Thursday, February 19, 2015

Inspections Circumvented for Condemned Cows STATEMENT OF THE HONORABLE PHYLLIS K. FONG INSPECTOR GENERAL

UNITED STATES DEPARTMENT OF AGRICULTURE OFFICE OF INSPECTOR GENERAL

 

STATEMENT OF THE HONORABLE PHYLLIS K. FONG INSPECTOR GENERAL

 

Submitted to The Subcommittee on Agriculture, Rural Development, Food and Drug Administration, and Related Agencies Committee on AppropriationsU.S. House of Representatives February 13, 2015

 

Inspections Circumvented for Condemned Cows

 

OIG has completed an investigation into a California meat processing plant that was processing diseased cattle for human consumption and avoiding regulatory inspections by the Food Safety and Inspection Service (FSIS). One of the plant’s owners indicated to the plant foreman which condemned cattle should be processed; the foreman in turn directed kill floor employees to carve “USDA Condemned” stamps out of the cattle carcasses. The owner further instructed the foreman to circumvent inspection procedures for certain cows with “cancer eye,” an illness that results in unsightly tumors on cows’ eyes and eyelids. The foreman, or another employee at his instruction, placed the heads from apