrong, and anyone that thinks the largest beef recall there from, was just because a few animals were abused, well, they too are just flat wrong as well. ...

WHO WILL WATCH THE CHILDREN FOR CJD OVER THE NEXT 5 + DECADES ???

Do you actually believe that the USDA et al jumped in on the law suit against Westland/Hallmark, at the time the largest beef recall in USA history, just because a few animals were abused on a video, or to cover their ass, for letting our children, from school district to school district, from state to state, be fed dead stock downer cows.

>>> In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks... <<<

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

Do you actually believe all these schools recalled this meat because of a few cattle being abused,

see list ;

FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

IF url does not work above, go to this link to find out if any of your children and their school were part of this recall ; go to this site ;

http://www.fns.usda.gov/fns/

left hand corner search ; Hallmark/Westland Meat Packing Co.

Beef Recall your should get this ; http://65.216.150.153/texis/search?pr=FNS
1 through 1 of 1 matching documents, best matches first. sort by date 1: Hallmark - Westland SFA Reporting by State - 3-24-2008.xls Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 The U.S. Department of Agriculture ...

http://www.fns.usda.gov/...ety/Hallmark-Westland_byState.pdf

PLEASE SEE ALSO ;

Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

Over the next 8-10 weeks, approximately 40% of all the adult mink on the farm died from TME. snip... The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle...

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

Friday, September 4, 2009

FOIA REQUEST ON FEED RECALL PRODUCT 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009

http://madcowfeed.blogspot.com/2009/09/foia-request-on-feed-recall-product.html

Saturday, August 29, 2009

FOIA REQUEST FEED RECALL 2009 Product may have contained prohibited materials Bulk Whole Barley, Recall # V-256-2009

http://madcowfeed.blogspot.com/2009/08/foia-request-feed-recall-2009-product.html

C O N F I R M E D

----- Original Message ----- From: "Terry S. Singeltary Sr." To: Sent: Thursday, November 05, 2009 9:25 PM Subject: [BSE-L] re-FOIA REQUEST ON FEED RECALL PRODUCT contaminated with prohibited material Recall # V-258-2009 and Recall # V-256-2009

http://madcowfeed.blogspot.com/2009/11/re-foia-request-on-feed-recall-product.html

PLEASE be aware, for 4 years, the USDA fed our children all across the Nation dead stock downer cows, the most high risk cattle for BSE aka mad cow disease and other dangerous pathogens. who will watch our children for CJD for the next 5+ decades ???

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

please see full text here ;

Tuesday, November 17, 2009

SEAC EFFECT OF AGE ON THE PATHOGENESIS OF TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES SEAC 103/2

http://downercattle.blogspot.com/2009/11/seac-effect-of-age-on-pathogenesis-of.html

Wednesday, November 18, 2009

R-CALF: 40 Groups Disagree With USDA's Latest BSE Court Submission

http://bse-atypical.blogspot.com/2009/11/r-calf-40-groups-disagree-with-usdas.html

Monday, October 19, 2009

Atypical BSE, BSE, and other human and animal TSE in North America Update October 19, 2009

http://bse-atypical.blogspot.com/2009/10/atypical-bse-bse-and-other-human-and.html

Sunday, September 6, 2009

MAD COW USA 1997 SECRET VIDEO

http://madcowusda.blogspot.com/2009/09/mad-cow-usa-1997-video.html

U.S.A. HIDING MAD COW DISEASE VICTIMS AS SPORADIC CJD ? see video at bottom

http://creutzfeldt-jakob-disease.blogspot.com/2009/07/usa-hiding-mad-cow-disease-victims-as.html

DAMNING TESTIMONY FROM STANLEY PRUSINER THE NOBEL PEACE PRIZE WINNER ON PRIONS SPEAKING ABOUT ANN VENEMAN see video

http://maddeer.org/video/embedded/prusinerclip.html

CVM Annual Report Fiscal Year 2008: October 1, 2007-September 30, 2008

PUTTING LIPSTICK ON A PIG AND TAKING HER TO A DANCE...TSS

BSE Feed Rule Enforcement: A Decade of Success OFF TO A FAST START

http://madcowfeed.blogspot.com/2008/06/texas-firm-recalls-cattle-heads-that.html

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html

Tuesday, December 1, 2009

IMPORTATION OF CANADIAN CATTLE, BISON, SHEEP, AND GOATS INTO THE UNITED STATES 12/1/09

http://usdameatexport.blogspot.com/2009/12/importation-of-canadian-cattle-bison.html

Monday, November 30, 2009

USDA AND OIE COLLABORATE TO EXCLUDE ATYPICAL SCRAPIE NOR-98 ANIMAL HEALTH CODE

http://nor-98.blogspot.com/2009/11/usda-and-oie-collaborate-to-exclude.html

Monday, November 23, 2009

BSE GBR RISK ASSESSMENTS UPDATE NOVEMBER 23, 2009 COMMISSION OF THE EUROPEAN COMMUNITIES AND O.I.E.

http://docket-aphis-2006-0041.blogspot.com/2009/11/bse-gbr-risk-assessments-update.html

Tuesday, December 15, 2009

Intraspecies transmission of L-type-like bovine spongiform encephalopathy detected in Japan

http://bse-atypical.blogspot.com/2009/12/intraspecies-transmission-of-l-type.html

*** Monday, December 14, 2009 R.I.P. MOM ***

Similarities between Forms of Sheep Scrapie and Creutzfeldt-Jakob Disease Are Encoded by Distinct Prion Types

http://nor-98.blogspot.com/2009/12/similarities-between-forms-of-sheep.html

NAIS COOL BSE

http://naiscoolyes.blogspot.com/2009/12/nais-cool-from-farm-to-fork-mad-cow.html

Saturday, December 05, 2009

Molecular Model of Prion Transmission to Humans

http://creutzfeldt-jakob-disease.blogspot.com/2009/12/molecular-model-of-prion-transmission.html

Thursday, November 05, 2009

Incidence and spectrum of sporadic Creutzfeldt-Jakob disease variants with mixed phenotype and co-occurrence of PrPSc types: an updated classification

http://creutzfeldt-jakob-disease.blogspot.com/2009/11/incidence-and-spectrum-of-sporadic.html

Tuesday, August 11, 2009

Characteristics of Established and Proposed Sporadic Creutzfeldt-Jakob Disease Variants

http://creutzfeldt-jakob-disease.blogspot.com/2009/08/characteristics-of-established-and.html

Sunday, August 09, 2009

CJD...Straight talk with...James Ironside...and...Terry Singeltary... 2009

http://creutzfeldt-jakob-disease.blogspot.com/2009/08/cjdstraight-talk-withjames.html

Saturday, June 13, 2009

Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States 2003 revisited 2009

http://cjdusa.blogspot.com/2009/06/monitoring-occurrence-of-emerging-forms.html

JOURNAL OF NEUROLOGY

MARCH 26, 2003

Send Post-Publication Peer Review to journal:

Re: RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob

disease in the United States

Email Terry S. Singeltary:

flounder9@verizon.net

I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which are sporadic are all spontaneous, without route/source. We have many TSEs in the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey by intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly expose others. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic CJDs in the USA are type 2 PrPSc?

http://www.neurology.org/cgi/eletters/60/2/176#535

LANCET INFECTIOUS DISEASE JOURNAL

Volume 3, Number 8 01 August 2003

Newsdesk

Tracking spongiform encephalopathies in North America

Xavier Bosch

My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my mom to hvCJD (Heidenhain variant CJD) and have been searching for answers ever since. What I have found is that we have not been told the truth. CWD in deer and elk is a small portion of a much bigger problem.

49-year-old Singeltary is one of a number of people who have remained largely unsatisfied after being told that a close relative died from a rapidly progressive dementia compatible with spontaneous Creutzfeldt-Jakob disease (CJD). So he decided to gather hundreds of documents on transmissible spongiform encephalopathies (TSE) and realised that if Britons could get variant CJD from bovine spongiform encephalopathy (BSE), Americans might get a similar disorder from chronic wasting disease (CWD)the relative of mad cow disease seen among deer and elk in the USA. Although his feverish search did not lead him to the smoking gun linking CWD to a similar disease in North American people, it did uncover a largely disappointing situation.

Singeltary was greatly demoralised at the few attempts to monitor the occurrence of CJD and CWD in the USA. Only a few states have made CJD reportable. Human and animal TSEs should be reportable nationwide and internationally, he complained in a letter to the Journal of the American Medical Association (JAMA 2003; 285: 733). I hope that the CDC does not continue to expect us to still believe that the 85% plus of all CJD cases which are sporadic are all spontaneous, without route or source.

Until recently, CWD was thought to be confined to the wild in a small region in Colorado. But since early 2002, it has been reported in other areas, including Wisconsin, South Dakota, and the Canadian province of Saskatchewan. Indeed, the occurrence of CWD in states that were not endemic previously increased concern about a widespread outbreak and possible transmission to people and cattle.

To date, experimental studies have proven that the CWD agent can be transmitted to cattle by intracerebral inoculation and that it can cross the mucous membranes of the digestive tract to initiate infection in lymphoid tissue before invasion of the central nervous system. Yet the plausibility of CWD spreading to people has remained elusive.

Part of the problem seems to stem from the US surveillance system. CJD is only reported in those areas known to be endemic foci of CWD. Moreover, US authorities have been criticised for not having performed enough prionic tests in farm deer and elk.

Although in November last year the US Food and Drug Administration issued a directive to state public-health and agriculture officials prohibiting material from CWD-positive animals from being used as an ingredient in feed for any animal species, epidemiological control and research in the USA has been quite different from the situation in the

UK and Europe regarding BSE.

Getting data on TSEs in the USA from the government is like pulling teeth, Singeltary argues. You get it when they want you to have it and only what they want you to have.Norman Foster, director of the Cognitive Disorders Clinic at the University of Michigan (Ann Arbor, MI, USA), says that current surveillance of prion disease in people in the USA is inadequate to detect whether CWD is occurring in human beings; adding that, the cases that we know about are reassuring, because they do not suggest the appearance of a new variant of CJD in the USA or atypical features in patients that might be exposed to CWD. However, until we establish a system that identifies and analyses a high proportion of suspected prion disease cases we will not know for sure. The USA should develop a system modelled on that established in the UK, he points out.

Ali Samii, a neurologist at Seattle VA Medical Center who recently reported the cases of three hunterstwo of whom were friendswho died from pathologically confirmed CJD, says that at present there are insufficient data to claim transmission of CWD into humans; adding that [only] by asking [the questions of venison consumption and deer/elk hunting] in every case can we collect suspect cases and look into the plausibility of transmission further. Samii argues that by making both doctors and hunters more aware of the possibility of prions spreading through eating venison, doctors treating hunters with dementia can consider a possible prion disease, and doctors treating CJD patients will know to ask whether they ate venison. CDC spokesman Ermias Belay says that the CDC will not be investigating the [Samii] cases because there is no evidence that the men ate CWD-infected meat. He notes that although the likelihood of CWD jumping the species barrier to infect humans cannot be ruled out 100% and that [we] cannot be 100% sure that CWD does not exist in humans & the data seeking evidence of CWD transmission to humans have been very limited.

http://www.thelancet.com/journals/laninf/article/PIIS1473309903007151/%20fulltext

>>>he complained in a letter to the Journal of the American Medical Association (JAMA 2003; 285: 733).

I hope that the CDC does not continue to expect us to still believe that the 85% plus of all CJD cases which are sporadic are all spontaneous, without route or source.<<<

actually, that quote was from a more recent article in the Journal of Neurology (see below), not the JAMA article...

Diagnosis and Reporting of Creutzfeldt-Jakob Disease Singeltary, Sr et al. JAMA.2001; 285: 733-734. Vol. 285 No. 6, February 14, 2001 JAMA Diagnosis and Reporting of Creutzfeldt-Jakob Disease

To the Editor: In their Research Letter, Dr Gibbons and colleagues1 reported that the annual US death rate due to Creutzfeldt-Jakob disease (CJD) has been stable since 1985. These estimates, however, are based only on reported cases, and do not include misdiagnosed or preclinical cases. It seems to me that misdiagnosis alone would drastically change these figures. An unknown number of persons with a diagnosis of Alzheimer disease in fact may have CJD, although only a small number of these patients receive the postmortem examination necessary to make this diagnosis. Furthermore, only a few states have made CJD reportable. Human and animal transmissible spongiform encephalopathies should be reportable nationwide and internationally. Terry S. Singeltary, Sr Bacliff, Tex 1. Gibbons RV, Holman RC, Belay ED, Schonberger LB. Creutzfeldt-Jakob disease in the United States: 1979-1998. JAMA. 2000;284:2322-2323.

FREE FULL TEXT

http://jama.ama-assn.org/cgi/content/extract/285/6/733?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&fulltext=singeltary&searchid=1&FIRSTINDEX=0&resourcetype=HWCIT

http://jama.ama-assn.org/cgi/content/full/285/6/733?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&fulltext=singeltary&searchid=1&FIRSTINDEX=0&resourcetype=HWCIT

Tuesday, August 04, 2009

Susceptibilities of Nonhuman Primates to Chronic Wasting Disease

snip...

From: TSS (216-119-163-189.ipset45.wt.net)

Subject: CWD aka MAD DEER/ELK TO HUMANS ???

Date: September 30, 2002 at 7:06 am PST

From: "Belay, Ermias"

To:

Cc: "Race, Richard (NIH)" ; ; "Belay,

Ermias"

Sent: Monday, September 30, 2002 9:22 AM

Subject: RE: TO CDC AND NIH - PUB MED- 3 MORE DEATHS - CWD - YOUNG HUNTERS

Dear Sir/Madam,

In the Archives of Neurology you quoted (the abstract of which was attached to your email), we did not say CWD in humans will present like variant CJD.

That assumption would be wrong. I encourage you to read the whole article and call me if you have questions or need more clarification (phone: 404-639-3091). Also, we do not claim that "no-one has ever been infected with prion disease from eating venison." Our conclusion stating that we found no strong evidence of CWD transmission to humans in the article you quoted or in any other forum is limited to the patients we investigated.

Ermias Belay, M.D.

Centers for Disease Control and Prevention

-----Original Message-----

From:

Sent: Sunday, September 29, 2002 10:15 AM

To: rr26k@nih.gov; rrace@niaid.nih.gov; ebb8@CDC.GOV

Subject: TO CDC AND NIH - PUB MED- 3 MORE DEATHS - CWD - YOUNG

HUNTERS

Sunday, November 10, 2002 6:26 PM ......snip........end..............TSS

snip...

see full text ;

http://chronic-wasting-disease.blogspot.com/2009/08/susceptibilities-of-nonhuman-primates.html

http://chronic-wasting-disease.blogspot.com/

2 January 2000

British Medical Journal

U.S. Scientist should be concerned with a CJD epidemic in the U.S., as well

http://www.bmj.com/cgi/eletters/320/7226/8/b#6117

15 November 1999

British Medical Journal

vCJD in the USA * BSE in U.S.

http://www.bmj.com/cgi/eletters/319/7220/1312/b#5406

THE PATHOLOGICAL PROTEIN

BY Philip Yam

Yam Philip Yam News Editor Scientific American http://www.sciam.com/

http://www.thepathologicalprotein.com/

SEE REVISITING SPORADIC CJD BY PHILIP YAM THE PATHOLOGICAL PROTEIN

Answering critics like Terry Singeltary, who feels that the U.S. undercounts CJD, Schonberger conceded that the current surveillance system has errors but stated that most of the errors will be confined to the older population. ...

http://books.google.com/books?id=ePbrQNFrHtoC&pg=PA224&lpg=PA224&dq=pathological+protein+philip+yam+singeltary&source=bl&ots=um-LytTT2E&sig=hQVJotGvhvffOsN2fsIDfk2SHXw&hl=en&ei=CaWBSrDLCIKUtgeg_eTVCg&sa=X&oi=book_result&ct=result&resnum=1#v=onepage&q=&f=false

2008

The statistical incidence of CJD cases in the United States has been revised to reflect that there is one case per 9000 in adults age 55 and older. Eighty-five percent of the cases are sporadic, meaning there is no known cause at present.

http://www.cjdfoundation.org/fact.html

Friday, November 30, 2007

CJD QUESTIONNAIRE USA CWRU AND CJD FOUNDATION

http://cjdquestionnaire.blogspot.com/

Saturday, June 13, 2009

Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States 2003 revisited 2009

http://cjdusa.blogspot.com/2009/06/monitoring-occurrence-of-emerging-forms.html

Terry S. Singeltary Sr., P.O. Box 42, Bacliff, Texas USA 77518

wasted days and wasted nights...Freddy Fender

stupid is, as stupid does...Forest Gump

SEAC EFFECT OF AGE ON THE PATHOGENESIS OF TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES

2009-11-17T09:34:00.000-08:00

SEAC 103/2

EFFECT OF AGE ON THE PATHOGENESIS OF TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES

ISSUE

1. Most clinical cases of variant CJD (vCJD) have occurred in young adults. The reasons behind this apparent age-related susceptibility are uncertain. A recent paper by Brown et al in the Journal of Immunology, (attached at annex A), reports the findings of a study in mice, which suggest that age related decline in the functioning of follicular dendritic cells might impair TSE pathogenesis.

STUDY DESIGN

2. The study used C57BL/Dk mice challenged with ME7 scrapie brain homongenate. The mice were assessed weekly for signs of clinical disease and culled at a standard clinical endpoint. Survival times were recorded for mice that did not develop clinical signs of disease and were culled when they showed signs of intercurrent disease.

FINDINGS

3. This study finds that early TSE agent accumulation in the spleens of aged mice was significantly impaired compared to that in young adults. Furthermore, following peripheral exposure, none of the aged mice developed clinical TSE disease during their lifespans, although most mice displayed histopathological signs of TSE disease in their brains.

4. Comparison of follicular dendritic cell networks (FDCs) in the spleens of aged and young adult mice, showed a highly significant reduction in the total number of PrPC expressing FDCs in aged mice when compared with those of young adults. 5. The data imply that the reduced status of FDCs in aged mice significantly impairs the early TSE agent accumulation in

© SEAC 2009

lymphoid tissues and subsequent neuroinvasion. Furthermore, the inefficient neuroinvasion in aged individuals may lead to significant levels of subclinical TSE disease in the population.

BACKGROUND

6. Following peripheral exposure, many TSE agents accumulate first in lymphoid tissues before spreading to the Central Nervous System, where they cause neurodegeneration. Early TSE agent accumulation upon FDCs in lymphoid follicles appears to be critical for efficient neuroinvasion.1

7. Most clinical cases of vCJD have occurred in young adults; the median age at onset of disease was 26 years and the median age at death 28 years (the corresponding ages for sporadic CJD are 67 and 67). The youngest case of vCJD was aged 12 years at onset, while the oldest case of vCJD was aged 74 years.2

ADVICE SOUGHT

8. The committee is invited to note the findings of this study.

SEAC SECRETARIAT

NOVEMBER 2009

1 Klein, M. A., R. Frigg, A. J. Raeber, E. Flechsig, I. Hegyi, R. M. Zinkernagel, C. Weissmann, and A. Aguzzi. 1998. PrP expression in B lymphocytes is not required for prion neuroinvasion. Nat. Med. 4: 1429–1433; Brown, K. L., K. Stewart, D. Ritchie, N. A. Mabbott, A. Williams, H. Fraser, W. I. Morrison, and M. E. Bruce. 1999. Scrapie replication in lymphoid tissuesdepends on PrP-expressing follicular dendritic cells. Nat. Med. 5: 1308–1312. 2 National CJD Surveillance Unit, Seventeenth Annual Report, 2008, p.13. ©

http://www.seac.gov.uk/papers/103-2.pdf

WHO WILL WATCH THE CHILDREN for CJD over the next 5 decades ?

FOR 4 years, the USDA fed dead stock downer cows, the most high risk cattle for mad cow disease and other dangerous pathogens to children all across the USA via the USDA certified dead stock downer cow school lunch program...

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 – February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

or try ;

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf#xml=http://65.216.150.153/texis/search/pdfhi.txt?query=FNS+All+Regions+Affected+School+Food+Authorities+By+State+United+States+Department+of+Agriculture+Food+and+Nutrition+Service+National+School+Lunch+Program+&pr=FNS&prox=page&rorder=500&rprox=500&rdfreq=500&rwfreq=500&rlead=500&rdepth=0&sufs=0&order=r&cq=&id=4ace90e711

or,

go to this site ;

http://www.fns.usda.gov/fns/

left hand corner search ;

Hallmark/Westland Meat Packing Co. Beef Recall

your should get this ;

http://65.216.150.153/texis/search?pr=FNS

1 through 1 of 1 matching documents, best matches first. sort by date 1: Hallmark - Westland SFA Reporting by State - 3-24-2008.xls Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 - February 2008 The U.S. Department of Agriculture ... http://www.fns.usda.gov/...ety/Hallmark-Westland_byState.pdf

Approximately 50.3 million pounds of the beef recalled by HallmarkNVestland went to federal nutrition programs, including the National School Lunch Program, and of those 50.3 million pounds, about 19.6 million pounds had already been consumed at the time the recall was issued. Release No. 0054.08, USDA, Transcript of Technical Briefing - HallmarldWestland Meat Packing Company (Feb. 21, 2008).

9. HSUS members that consume meat products, including beef products, are concerned about eating adulterated meat products and the health risks associated with such adulterated meat. Specifically, they are concerned that downed cattle are at an increased risk for harboring and transmitting BSE prions and other pathogens. The consumption of meat products derived from BSE-infected cattle is believed to cause a human neurological disease known as variant Creutzfeldt-Jakob disease ("vCJD"). The disease is progressive, invariably fatal, and there is no known effective treatment or cure. Downed cattle may also be at higher risk for harboring other foodborne transmissible pathogens, including E. coli 0157:H7, Salmonella, and anthrax. By allowing downed cattle to enter the food supply, USDA's regulatory loophole injures members of The HSUS by placing them at an increased risk of contracting these food-borne illnesses each time they eat beef. 10. Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

United States of America Ex rel. The Humane Society of the United States v. Hallmark Meat Packing Company, Westland Meat Company Inc. (Downed animal abuse/government fraud)

Court or Agency: United States District Court for the Central District of California Plaintiff(s): United States of America Ex rel. The Humane Society of the United States Defendant: Hallmark Meat Packing Company, Westland Meat Company Inc. HSUS Counsel: Peter J. Petersan, Leana Stormont Outside Counsel: Milbank, Tweed, Hadley & McCloy LLP Status: In Briefing

Federal court action under the False Claims Act alleging that Westland/Hallmark defrauded the federal government by violating the terms of its school lunch program contracts requiring the humane handling of animals. The lawsuit was the result of an investigation by The HSUS which exposed the facility's mistreatment of animals too sick or injured to walk and led to the largest meat recall in the nation’s history.

http://www.hsus.org/in_the_courts/docket/us_hallmark.html

http://www.law.gmu.edu/assets/files/academics/schedule/2009/fall/HENRYGREEN_AnimalLaw20090818.pdf

http://www.hsus.org/farm/news/ournews/dept_of_justice_hallmark_050409.html

FOIA

http://www.fsis.usda.gov/PDF/FOIA_Requests_0308.pdf

"California Firm Recalls Beef Products". USDA. February 17, 2008.

http://www.fsis.usda.gov/PDF/Recall_005-2008_Release.pdf

AP (February 17, 2008). "USDA recalls 143 million pounds of beef". MSNBC.

http://www.msnbc.msn.com/id/23212514/

"Statement by Secretary of Agriculture Ed Schafer Regarding Animal Cruelty Charges Filed Against Employees at Hallmark/Westland Meat Packing Company". USDA. February 15, 2008.

http://www.usda.gov/wps/portal/!ut/p/_s.7_0_A/7_0_1OB?contentidonly=true&contentid=2008/02/0044.xml

"USDA Q&A". USDA. February 19, 2008.

http://www.usda.gov/wps/portal/usdahome?contentidonly=true&contentid=Recall_Information.xml

The Case is captioned as United States of America ex rel. The Humane Society of the United States v. Hallmark Meat Packing Company; Westland Meat Company, Inc.

http://www.hsus.org/farm/news/ournews/dept_of_justice_hallmark_050409.html

http://www.econ.iastate.edu/classes/econ362/hallam/NewspaperArticles/DownerCows.pdf

Thursday, September 24, 2009

(09-24) 15:00 PDT LOS ANGELES (AP) --

A Southern California meatpacking plant that supplied beef to the nation's school lunch program slaughtered stumbling, potentially contaminated cows for four years before undercover video of animal abuse prompted a massive beef recall, federal court filings say...

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

also ;

http://www.usda.gov/oig/webdocs/24601-07-KC.pdf

>>>USDA officials cited three other interlocking safeguards that protect the public even if other safeguards, such as ante-mortem inspection, should fail; these safeguards are the removal of Specified Risk Materials (SRM),5 BSE surveillance testing, and the feed ban.6 Under the Federal Meat Inspection Act (FMIA),7<<< href="http://madcowfeed.blogspot.com/2009/11/bse-feed-recall-misbranding-of-product.html">http://madcowfeed.blogspot.com/2009/11/bse-feed-recall-misbranding-of-product.html

Friday, September 4, 2009

FOIA REQUEST ON FEED RECALL PRODUCT 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009

http://madcowfeed.blogspot.com/2009/09/foia-request-on-feed-recall-product.html

Saturday, August 29, 2009

FOIA REQUEST FEED RECALL 2009 Product may have contained prohibited materials Bulk Whole Barley, Recall # V-256-2009

http://madcowfeed.blogspot.com/2009/08/foia-request-feed-recall-2009-product.html

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html

"Everything they did on the Texas cow makes everything USDA did before 2005 suspect," Brown said. ...snip...end

http://www.upi.com/ConsumerHealthDaily/view.php?StoryID=20060315-055557-1284r

PAUL BROWN COMMENT TO ME ON THIS ISSUE

Tuesday, September 12, 2006 11:10 AM

"Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency."

http://madcowtesting.blogspot.com/

see full text sporadic CJD the big lie;

http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0705&L=sanet-mg&T=0&F=&S=&P=135027

PLUS

http://cjdmadcowbaseoct2007.blogspot.com/2008/07/novel-human-disease-with-abnormal-prion.html

Sunday, August 10, 2008

A New Prionopathy OR more of the same old BSe and sporadic CJD

http://creutzfeldt-jakob-disease.blogspot.com/2008/08/new-prionopathy-or-more-of-same-old-bse.html

HUMAN and ANIMAL TSE Classifications i.e. mad cow disease and the UKBSEnvCJD only theory

http://www.regulations.gov/search/Regs/contentStreamer?objectId=090000648027c28e&disposition=attachment&contentType=pdf

AS I SAID BEFORE, WHO WATCH THE CHILDREN FOR CJD FOR THE NEXT 5 DECADES ???

Thursday, October 15, 2009

Cell Study Explains Why Younger People More At Risk Of Variant Creutzfeldt-Jakob Disease (vCJD)

http://downercattle.blogspot.com/2009/10/cell-study-explains-why-younger-people.html

TSS

Cell Study Explains Why Younger People More At Risk Of Variant Creutzfeldt-Jakob Disease (vCJD)

2009-10-15T11:41:00.000-07:00

Cell Study Explains Why Younger People More At Risk Of Variant Creutzfeldt-Jakob Disease (vCJD) ScienceDaily (Oct. 15, 2009) — Specific cells within the immune system could help explain why younger people are more susceptible to variant Creutzfeldt-Jakob disease, scientists believe.

--------------------------------------------------------------------------------

See also: Health & Medicine •Immune System •Lymphoma •Brain Tumor Mind & Brain •Mad Cow Disease •Huntington's Disease •Neuroscience Reference •Transmissible spongiform encephalopathy •Bovine spongiform encephalopathy •Prion •Lymphatic system Patients diagnosed with variant CJD are, on average, 28 years old but it has been unclear why older people are not as affected by the disease. Variant CJD is a rare, degenerative, fatal brain disorder in humans, according to the U.S. Centers for Disease Control and Prevention.

Research at The Roslin Institute of the University of Edinburgh has identified specific cells within the immune system that attract corrupted proteins – known as prions – linked to variant CJD and encourage them to multiply and spread.

The study, published in the Journal of Immunology, looked at how these cells behaved in mice and found that the cells were impaired in older mice. As a result, they were unable to trap and replicate the prions and the mice did not develop clinical disease.

Neil Mabbott, of The Roslin Institute, said: "It has always been unclear why younger people were more susceptible to variant CJD and the assumption that they were more likely to eat cheap meat products is far too simplistic.

"Understanding what happens to these cells, which are important for the body's immune responses, could help us develop better ways of diagnosing variant CJD or even find ways of preventing prions from spreading to the brain. It could also help to create a vaccine."

Prions accumulate in lymphoid tissues – part of the body's immune system that include the spleen, lymph nodes and tonsils – before spreading to the central nervous system where they kill off brain cells and cause neurological disease.

Attempts to estimate the number of people carrying variant CJD have relied upon identifying the presence of prions in tonsil and appendix samples collected during routine operations.

The latest study, funded by the Biotechnology and Biological Sciences Research Council, suggests that even more people may be infected than previously thought as researchers also found prions present in brain tissue from older mice, which had not developed clinical disease.

Even when prions were present in the brains of older mice, however, they were not always found in lymphoid tissues, suggesting that the prediction of cases may be underestimated. It is thought the prions may have spread to the brain before they died off in the lymphoid tissues.

http://www.sciencedaily.com/releases/2009/10/091014102032.htm

The Effects of Host Age on Follicular Dendritic Cell Status Dramatically Impair Scrapie Agent Neuroinvasion in Aged Mice1 Karen L. Brown2,*, Gwennaelle J. Wathne*, Jill Sales, Moira E. Bruce* and Neil A. Mabbott2,* * The Roslin Institute and Royal (Dick) School of Veterinary Sciences, University of Edinburgh, Roslin, United Kingdom; and Biomathematics & Statistics Scotland, Edinburgh, United Kingdom

Following peripheral exposure, many transmissible spongiform encephalopathy (TSE) agents accumulate first in lymphoid tissues before spreading to the CNS (termed neuroinvasion) where they cause neurodegeneration. Early TSE agent accumulation upon follicular dendritic cells (FDCs) in lymphoid follicles appears critical for efficient neuroinvasion. Most clinical cases of variant Creutzfeldt-Jakob disease have occurred in young adults, although the reasons behind this apparent age-related susceptibility are uncertain. Host age has a significant influence on immune function. As FDC status and immune complex trapping is reduced in aged mice (600 days old), we hypothesized that this aging-related decline in FDC function might impair TSE pathogenesis. We show that coincident with the effects of host age on FDC status, the early TSE agent accumulation in the spleens of aged mice was significantly impaired. Furthermore, following peripheral exposure, none of the aged mice developed clinical TSE disease during their lifespans, although most mice displayed histopathological signs of TSE disease in their brains. Our data imply that the reduced status of FDCs in aged mice significantly impairs the early TSE agent accumulation in lymphoid tissues and subsequent neuroinvasion. Furthermore, the inefficient neuroinvasion in aged individuals may lead to significant levels of subclinical TSE disease in the population.

The costs of publication of this article were defrayed in part by the payment of page charges. This article must therefore be hereby marked advertisement in accordance with 18 U.S.C. Section 1734 solely to indicate this fact.

1 This work was supported by funding from the European Commission, UK Biotechnology and Biological Sciences Research Council, and the Medical Research Council.

2 Address correspondence and reprint requests to Dr. Karen L. Brown and Dr. Neil A. Mabbott, The Roslin Institute and Royal (Dick) School of Veterinary Sciences, University of Edinburgh, Roslin, Midlothian EH25 9PS, U.K. E-mail: mhtml:%7B33B38F65-8D2E-434D-8F9B-8BDCD77D3066%7Dmid://00000428/!x-usc:mailto:karen.brown@roslin.ed.ac.uk and mhtml:%7B33B38F65-8D2E-434D-8F9B-8BDCD77D3066%7Dmid://00000428/!x-usc:mailto:neil.mabbott@roslin.ed.ac.uk

3 Abbreviations used in this paper: TSE, transmissible spongiform encephalopathy; FDC, follicular dendritic cell; PrP, prion protein; vCJD, variant Creutzfeldt-Jakob disease; BSE, bovine spongiform encephalopathy; CR, complement receptor; i.c., intracerebral; TH, tyrosine hydroxylase; PK, proteinase K.

4 The online version of this article contains supplemental material.

http://www.jimmunol.org/cgi/content/abstract/183/8/5199?maxtoshow=&HITS=10&hits=10&RESULTFORMAT=&fulltext=prion&searchid=1&FIRSTINDEX=0&volume=183&issue=8&resourcetype=HWCIT

>>>In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — <<<

>> 95%) downer or dead dairy cattle and a few horses. Sheep had never been fed.

We believe that these findings may indicate the presence of a previously unrecognized scrapie-like disease in cattle and wish to alert dairy practitioners to this possibility.

snip...

PROCEEDINGS OF THE SEVENTH ANNUAL WESTERN CONFERENCE FOR FOOD ANIMAL VETERINARY MEDICINE, University of Arizona, March 17-19, 1986

http://web.archive.org/web/20030331063559/http://www.bseinquiry.gov.uk/files/mb/m09a/tab01.pdf

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

IS THERE A SCRAPIE-LIKE DISEASE IN CATTLE ?

YOU BET THERE IS, AND HAS BEEN, AND WE BEEN FEEDING THE MOST HIGH RISK I.E. DEAD STOCK DOWNER COWS TO OUR CHILDREN FOR DECADES, who will follow these children for human TSE from mad cow disease here in the USA in the years, decades to come, and how many will they expose from the 'pass it forward' friendly fire modes ???

http://downercattle.blogspot.com/2008/12/evaluation-of-fsis-management-controls.html

Saturday, May 2, 2009

U.S. GOVERNMENT SUES WESTLAND/HALLMARK MEAT OVER USDA CERTIFIED DEADSTOCK DOWNER COW SCHOOL LUNCH PROGRAM

http://downercattle.blogspot.com/2009/05/us-government-sues-westlandhallmark.html

Thursday, May 1, 2008 DEAD STOCK DOWNER COW BAN i.e. non-ambulatory policy still not changed by USDA May 1, 2008

http://downercattle.blogspot.com/2008/05/dead-stock-downer-cow-ban-ie-non.html

http://stanford.wellsphere.com/cjd-article/usda-certified-h-base-mad-cow-school-lunch-program/641216

Sunday, May 17, 2009

WHO WILL WATCH THE CHILDREN ? SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 – February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

Approximately 50.3 million pounds of the beef recalled by HallmarkNVestland went to federal nutrition programs, including the National School Lunch Program, and of those 50.3 million pounds, about 19.6 million pounds had already been consumed at the time the recall was issued. Release No. 0054.08, USDA, Transcript of Technical Briefing - HallmarldWestland Meat Packing Company (Feb. 21, 2008).

9. HSUS members that consume meat products, including beef products, are concerned about eating adulterated meat products and the health risks associated with such adulterated meat. Specifically, they are concerned that downed cattle are at an increased risk for harboring and transmitting BSE prions and other pathogens. The consumption of meat products derived from BSE-infected cattle is believed to cause a human neurological disease known as variant Creutzfeldt-Jakob disease ("vCJD"). The disease is progressive, invariably fatal, and there is no known effective treatment or cure. Downed cattle may also be at higher risk for harboring other foodborne transmissible pathogens, including E. coli 0157:H7, Salmonella, and anthrax. By allowing downed cattle to enter the food supply, USDA's regulatory loophole injures members of The HSUS by placing them at an increased risk of contracting these food-borne illnesses each time they eat beef. 10. Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

United States of America Ex rel. The Humane Society of the United States v. Hallmark Meat Packing Company, Westland Meat Company Inc. (Downed animal abuse/government fraud)

Court or Agency: United States District Court for the Central District of California Plaintiff(s): United States of America Ex rel. The Humane Society of the United States Defendant: Hallmark Meat Packing Company, Westland Meat Company Inc. HSUS Counsel: Peter J. Petersan, Leana Stormont Outside Counsel: Milbank, Tweed, Hadley & McCloy LLP Status: In Briefing

Federal court action under the False Claims Act alleging that Westland/Hallmark defrauded the federal government by violating the terms of its school lunch program contracts requiring the humane handling of animals. The lawsuit was the result of an investigation by The HSUS which exposed the facility's mistreatment of animals too sick or injured to walk and led to the largest meat recall in the nation’s history.

http://www.hsus.org/in_the_courts/docket/us_hallmark.html

http://www.law.gmu.edu/assets/files/academics/schedule/2009/fall/HENRYGREEN_AnimalLaw20090818.pdf

http://www.hsus.org/farm/news/ournews/dept_of_justice_hallmark_050409.html

FOIA

http://www.fsis.usda.gov/PDF/FOIA_Requests_0308.pdf

"California Firm Recalls Beef Products". USDA. February 17, 2008.

http://www.fsis.usda.gov/PDF/Recall_005-2008_Release.pdf.

(PDF)

AP (February 17, 2008). "USDA recalls 143 million pounds of beef". MSNBC.

http://www.msnbc.msn.com/id/23212514/.

"Statement by Secretary of Agriculture Ed Schafer Regarding Animal Cruelty Charges Filed Against Employees at Hallmark/Westland Meat Packing Company". USDA. February 15, 2008.

http://www.usda.gov/wps/portal/!ut/p/_s.7_0_A/7_0_1OB?contentidonly=true&contentid=2008/02/0044.xml.

"USDA Q&A". USDA. February 19, 2008.

http://www.usda.gov/wps/portal/usdahome?contentidonly=true&contentid=Recall_Information.xml.

The Case is captioned as United States of America ex rel. The Humane Society of the United States v. Hallmark Meat Packing Company; Westland Meat Company, Inc.

http://www.hsus.org/farm/news/ournews/dept_of_justice_hallmark_050409.html

http://www.econ.iastate.edu/classes/econ362/hallam/NewspaperArticles/DownerCows.pdf

Thursday, September 24, 2009

(09-24) 15:00 PDT LOS ANGELES (AP) --

A Southern California meatpacking plant that supplied beef to the nation's school lunch program slaughtered stumbling, potentially contaminated cows for four years before undercover video of animal abuse prompted a massive beef recall, federal court filings say...

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

also ;

http://www.usda.gov/oig/webdocs/24601-07-KC.pdf

>>>USDA officials cited three other interlocking safeguards that protect the public even if other safeguards, such as ante-mortem inspection, should fail; these safeguards are the removal of Specified Risk Materials (SRM),5 BSE surveillance testing, and the feed ban.6 Under the Federal Meat Inspection Act (FMIA),7<<< href="http://madcowfeed.blogspot.com/2009/11/bse-feed-recall-misbranding-of-product.html">http://madcowfeed.blogspot.com/2009/11/bse-feed-recall-misbranding-of-product.html

Friday, September 4, 2009

FOIA REQUEST ON FEED RECALL PRODUCT 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009

http://madcowfeed.blogspot.com/2009/09/foia-request-on-feed-recall-product.html

Saturday, August 29, 2009 FOIA REQUEST FEED RECALL 2009 Product may have contained prohibited materials Bulk Whole Barley, Recall # V-256-2009

http://madcowfeed.blogspot.com/2009/08/foia-request-feed-recall-2009-product.html

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html

"Everything they did on the Texas cow makes everything USDA did before 2005 suspect," Brown said. ...snip...end

http://www.upi.com/ConsumerHealthDaily/view.php?StoryID=20060315-055557-1284r

PAUL BROWN COMMENT TO ME ON THIS ISSUE Tuesday, September 12, 2006 11:10 AM "Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency."

http://madcowtesting.blogspot.com/

see full text sporadic CJD the big lie;

http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0705&L=sanet-mg&T=0&F=&S=&P=135027

PLUS

http://cjdmadcowbaseoct2007.blogspot.com/2008/07/novel-human-disease-with-abnormal-prion.html

Sunday, August 10, 2008 A New Prionopathy OR more of the same old BSe and sporadic CJD

http://creutzfeldt-jakob-disease.blogspot.com/2008/08/new-prionopathy-or-more-of-same-old-bse.html

HUMAN and ANIMAL TSE Classifications i.e. mad cow disease and the UKBSEnvCJD only theory

http://www.regulations.gov/search/Regs/contentStreamer?objectId=090000648027c28e&disposition=attachment&contentType=pdf

AS I SAID BEFORE, WHO WATCH THE CHILDREN FOR CJD FOR THE NEXT 5 DECADES ???

Thursday, October 15, 2009 CVM Annual Report Fiscal Year 2008: October 1, 2007-September 30, 2008 (BSE)

http://madcowusda.blogspot.com/2009/10/cvm-annual-report-fiscal-year-2008.html

Sunday, September 6, 2009

MAD COW USA 1997 SECRET VIDEO

http://madcowusda.blogspot.com/2009/09/mad-cow-usa-1997-video.html

U.S.A. HIDING MAD COW DISEASE VICTIMS AS SPORADIC CJD ? see video at bottom

http://creutzfeldt-jakob-disease.blogspot.com/2009/07/usa-hiding-mad-cow-disease-victims-as.html

DAMNING TESTIMONY FROM STANLEY PRUSINER THE NOBEL PEACE PRIZE WINNER ON PRIONS SPEAKING ABOUT ANN VENEMAN see video

http://maddeer.org/video/embedded/prusinerclip.html

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html

please read and understand this. the UKBSEnvCJD only theory was bogus.

http://creutzfeldt-jakob-disease.blogspot.com/2009/08/characteristics-of-established-and.html

Office of Inspector General Semiannual Report to Congress FY 2007 - 2nd Half

Two Texas Companies Sentenced and Fined for Misbranding Meat Products In April 2007, two closely held and related Texas companies pled guilty in Federal court and were sentenced to 12 months of probation and ordered to pay $10,250 in fines for misbranding meat products. One of the companies sold adulterated meat products to a retail store in New Mexico. Additionally, portions of the invoices failed to properly and consistently identify the meat products as being from cattle more than 30 months old at time of slaughter. This information is required to be disclosed because of bovine spongiform encephalopathy (BSE, or "mad cow disease") concerns. No adulterated meat reached consumers.

http://www.usda.gov/oig/webdocs/sarc071212.pdf

Saturday, August 29, 2009

FOIA REQUEST FEED RECALL 2009 Product may have contained prohibited materials Bulk Whole Barley, Recall # V-256-2009

http://madcowfeed.blogspot.com/2009/08/foia-request-feed-recall-2009-product.html

Friday, September 4, 2009

FOIA REQUEST ON FEED RECALL PRODUCT 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009

http://madcowfeed.blogspot.com/2009/09/foia-request-on-feed-recall-product.html

THIS recall is not confusing ;

10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. BLOOD LACED MBM IN COMMERCE USA 2007

Date: March 21, 2007 at 2:27 pm PST

RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II

___________________________________

PRODUCT

Bulk cattle feed made with recalled Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007

CODE

Cattle feed delivered between 01/12/2007 and 01/26/2007

RECALLING FIRM/MANUFACTURER

Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.

Firm initiated recall is ongoing.

REASON

Blood meal used to make cattle feed was recalled because it was cross- contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement.

VOLUME OF PRODUCT IN COMMERCE

42,090 lbs.

DISTRIBUTION

WI

___________________________________

PRODUCT

Custom dairy premix products: MNM ALL PURPOSE Pellet, HILLSIDE/CDL Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal, TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML W/SMARTA, Recall # V-025-2007

CODE

The firm does not utilize a code - only shipping documentation with commodity and weights identified.

RECALLING FIRM/MANUFACTURER

Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007. Firm initiated recall is complete.

REASON

Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement.

VOLUME OF PRODUCT IN COMMERCE

9,997,976 lbs.

DISTRIBUTION

ID and NV

END OF ENFORCEMENT REPORT FOR MARCH 21, 2007

http://www.fda.gov/bbs/topics/enforce/2007/ENF00996.html

NEW URL

http://www.fda.gov/Safety/Recalls/EnforcementReports/2007/ucm120446.htm

Thursday, March 19, 2009

MILLIONS AND MILLIONS OF POUNDS OF MAD COW FEED IN COMMERCE USA WITH ONGOING 12 YEARS OF DENIAL

http://madcowfeed.blogspot.com/2009/03/millions-and-millions-of-pounds-of-mad.html

Tuesday, July 14, 2009

U.S. Emergency Bovine Spongiform Encephalopathy Response Plan Summary and BSE Red Book Date: February 14, 2000 at 8:56 am PST

WHERE did we go wrong $$$

http://madcowtesting.blogspot.com/2009/07/us-emergency-bovine-spongiform.html

Sunday, December 28, 2008

MAD COW DISEASE USA DECEMBER 28, 2008 an 8 year review of a failed and flawed policy

http://bse-atypical.blogspot.com/2008/12/mad-cow-disease-usa-december-28-2008-8.html

Wednesday, August 20, 2008

Bovine Spongiform Encephalopathy Mad Cow Disease typical and atypical strains, was there a cover-up ? August 20, 2008

http://bse-atypical.blogspot.com/2008/08/bovine-spongiform-encephalopathy-mad.html

Saturday, June 13, 2009

Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States 2003 revisited 2009

http://cjdusa.blogspot.com/2009/06/monitoring-occurrence-of-emerging-forms.html

Tuesday, August 11, 2009

Characteristics of Established and Proposed Sporadic Creutzfeldt-Jakob Disease Variants

Brian S. Appleby, MD; Kristin K. Appleby, MD; Barbara J. Crain, MD, PhD; Chiadi U. Onyike, MD, MHS; Mitchell T. Wallin, MD, MPH; Peter V. Rabins, MD, MPH

Background: The classic Creutzfeldt-Jakob disease (CJD), Heidenhain, and Oppenheimer-Brownell variants are sporadic CJD (sCJD) phenotypes frequently described in the literature, but many cases present with neuropsychiatric symptoms, suggesting that there may be additional sCJD phenotypes.

Objective: To characterize clinical, diagnostic, and molecular features of 5 sCJD variants.

Design: Retrospective analysis.

Setting: The Johns Hopkins and Veterans Administration health care systems.

Participants: Eighty-eight patients with definite or probable sCJD.

Main Outcome Measures: Differences in age at onset, illness progression, diagnostic test results, and molecular subtype.

Results: The age at onset differed among sCJD variants (P=.03); the affective variant had the youngest mean age at onset (59.7 years). Survival time (P.001) and the time to clinical presentation (P=.003) differed among groups. Patients with the classic CJD phenotype had the shortest median survival time from symptom onset (66 days) and those who met criteria for the affective sCJD variant had the longest (421 days) and presented to clinicians significantly later (median time from onset to presentation, 92 days; P=.004). Cerebrospinal fluid analyses were positive for 14-3-3 protein in all of the affective variants, regardless of illness duration. Periodic sharp-wave complexes were not detected on any of the electroencephalography tracings in the Oppenheimer-Brownell group; basal ganglia hyperintensity was not detected on brain magnetic resonance imaging in this group either. All of the Heidenhain variants were of the methionine/ methionine type 1 molecular subtype.

Conclusions: The classic CJD phenotype and the Heidenhain, Oppenheimer-Brownell, cognitive, and affective sCJD variants differ by age at disease onset, survival time, and diagnostic test results. Characteristics of these 5 phenotypes are provided to facilitate further clinicopathologic investigation that may lead to more reliable and timely diagnoses of sCJD.

Arch Neurol. 2009;66(2):208-215

snip...

COMMENT

snip...see full text ;

http://creutzfeldt-jakob-disease.blogspot.com/2009/08/characteristics-of-established-and.html

Thursday, September 24, 2009

Suit: Meatpacker used `downer' cows for 4 years TO FEED OUT CHILDREN ALL ACROSS THE NATION, the most high risk for mad cow disease

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

Monday, May 11, 2009

Rare BSE mutation raises concerns over risks to public health

http://bse-atypical.blogspot.com/2009/05/rare-bse-mutation-raises-concerns-over.html

Tuesday, July 14, 2009

U.S. Emergency Bovine Spongiform Encephalopathy Response Plan Summary and BSE Red Book Date: February 14, 2000 at 8:56 am PST

WHERE did we go wrong $$$

http://madcowtesting.blogspot.com/2009/07/us-emergency-bovine-spongiform.html

Sunday, June 07, 2009

L-TYPE-BSE, H-TYPE-BSE, C-TYPE-BSE, IBNC-TYPE-BSE, TME, CWD, SCRAPIE, CJD, NORTH AMERICA

http://bse-atypical.blogspot.com/2009/06/l-type-bse-h-type-bse-c-type-bse-ibnc.html

Sunday, May 10, 2009

Identification and characterization of bovine spongiform encephalopathy cases diagnosed and NOT diagnosed in the United States

http://bse-atypical.blogspot.com/2009/05/identification-and-characterization-of.html

Docket APHIS-2006-0026 Docket Title Bovine Spongiform Encephalopathy; Animal Identification and Importation of Commodities Docket Type Rulemaking Document APHIS-2006-0026-0001 Document Title Bovine Spongiform Encephalopathy; Minimal-Risk Regions, Identification of Ruminants and Processing and Importation of Commodities Public Submission APHIS-2006-0026-0012 Public Submission Title Comment from Terry S Singletary

http://www.regulations.gov/fdmspublic/component/main?main=DocumentDetail&o=09000064801e47e1

Docket APHIS-2006-0041 Docket Title Bovine Spongiform Encephalopathy; Minimal-Risk Regions; Importation of Live Bovines and Products Derived from Bovines Commodities Docket Type Rulemaking Document APHIS-2006-0041-0001 Document Title Bovine Spongiform Encephalopathy; Minimal-Risk Regions; Importation of Live Bovines and Products Derived From Bovines Public Submission APHIS-2006-0041-0028 Public Submission Title Comment from Terry S Singletary

Comment 2006-2007 USA AND OIE POISONING GLOBE WITH BSE MRR POLICY

THE USA is in a most unique situation, one of unknown circumstances with human and animal TSE. THE USA has the most documented TSE in different species to date, with substrains growing in those species (BSE/BASE in cattle and CWD in deer and elk, there is evidence here with different strains), and we know that sheep scrapie has over 20 strains of the typical scrapie with atypical scrapie documented and also BSE is very likely to have passed to sheep. all of which have been rendered and fed back to animals for human and animal consumption, a frightening scenario. WE do not know the outcome, and to play with human life around the globe with the very likely TSE tainted products from the USA, in my opinion is like playing Russian roulette, of long duration, with potential long and enduring consequences, of which once done, cannot be undone. These are the facts as I have come to know through daily and extensive research of TSE over 9 years, since 12/14/97. I do not pretend to have all the answers, but i do know to continue to believe in the ukbsenvcjd only theory of transmission to humans of only this one strain from only this one TSE from only this one part of the globe, will only lead to further failures, and needless exposure to humans from all strains of TSE, and possibly many more needless deaths from TSE via a multitude of proven routes and sources via many studies with primates and rodents and other species.

MY personal belief, since you ask, is that not only the Canadian border, but the USA border, and the Mexican border should be sealed up tighter than a drum for exporting there TSE tainted products, until a validated, 100% sensitive test is available, and all animals for human and animal consumption are tested. all we are doing is the exact same thing the UK did with there mad cow poisoning when they exported it all over the globe, all the while knowing what they were doing. this BSE MRR policy is nothing more than a legal tool to do just exactly what the UK did, thanks to the OIE and GW, it's legal now. and they executed Saddam for poisoning ???

go figure. ...

http://www.regulations.gov/fdmspublic/component/main?main=DocumentDetail&o=09000064801f8151

Docket APHIS-2006-0041 Docket Title Bovine Spongiform Encephalopathy; Minimal-Risk Regions; Importation of Live Bovines and Products Derived from Bovines Commodities Docket Type Rulemaking Document APHIS-2006-0041-0001 Document Title Bovine Spongiform Encephalopathy; Minimal-Risk Regions; Importation of Live Bovines and Products Derived From Bovines Public Submission APHIS-2006-0041-0028.1 Public Submission Title Attachment to Singletary comment

January 28, 2007

Greetings APHIS,

I would kindly like to submit the following to ;

BSE; MRR; IMPORTATION OF LIVE BOVINES AND PRODUCTS DERIVED FROM BOVINES [Docket No. APHIS-2006-0041] RIN 0579-AC01

http://www.regulations.gov/fdmspublic/ContentViewer?objectId=09000064801f8152&disposition=attachment&contentType=msw8

Sunday, August 10, 2008

A New Prionopathy OR more of the same old BSe and sporadic CJD

http://creutzfeldt-jakob-disease.blogspot.com/2008/08/new-prionopathy-or-more-of-same-old-bse.html

SEAC OCTOBER 2009

• Are some commoner types of neurodegenerative disease (including Alzheimer's disease and Parkinson's disease) also transmissible? Some recent scientific research has suggested this possibility

http://www.seac.gov.uk/pdf/hol-response091008.pdf

Thursday, February 26, 2009

'Harmless' prion protein linked to Alzheimer's disease Non-infectious form of prion protein could cause brain degeneration ???

http://betaamyloidcjd.blogspot.com/2009/02/harmless-prion-protein-linked-to.html

CJD1/9 0185

Ref: 1M51A

IN STRICT CONFIDENCE

TRANSMISSION OF ALZHEIMER-TYPE PLAQUES TO PRIMATES

1. CMO will wish to be aware that a meeting was held at DH yesterday, 4 January, to discuss the above findings. It was chaired by Professor Murray (Chairman of the MRC Co-ordinating Committee on Research in the Spongiform Encephalopathies in Man), and attended by relevant experts in the fields of Neurology, Neuropathology, molecular biology, amyloid biochemistry, and the spongiform encephalopathies, and by representatives of the MRC and AFRC.

2. Briefly, the meeting agreed that:

i) Dr Ridley et als findings of experimental induction of p amyloid in primates were valid, interesting and a significant advance in the understanding of neurodegeneradve disorders;

ii) there were no immediate implications for the public health, and no further safeguards were thought to be necessary at present; and

iii) additional research was desirable, both epidemiological and at the molecular level. Possible avenues are being followed up by DH and the MRC, but the details will require further discussion.

93/01.05/4.1tss

http://www.bseinquiry.gov.uk/files/yb/1993/01/05004001.pdf

Regarding Alzheimer's disease

(note the substantial increase on a yearly basis)

http://www.bseinquiry.gov.uk/files/yb/1988/07/08014001.pdf

snip...

The pathogenesis of these diseases was compared to Alzheimer's disease at a molecular level...

snip...

http://www.bseinquiry.gov.uk/files/yb/1990/03/12003001.pdf

And NONE of this is relevant to BSE?

There is also the matter whether the spectrum of ''prion disease'' is wider than that recognized at present.

http://www.bseinquiry.gov.uk/files/yb/1990/07/06005001.pdf

???

http://www.bseinquiry.gov.uk/files/yb/1990/07/09001001.pdf

BSE101/1 0136

IN CONFIDENCE

5 NOV 1992

CMO From: Dr J S Metters DCMO 4 November 1992

TRANSMISSION OF ALZHEIMER TYPE PLAQUES TO PRIMATES

http://www.bseinquiry.gov.uk/files/yb/1992/11/04001001.pdf

also, see the increase of Alzheimer's from 1981 to 1986

http://www.bseinquiry.gov.uk/files/yb/1988/07/08014001.pdf

Tuesday, August 26, 2008

Alzheimer's Transmission of AA-amyloidosis: Similarities with Prion Disorders NEUROPRION 2007 FC4.3

http://betaamyloidcjd.blogspot.com/2008/08/alzheimers-transmission-of-aa.html

see full text ;

http://betaamyloidcjd.blogspot.com/2009/02/harmless-prion-protein-linked-to.html

Alzheimer's and CJD

http://betaamyloidcjd.blogspot.com/

Saturday, March 22, 2008

10 Million Baby Boomers to have Alzheimer's in the coming decades 2008 Alzheimer's disease facts and figures

http://betaamyloidcjd.blogspot.com/2008/03/association-between-deposition-of-beta.html

re-Association between Deposition of Beta-Amyloid and Pathological Prion Protein in Sporadic Creutzfeldt-Jakob Disease

http://betaamyloidcjd.blogspot.com/2008/04/re-association-between-deposition-of.html

Thursday, December 04, 2008 2:37 PM

"we have found that H-BSE can infect humans."

personal communication with Professor Kong. ...TSS

see full text ;

http://bse-atypical.blogspot.com/2009/02/atypical-bse-north-america-update.html

Terry S. Singeltary Sr.

P.O. Box 42

Bacliff, Texas USA 77518

Suit: Meatpacker used `downer' cows for 4 years TO FEED OUT CHILDREN ALL ACROSS THE NATION, the most high risk for mad cow disease

2009-09-24T18:35:00.000-07:00

Suit: Meatpacker used `downer' cows for 4 years TO FEED OUT CHILDREN ALL ACROSS THE NATION, THE MOST HIGH RISK COW FOR MAD COW DISEASE, i.e. DEADSTOCK DOWNER DOWS.

who will follow the children ???

Suit: Meatpacker used `downer' cows for 4 years

By GILLIAN FLACCUS, Associated Press Writer

Thursday, September 24, 2009

(09-24) 15:00 PDT LOS ANGELES (AP) --

A Southern California meatpacking plant that supplied beef to the nation's school lunch program slaughtered stumbling, potentially contaminated cows for four years before undercover video of animal abuse prompted a massive beef recall, federal court filings say.

The amended complaint filed late last month in U.S. District Court in Riverside is part of an ongoing civil lawsuit filed by The Humane Society of the United States against the Chino-based Westland/Hallmark Meat Co.

The U.S. Department of Justice intervened in the case with the new complaint after months of additional research and interviews that uncovered the startling new allegations against the now-defunct packing plant. Among them, the company failed to disclose that one of its partners had two felony convictions related to illegal industry practices.

The Humane Society released video in late 2007 showing "downer" cows — animals too weak or sick to walk — being dragged by chains, rammed by forklifts and sprayed with high-pressure water by plant employees who wanted them to stand for processing.

The video sparked the largest beef recall in U.S. history. Officials estimated at the time that 37 million pounds of the 143 million pounds of recalled beef went to school lunch programs, and most of the meat had already been eaten.

Donald W. Hallmark, whom the lawsuit lists as a company partner, told The Associated Press he retired six years ago and had no comment. Calls to a number for another partner, Steve Mendell, rang unanswered Thursday.

The new filing alleges the government paid Westland/Hallmark millions of dollars to which it was not entitled because the company lied about meeting all the conditions for the 140 government contracts it held between 2003 and 2008.

The lawsuit seeks unspecified damages and a jury trial. The original filing by the Humane Society sought $150 million.

In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — at the average rate of one every six weeks and abused animals daily using chains, forklifts, high-pressure water hoses or electric prods, or by punching and kicking them.

Downer cows pose increased risk for mad cow disease, E. coli and other infections, partly because they typically wallow in feces.

The new complaint also alleges that a partner at Westland/Hallmark, Aaron Magidow, had two felony convictions that were not reported to the government when it awarded the meatpacker's contracts. Magidow, who has since died, was convicted in 1974 of bribing federal meat inspectors and in 1983 for participating in a fraudulent meat buying scheme, the lawsuit said.

The executor of Magidow's estate, which is named as a defendant in the case, said he had no comment.

"I think whatever we have to say will be said in court," said attorney Walter Weiss.

Officials with the Humane Society said the government's filing marked the first time the Justice Department had intervened in a federal false claims case involving the mistreatment of farm animals.

The lawsuit "not only confirms everything that our investigation found, but suggests that it was even more widespread than we had documented," said Jonathan Lovvorn, the Humane Society's chief counsel. "We're concerned by what they uncovered — but not surprised."

San Bernardino County prosecutors charged two of the employees seen on the undercover video. One was sentenced to six months in jail, the other to nine months in jail.

http://www.sfgate.com/cgi-bin/article.cgi?f=/n/a/2009/09/24/national/a130509D56.DTL&tsp=1

>>>In the papers, the government alleges the meatpacking plant slaughtered and processed downer cows for nearly four years — from January 2004 to September 2007 — <<<

>> 95%) downer or dead dairy cattle and a few horses. Sheep had never been fed.

We believe that these findings may indicate the presence of a previously unrecognized scrapie-like disease in cattle and wish to alert dairy practitioners to this possibility.

snip...

PROCEEDINGS OF THE SEVENTH ANNUAL WESTERN CONFERENCE FOR FOOD ANIMAL VETERINARY MEDICINE, University of Arizona, March 17-19, 1986

http://web.archive.org/web/20030331063559/http://www.bseinquiry.gov.uk/files/mb/m09a/tab01.pdf

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

IS THERE A SCRAPIE-LIKE DISEASE IN CATTLE ?

YOU BET THERE IS, AND HAS BEEN, AND WE BEEN FEEDING THE MOST HIGH RISK I.E. DEAD STOCK DOWNER COWS TO OUR CHILDREN FOR DECADES, who will follow these children for human TSE from mad cow disease here in the USA in the years, decades to come, and how many will they expose from the 'pass it forward' friendly fire modes ???

http://downercattle.blogspot.com/2008/12/evaluation-of-fsis-management-controls.html

Saturday, May 2, 2009

U.S. GOVERNMENT SUES WESTLAND/HALLMARK MEAT OVER USDA CERTIFIED DEADSTOCK DOWNER COW SCHOOL LUNCH PROGRAM

http://downercattle.blogspot.com/2009/05/us-government-sues-westlandhallmark.html

Thursday, May 1, 2008 DEAD STOCK DOWNER COW BAN i.e. non-ambulatory policy still not changed by USDA May 1, 2008

http://downercattle.blogspot.com/2008/05/dead-stock-downer-cow-ban-ie-non.html

http://stanford.wellsphere.com/cjd-article/usda-certified-h-base-mad-cow-school-lunch-program/641216

Sunday, May 17, 2009

WHO WILL WATCH THE CHILDREN ? SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

http://downercattle.blogspot.com/2009/05/who-will-watch-children.html

http://downercattle.blogspot.com/

FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 – February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

Approximately 50.3 million pounds of the beef recalled by HallmarkNVestland went to federal nutrition programs, including the National School Lunch Program, and of those 50.3 million pounds, about 19.6 million pounds had already been consumed at the time the recall was issued. Release No. 0054.08, USDA, Transcript of Technical Briefing - HallmarldWestland Meat Packing Company (Feb. 21, 2008).

9. HSUS members that consume meat products, including beef products, are concerned about eating adulterated meat products and the health risks associated with such adulterated meat. Specifically, they are concerned that downed cattle are at an increased risk for harboring and transmitting BSE prions and other pathogens. The consumption of meat products derived from BSE-infected cattle is believed to cause a human neurological disease known as variant Creutzfeldt-Jakob disease ("vCJD"). The disease is progressive, invariably fatal, and there is no known effective treatment or cure. Downed cattle may also be at higher risk for harboring other foodborne transmissible pathogens, including E. coli 0157:H7, Salmonella, and anthrax. By allowing downed cattle to enter the food supply, USDA's regulatory loophole injures members of The HSUS by placing them at an increased risk of contracting these food-borne illnesses each time they eat beef. 10. Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

United States of America Ex rel. The Humane Society of the United States v. Hallmark Meat Packing Company, Westland Meat Company Inc. (Downed animal abuse/government fraud)

Court or Agency: United States District Court for the Central District of California Plaintiff(s): United States of America Ex rel. The Humane Society of the United States Defendant: Hallmark Meat Packing Company, Westland Meat Company Inc. HSUS Counsel: Peter J. Petersan, Leana Stormont Outside Counsel: Milbank, Tweed, Hadley & McCloy LLP Status: In Briefing

Federal court action under the False Claims Act alleging that Westland/Hallmark defrauded the federal government by violating the terms of its school lunch program contracts requiring the humane handling of animals. The lawsuit was the result of an investigation by The HSUS which exposed the facility's mistreatment of animals too sick or injured to walk and led to the largest meat recall in the nation’s history.

http://www.hsus.org/in_the_courts/docket/us_hallmark.html

http://www.law.gmu.edu/assets/files/academics/schedule/2009/fall/HENRYGREEN_AnimalLaw20090818.pdf

http://www.hsus.org/farm/news/ournews/dept_of_justice_hallmark_050409.html

FOIA

http://www.fsis.usda.gov/PDF/FOIA_Requests_0308.pdf

"California Firm Recalls Beef Products". USDA. February 17, 2008.

http://www.fsis.usda.gov/PDF/Recall_005-2008_Release.pdf

AP (February 17, 2008). "USDA recalls 143 million pounds of beef". MSNBC.

http://www.msnbc.msn.com/id/23212514/

"Statement by Secretary of Agriculture Ed Schafer Regarding Animal Cruelty Charges Filed Against Employees at Hallmark/Westland Meat Packing Company". USDA. February 15, 2008.

http://www.usda.gov/wps/portal/!ut/p/_s.7_0_A/7_0_1OB?contentidonly=true&contentid=2008/02/0044.xml

"USDA Q&A". USDA. February 19, 2008.

http://www.usda.gov/wps/portal/usdahome?contentidonly=true&contentid=Recall_Information.xml

The Case is captioned as United States of America ex rel. The Humane Society of the United States v. Hallmark Meat Packing Company; Westland Meat Company, Inc.

http://www.hsus.org/farm/news/ournews/dept_of_justice_hallmark_050409.html

http://www.econ.iastate.edu/classes/econ362/hallam/NewspaperArticles/DownerCows.pdf

Thursday, September 24, 2009

(09-24) 15:00 PDT LOS ANGELES (AP) --

A Southern California meatpacking plant that supplied beef to the nation's school lunch program slaughtered stumbling, potentially contaminated cows for four years before undercover video of animal abuse prompted a massive beef recall, federal court filings say...

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

also ;

http://www.usda.gov/oig/webdocs/24601-07-KC.pdf

>>>USDA officials cited three other interlocking safeguards that protect the public even if other safeguards, such as ante-mortem inspection, should fail; these safeguards are the removal of Specified Risk Materials (SRM),5 BSE surveillance testing, and the feed ban.6 Under the Federal Meat Inspection Act (FMIA),7<<< href="http://madcowfeed.blogspot.com/2009/11/bse-feed-recall-misbranding-of-product.html">http://madcowfeed.blogspot.com/2009/11/bse-feed-recall-misbranding-of-product.html

Friday, September 4, 2009

FOIA REQUEST ON FEED RECALL PRODUCT 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009

http://madcowfeed.blogspot.com/2009/09/foia-request-on-feed-recall-product.html

Saturday, August 29, 2009

FOIA REQUEST FEED RECALL 2009 Product may have contained prohibited materials Bulk Whole Barley, Recall # V-256-2009

http://madcowfeed.blogspot.com/2009/08/foia-request-feed-recall-2009-product.html

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html

PAUL BROWN CDC

"Everything they did on the Texas cow makes everything USDA did before 2005 suspect," Brown said. ...snip...end

http://www.upi.com/ConsumerHealthDaily/view.php?StoryID=20060315-055557-1284r

PAUL BROWN COMMENT TO ME ON THIS ISSUE Tuesday, September 12, 2006 11:10 AM "Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency."

http://madcowtesting.blogspot.com/

see full text sporadic CJD the big lie;

http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0705&L=sanet-mg&T=0&F=&S=&P=135027

PLUS

http://cjdmadcowbaseoct2007.blogspot.com/2008/07/novel-human-disease-with-abnormal-prion.html

Sunday, August 10, 2008

A New Prionopathy OR more of the same old BSe and sporadic CJD

http://creutzfeldt-jakob-disease.blogspot.com/2008/08/new-prionopathy-or-more-of-same-old-bse.html

HUMAN and ANIMAL TSE Classifications i.e. mad cow disease and the UKBSEnvCJD only theory

http://www.regulations.gov/search/Regs/contentStreamer?objectId=090000648027c28e&disposition=attachment&contentType=pdf

AS I SAID BEFORE, WHO WATCH THE CHILDREN FOR CJD FOR THE NEXT 5 DECADES ???

Sunday, September 6, 2009 MAD COW USA 1997 SECRET VIDEO

http://madcowusda.blogspot.com/2009/09/mad-cow-usa-1997-video.html

U.S.A. HIDING MAD COW DISEASE VICTIMS AS SPORADIC CJD ? see video at bottom

http://creutzfeldt-jakob-disease.blogspot.com/2009/07/usa-hiding-mad-cow-disease-victims-as.html

DAMNING TESTIMONY FROM STANLEY PRUSINER THE NOBEL PEACE PRIZE WINNER ON PRIONS SPEAKING ABOUT ANN VENEMAN

http://maddeer.org/video/embedded/prusinerclip.html

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html

http://creutzfeldt-jakob-disease.blogspot.com/2009/08/characteristics-of-established-and.html

Office of Inspector General Semiannual Report to Congress FY 2007 – 2nd Half

Two Texas Companies Sentenced and Fined for Misbranding Meat Products In April 2007, two closely held and related Texas companies pled guilty in Federal court and were sentenced to 12 months of probation and ordered to pay $10,250 in fines for misbranding meat products. One of the companies sold adulterated meat products to a retail store in New Mexico. Additionally, portions of the invoices failed to properly and consistently identify the meat products as being from cattle more than 30 months old at time of slaughter. This information is required to be disclosed because of bovine spongiform encephalopathy (BSE, or “mad cow disease”) concerns. No adulterated meat reached consumers.

http://www.usda.gov/oig/webdocs/sarc071212.pdf

Tuesday, July 14, 2009

U.S. Emergency Bovine Spongiform Encephalopathy Response Plan Summary and BSE Red Book Date: February 14, 2000 at 8:56 am PST

WHERE did we go wrong $$$

http://madcowtesting.blogspot.com/2009/07/us-emergency-bovine-spongiform.html

Sunday, December 28, 2008

MAD COW DISEASE USA DECEMBER 28, 2008 an 8 year review of a failed and flawed policy

http://bse-atypical.blogspot.com/2008/12/mad-cow-disease-usa-december-28-2008-8.html

Wednesday, August 20, 2008

Bovine Spongiform Encephalopathy Mad Cow Disease typical and atypical strains, was there a cover-up ? August 20, 2008

http://bse-atypical.blogspot.com/2008/08/bovine-spongiform-encephalopathy-mad.html

Saturday, June 13, 2009

Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States 2003 revisited 2009

http://cjdusa.blogspot.com/2009/06/monitoring-occurrence-of-emerging-forms.html

Tuesday, August 11, 2009

Characteristics of Established and Proposed Sporadic Creutzfeldt-Jakob Disease Variants

Brian S. Appleby, MD; Kristin K. Appleby, MD; Barbara J. Crain, MD, PhD; Chiadi U. Onyike, MD, MHS; Mitchell T. Wallin, MD, MPH; Peter V. Rabins, MD, MPH

Background: The classic Creutzfeldt-Jakob disease (CJD), Heidenhain, and Oppenheimer-Brownell variants are sporadic CJD (sCJD) phenotypes frequently described in the literature, but many cases present with neuropsychiatric symptoms, suggesting that there may be additional sCJD phenotypes.Objective: To characterize clinical, diagnostic, and molecular features of 5 sCJD variants.Design: Retrospective analysis.Setting: The Johns Hopkins and Veterans Administration health care systems.Participants: Eighty-eight patients with definite or probable sCJD.Main Outcome Measures: Differences in age at onset, illness progression, diagnostic test results, and molecular subtype.Results: The age at onset differed among sCJD variants (P=.03); the affective variant had the youngest mean age at onset (59.7 years). Survival time (P.001) and the time to clinical presentation (P=.003) differed among groups. Patients with the classic CJD phenotype had the shortest median survival time from symptom onset (66 days) and those who met criteria for the affective sCJD variant had the longest (421 days) and presented to clinicians significantly later (median time from onset to presentation, 92 days; P=.004). Cerebrospinal fluid analyses were positive for 14-3-3 protein in all of the affective variants, regardless of illness duration. Periodic sharp-wave complexes were not detected on any of the electroencephalography tracings in the Oppenheimer-Brownell group; basal ganglia hyperintensity was not detected on brain magnetic resonance imaging in this group either. All of the Heidenhain variants were of the methionine/ methionine type 1 molecular subtype.Conclusions: The classic CJD phenotype and the Heidenhain, Oppenheimer-Brownell, cognitive, and affective sCJD variants differ by age at disease onset, survival time, and diagnostic test results. Characteristics of these 5 phenotypes are provided to facilitate further clinicopathologic investigation that may lead to more reliable and timely diagnoses of sCJD.Arch Neurol. 2009;66(2):208-215

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COMMENT

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see full text ;

http://creutzfeldt-jakob-disease.blogspot.com/2009/08/characteristics-of-established-and.html

Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518

WHO WILL WATCH THE CHILDREN ?

2009-05-17T08:43:00.000-07:00

SCHOOL LUNCH PROGRAM FROM DOWNER CATTLE UPDATE

IS THERE A SCRAPIE-LIKE DISEASE IN CATTLE ?

In April of 1985, a mink rancher in Wisconsin reported a debilitating neurologic disease in his herd which we diagnosed as TME by histopathologic findings confirmed by experimental transmission to mink and squirrel monkeys. The rancher was a ''dead stock'' feeder using mostly (>95%) downer or dead dairy cattle and a few horses. She had never been fed.

We believe that these findings may indicate the presence of a previously unrecognized scrapie-like disease in cattle and wish to alert dairy practitioners to this possibility.

snip...

PROCEEDINGS OF THE SEVENTH ANNUAL WESTERN CONFERENCE FOR FOOD ANIMAL VETERINARY MEDICINE, University of Arizona, March 17-19, 1986

http://web.archive.org/web/20030331063559/http://www.bseinquiry.gov.uk/files/mb/m09a/tab01.pdf

http://web.archive.org/web/20030516051623/http://www.bseinquiry.gov.uk/files/mb/m09/tab05.pdf

IS THERE A SCRAPIE-LIKE DISEASE IN CATTLE ?

YOU BET THERE IS, AND HAS BEEN, AND WE BEEN FEEDING THE MOST HIGH RISK I.E. DEAD STOCK DOWNER COWS TO OUR CHILDREN FOR DECADES, who will follow these children for human TSE from mad cow disease here in the USA in the years, decades to come, and how many will they expose from the 'pass it forward' friendly fire modes ???

http://downercattle.blogspot.com/2008/12/evaluation-of-fsis-management-controls.html

http://downercattle.blogspot.com/

Saturday, March 14, 2009 Agriculture Secretary Tom Vilsack Announces Final Rule for Handling of Non-Ambulatory Cattle

Release No. 0060.09 Contact: Amanda Eamich (202) 720-9113

http://downercattle.blogspot.com/2009/03/agriculture-secretary-tom-vilsack.html

FNS All Regions Affected School Food Authorities By State United States Department of Agriculture Food and Nutrition Service National School Lunch Program March 24, 2008 School Food Authorities Affected by Hallmark/Westland Meat Packing Co. Beef Recall February 2006 – February 2008

http://www.fns.usda.gov/fns/safety/Hallmark-Westland_byState.pdf

Approximately 50.3 million pounds of the beef recalled by HallmarkNVestland went to federal nutrition programs, including the National School Lunch Program, and of those 50.3 million pounds, about 19.6 million pounds had already been consumed at the time the recall was issued. Release No. 0054.08, USDA, Transcript of Technical Briefing - HallmarldWestland Meat Packing Company (Feb. 21, 2008).

9. HSUS members that consume meat products, including beef products, are concerned about eating adulterated meat products and the health risks associated with such adulterated meat. Specifically, they are concerned that downed cattle are at an increased risk for harboring and transmitting BSE prions and other pathogens. The consumption of meat products derived from BSE-infected cattle is believed to cause a human neurological disease known as variant Creutzfeldt-Jakob disease ("vCJD"). The disease is progressive, invariably fatal, and there is no known effective treatment or cure. Downed cattle may also be at higher risk for harboring other foodborne transmissible pathogens, including E. coli 0157:H7, Salmonella, and anthrax. By allowing downed cattle to enter the food supply, USDA's regulatory loophole injures members of The HSUS by placing them at an increased risk of contracting these food-borne illnesses each time they eat beef. 10. Members of The HSUS are also concerned about the meat products provided to their children through the National School Lunch Program. More than 31 million school children receive lunches through the program each school day. To assist states in providing healthful, low-cost or free meals, USDA provides states with various commodities including ground beef. As evidenced by the HallmarkNVestland investigation and recall, the potential for downed animals to make their way into the National School Lunch Program is neither speculative nor hypothetical.

http://biotech.law.lsu.edu/cases/FDA/hsus-v-schafer-usda-complaint.pdf

United States of America Ex rel. The Humane Society of the United States v. Hallmark Meat Packing Company, Westland Meat Company Inc. (Downed animal abuse/government fraud)

Court or Agency: United States District Court for the Central District of California Plaintiff(s): United States of America Ex rel. The Humane Society of the United States Defendant: Hallmark Meat Packing Company, Westland Meat Company Inc. HSUS Counsel: Peter J. Petersan, Leana Stormont Outside Counsel: Milbank, Tweed, Hadley & McCloy LLP Status: In Briefing

Federal court action under the False Claims Act alleging that Westland/Hallmark defrauded the federal government by violating the terms of its school lunch program contracts requiring the humane handling of animals. The lawsuit was the result of an investigation by The HSUS which exposed the facility's mistreatment of animals too sick or injured to walk and led to the largest meat recall in the nation’s history.

http://www.hsus.org/in_the_courts/docket/us_hallmark.html

http://www.law.gmu.edu/assets/files/academics/schedule/2009/fall/HENRYGREEN_AnimalLaw20090818.pdf

http://www.hsus.org/farm/news/ournews/dept_of_justice_hallmark_050409.html

FOIA

http://www.fsis.usda.gov/PDF/FOIA_Requests_0308.pdf

"California Firm Recalls Beef Products". USDA. February 17, 2008.

http://www.fsis.usda.gov/PDF/Recall_005-2008_Release.pdf

AP (February 17, 2008). "USDA recalls 143 million pounds of beef". MSNBC.

http://www.msnbc.msn.com/id/23212514/

"Statement by Secretary of Agriculture Ed Schafer Regarding Animal Cruelty Charges Filed Against Employees at Hallmark/Westland Meat Packing Company". USDA. February 15, 2008.

http://www.usda.gov/wps/portal/!ut/p/_s.7_0_A/7_0_1OB?contentidonly=true&contentid=2008/02/0044.xml

"USDA Q&A". USDA. February 19, 2008.

http://www.usda.gov/wps/portal/usdahome?contentidonly=true&contentid=Recall_Information.xml

The Case is captioned as United States of America ex rel. The Humane Society of the United States v. Hallmark Meat Packing Company; Westland Meat Company, Inc.

http://www.hsus.org/farm/news/ournews/dept_of_justice_hallmark_050409.html

http://www.econ.iastate.edu/classes/econ362/hallam/NewspaperArticles/DownerCows.pdf

Thursday, September 24, 2009

(09-24) 15:00 PDT LOS ANGELES (AP) --

A Southern California meatpacking plant that supplied beef to the nation's school lunch program slaughtered stumbling, potentially contaminated cows for four years before undercover video of animal abuse prompted a massive beef recall, federal court filings say...

http://downercattle.blogspot.com/2009/09/suit-meatpacker-used-downer-cows-for-4.html

also ;

http://www.usda.gov/oig/webdocs/24601-07-KC.pdf

>>>USDA officials cited three other interlocking safeguards that protect the public even if other safeguards, such as ante-mortem inspection, should fail; these safeguards are the removal of Specified Risk Materials (SRM),5 BSE surveillance testing, and the feed ban.6 Under the Federal Meat Inspection Act (FMIA),7<<<

href="http://madcowfeed.blogspot.com/2009/11/bse-feed-recall-misbranding-of-product.html">http://madcowfeed.blogspot.com/2009/11/bse-feed-recall-misbranding-of-product.html

Friday, September 4, 2009

FOIA REQUEST ON FEED RECALL PRODUCT 429,128 lbs. feed for ruminant animals may have been contaminated with prohibited material Recall # V-258-2009

http://madcowfeed.blogspot.com/2009/09/foia-request-on-feed-recall-product.html

Saturday, August 29, 2009

FOIA REQUEST FEED RECALL 2009 Product may have contained prohibited materials Bulk Whole Barley, Recall # V-256-2009 http://madcowfeed.blogspot.com/2009/08/foia-request-feed-recall-2009-product.html 2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html

"Everything they did on the Texas cow makes everything USDA did before 2005 suspect," Brown said. ...snip...end

http://www.upi.com/ConsumerHealthDaily/view.php?StoryID=20060315-055557-1284r

PAUL BROWN COMMENT TO ME ON THIS ISSUE Tuesday, September 12, 2006 11:10 AM "Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency."

http://madcowtesting.blogspot.com/

see full text sporadic CJD the big lie;

http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0705&L=sanet-mg&T=0&F=&S=&P=135027

PLUS

http://cjdmadcowbaseoct2007.blogspot.com/2008/07/novel-human-disease-with-abnormal-prion.html

Sunday, August 10, 2008

A New Prionopathy OR more of the same old BSe and sporadic CJD

http://creutzfeldt-jakob-disease.blogspot.com/2008/08/new-prionopathy-or-more-of-same-old-bse.html

HUMAN and ANIMAL TSE Classifications i.e. mad cow disease and the UKBSEnvCJD only theory

http://www.regulations.gov/search/Regs/contentStreamer?objectId=090000648027c28e&disposition=attachment&contentType=pdf

AS I SAID BEFORE, WHO WATCH THE CHILDREN FOR CJD FOR THE NEXT 5 DECADES ??? UPDATE MAD COW DISEASE NORTH AMERICA, AND THE DEADSTOCK DOWNERS THAT WERE FED TO YOUR CHILDREN

Monday, May 11, 2009 Rare BSE mutation raises concerns over risks to public health

http://bse-atypical.blogspot.com/2009/05/rare-bse-mutation-raises-concerns-over.html

Thursday, April 09, 2009

Docket No. FDA2002N0031 (formerly Docket No. 2002N0273) RIN 0910AF46 Substances Prohibited From Use in Animal Food or Feed; Final Rule: Proposed

http://madcowfeed.blogspot.com/2009/04/docket-no-fda2002n0031-formerly-docket.html

http://prionunitusaupdate2008.blogspot.com/2009/04/cjd-foundation-sides-with-r-calfers-no.html

http://prionunitusaupdate2008.blogspot.com/2009/04/r-calf-and-usa-mad-cow-problem-dont.html

Sunday, May 10, 2009

Identification and characterization of bovine spongiform encephalopathy cases diagnosed and not diagnosed in the United States

http://bse-atypical.blogspot.com/2009/05/identification-and-characterization-of.html

Thursday, March 19, 2009

MILLIONS AND MILLIONS OF POUNDS OF MAD COW FEED IN COMMERCE USA

http://madcowfeed.blogspot.com/2009/03/millions-and-millions-of-pounds-of-mad.html

Sunday, May 10, 2009

Meeting of the Transmissible Spongiform Encephalopathies Committee On June 12, 2009 (Singeltary submission)

http://tseac.blogspot.com/2009/05/meeting-of-transmissible-spongiform.html

Monday, May 4, 2009

Back to the Past With New TSE Testing Agricultural Research/May-June 2009

http://madcowtesting.blogspot.com/2009/05/back-to-past-with-new-tse-testing.html

Sunday, April 12, 2009

TRANSMISSION OF ATYPICAL BOVINE SPONGIFORM ENCEPHALOPATHY (BSE) IN HUMANIZED MOUSE MODELS

http://bse-atypical.blogspot.com/2009/04/transmission-of-atypical-bovine.html

Rare BSE mutation raises concerns over risks to public health

SIR - Atypical forms (known as H- and L-type) of bovine spongiform encephalopathy (BSE) have recently appeared in several European countries as well as in Japan, Canada and the United States. This raises the unwelcome possibility that variant Creutzfeldt-Jakob disease (vCJD) could increase in the human population. Of the atypical BSE cases tested so far, a mutation in the prion protein gene (PRNP) has been detected in just one, a cow in Alabama with BSE; her healthy calf also carried the mutation (J. A. Richt and S. M. Hall PLoS Pathog. 4, e1000156; 2008). This raises the possibility that the disease could occasionally be genetic in origin. Indeed, the report of the UK BSE Inquiry in 2000 suggested that the UK epidemic had most likely originated from such a mutation and argued against the scrapierelated assumption. Such rare potential pathogenic PRNP mutations could occur in countries at present considered to be free of BSE, such as Australia and New Zealand. So it is important to maintain strict surveillance for BSE in cattle, with rigorous enforcement of the ruminant feed ban (many countries still feed ruminant proteins to pigs). Removal of specified risk material, such as brain and spinal cord, from cattle at slaughter prevents infected material from entering the human food chain. Routine genetic screening of cattle for PRNP mutations, which is now available, could provide additional data on the risk to the public. Because the point mutation identified in the Alabama animals is identical to that responsible for the commonest type of familial (genetic) CJD in humans, it is possible that the resulting infective prion protein might cross the bovine-human species barrier more easily. Patients with vCJD continue to be identified. The fact that this is happening less often should not lead to relaxation of the controls necessary to prevent future outbreaks. Malcolm A. Ferguson-Smith Cambridge University Department of Veterinary Medicine, Madingley Road, Cambridge CB3 0ES, UK e-mail: mhtml:%7B33B38F65-8D2E-434D-8F9B-8BDCD77D3066%7Dmid://00000188/!x-usc:mailto:maf12@cam.ac.uk Jürgen A. Richt College of Veterinary Medicine, Kansas State University, K224B Mosier Hall, Manhattan, Kansas 66506-5601, USA

NATUREVol 45726 February 2009

http://www.nature.com/nature/journal/v457/n7233/full/4571079b.html

Sunday, May 10, 2009

Identification and characterization of bovine spongiform encephalopathy cases diagnosed and not diagnosed in the United States

http://bse-atypical.blogspot.com/2009/05/identification-and-characterization-of.html

Wednesday, February 11, 2009

Atypical BSE North America Update February 2009 Both of the BSE cases ascertained in the US native-born cattle were atypical cases (H-type), which contributed to the initial ambiguity of the diagnosis. 174, 185 In Canada, there have been 2 atypical BSE cases in addition to the 14 cases of the classic UK strain of BSE2: one was the H-type, and the other was of the L-type.198 snip...end source : Enhanced Abstract Journal of the American Veterinary Medical Association January 1, 2009, Vol. 234, No. 1, Pages 59-72 Bovine spongiform encephalopathy Jane L. Harman, DVM, PhD; Christopher J. Silva, PhD

http://avmajournals.avma.org/doi/ref/10.2460/javma.234.1.59

Atypical BSE North America Update February 2009

http://bse-atypical.blogspot.com/2009/02/atypical-bse-north-america-update.html

Thursday, April 30, 2009

FDA Issues Final Guidance for Renderers on Substances Prohibited From Use in Animal Food or Feed CVM Update Back April 30, 2009

http://madcowfeed.blogspot.com/2009/04/fda-issues-final-guidance-for-renderers.html

Sunday, May 10, 2009

Meeting of the Transmissible Spongiform Encephalopathies Committee On June 12, 2009 (Singeltary submission)

http://tseac.blogspot.com/2009/05/meeting-of-transmissible-spongiform.html

April 20, 2009

National Prion Disease Pathology Surveillance Center Cases Examined1 (December 31, 2008)

National Prion Disease Pathology Surveillance Center Cases Examined1 (December 31, 2008)

Year Total Referrals2 Prion Disease Sporadic Familial Iatrogenic vCJD 1996 & earlier 42 32 28 4 0 0 1997 115 68 59 9 0 0 1998 93 53 45 7 1 0 1999 115 69 61 8 0 0 2000 151 103 89 14 0 0 2001 210 118 108 9 0 0 2002 258 147 123 22 2 0 2003 273 176 135 41 0 0 2004 335 184 162 21 0 13 2005 346 193 154 38 1 0 2006 380 192 159 32 0 14 2007 370 212 185 26 0 0 2008 383 228 182 23 0 0 TOTAL 30715 17756 1490 254 4 2 1 Listed based on the year of death or, if not available, on year of referral; 2 Cases with suspected prion disease for which brain tissue and/or blood (in familial cases) were submitted; 3 Disease acquired in the United Kingdom; 4 Disease acquired in Saudi Arabia; 5 Includes 20 cases in which the diagnosis is pending, and 17 inconclusive cases; 6 Includes 25 cases with type determination pending in which the diagnosis of vCJD has been excluded. Rev 2/13/09 National

http://www.cjdsurveillance.com/pdf/case-table.pdf

http://www.cjdsurveillance.com/resources-casereport.html

http://www.aan.com/news/?event=read&article_id=4397&page=72.45.45

*5 Includes 20 cases in which the diagnosis is pending, and 17 inconclusive cases; *6 Includes 25 cases with type determination pending in which the diagnosis of vCJD has been excluded. Greetings, it would be interesting to know what year these atypical cases occurred, as opposed to lumping them in with the totals only. are they accumulating ? did they occur in one year, two years, same state, same city ? location would be very interesting ? age group ? sex ? how was it determined that nvCJD was ruled out ? from 1997, the year i started dealing with this nightmare, there were 28 cases (per this report), up until 2007 where the total was 185 cases (per this report), and to date 2008 is at 182. a staggering increase in my opinion, for something that just happens spontaneously as some would have us believe. i don't believe it, not in 85%+ of all sporadic CJD cases. actually, i do not believe yet that anyone has proven that any of the sporadic CJD cases have been proven to be a spontaneous misfolding of a protein. there are many potential routes and sources for the sporadic CJD's. ...TSS Sunday, April 12, 2009 r-calf and the USA mad cow problem, don't look, don't find, and then blame Canada

http://prionunitusaupdate2008.blogspot.com/2009/04/r-calf-and-usa-mad-cow-problem-dont.html

snip...

SEE FULL TEXT BELOW ! Monday, April 20, 2009 National Prion Disease Pathology Surveillance Center Cases Examined1 (December 31, 2008)

http://prionunitusaupdate2008.blogspot.com/2009/04/national-prion-disease-pathology.html

Friday, November 30, 2007

CJD QUESTIONNAIRE USA CWRU AND CJD FOUNDATION

snip...

i am reminded of a few things deep throat told me years ago;

=================================================2001

The most frightening thing I have read all day is the report of Gambetti's finding of a new strain of sporadic cjd in young people......... Dear God, what in the name of all that is holy is that!!! If the US has different strains of scrapie..... why???? than the UK... then would the same mechanisms that make different strains of scrapie here make different strains of BSE... if the patterns are different in sheep and mice for scrapie..... could not the BSE be different in the cattle, in the mink, in the humans....... I really think the slides or tissues and everything from these young people with the new strain of sporadic cjd should be put up to be analyzed by many, many experts in cjd........ bse..... scrapie

Scrape the damn slide and put it into mice..... wait..... chop up the mouse brain and and spinal cord........ put into some more mice..... dammit amplify the thing and start the damned research..... This is NOT rocket science... we need to use what we know and get off our butts and move.... the whining about how long everything takes..... well it takes a whole lot longer if you whine for a year and then start the research!!!

Not sure where I read this but it was a recent press release or something like that: I thought I would fall out of my chair when I read about how there was no worry about infectivity from a histopath slide or tissues because they are preserved in formic acid, or formalin or formaldehyde..... for God's sake........ Ask any pathologist in the UK what the brain tissues in the formalin looks like after a year....... it is a big fat sponge... the agent continues to eat the brain ...... you can't make slides anymore because the agent has never stopped........ and the old slides that are stained with Hemolysin and Eosin...... they get holier and holier and degenerate and continue... what you looked at 6 months ago is not there........ Gambetti better be photographing every damned thing he is looking at.....

Okay, you need to know. You don't need to pass it on as nothing will come of it and there is not a damned thing anyone can do about it. Don't even hint at it as it will be denied and laughed at.......... USDA is gonna do as little as possible until there is actually a human case in the USA of the nvcjd........ if you want to move this thing along and shake the earth.... then we gotta get the victims families to make sure whoever is doing the autopsy is credible, trustworthy, and a saint with the courage of Joan of Arc........ I am not kidding!!!! so, unless we get a human death from EXACTLY the same form with EXACTLY the same histopath lesions as seen in the UK nvcjd........ forget any action........ it is ALL gonna be sporadic!!! And, if there is a case....... there is gonna be every effort to link it to international travel, international food, etc. etc. etc. etc. etc. They will go so far as to find out if a sex partner had ever traveled to the UK/europe, etc. etc. .... It is gonna be a long, lonely, dangerous twisted journey to the truth. They have all the cards, all the money, and are willing to threaten and carry out those threats.... and this may be their biggest downfall...

Thanks as always for your help. (Recently had a very startling revelation from a rather senior person in government here.......... knocked me out of my chair........ you must keep pushing. If I was a power person.... I would be demanding that there be at least a million bovine tested as soon as possible and agressively seeking this disease. The big players are coming out of the wood work as there is money to be made!!!

In short: "FIRE AT WILL"!!! for the very dumb.... who's "will"! "Will be the burden to bare if there is any coverup!"

again it was said years ago and it should be taken seriously.... BSE will NEVER be found in the US!

As for the BSE conference call... I think you did agreat service to freedom of information and making some people feign integrity... I find it scary to see that most of the "experts" are employed by the federal government or are supported on the "teat" of federal funds. A scary picture! I hope there is a confidential panel organized by the new government to really investigate this thing.

You need to watch your back........ but keep picking at them....... like a buzzard to the bone... you just may get to the truth!!! (You probably have more support than you know. Too many people are afraid to show you or let anyone else know. I have heard a few things myself... you ask the questions that everyone else is too afraid to ask.)

================================================

greetings again voice,

then i remind everyone to read this;

'As implied in the Inset 25 we must not assume that transmission of BSE to other species will invariably present pathology typical of a scrapie-like disease.'

http://web.archive.org/web/20010305222642/www.bseinquiry.gov.uk/files/yb/1991/01/04004001.pdf

CWRU CJD QUESTIONNAIRE HISTORY

http://cjdquestionnaire.blogspot.com/

Wednesday, February 04, 2009

Creutzfeldt-Jacob disease presenting as severe depression: a case report

http://creutzfeldt-jakob-disease.blogspot.com/2009/02/creutzfeldt-jacob-disease-presenting-as.html

CJD QUESTIONNAIRE USA CWRU AND CJD FOUNDATION

http://cjdquestionnaire.blogspot.com/

WHO WILL FOLLOW THE CHILDREN FOR CJD SYMPTOMS ???

Saturday, May 2, 2009

U.S. GOVERNMENT SUES WESTLAND/HALLMARK MEAT OVER USDA CERTIFIED DEADSTOCK DOWNER COW SCHOOL LUNCH PROGRAM

http://downercattle.blogspot.com/2009/05/us-government-sues-westlandhallmark.html

Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518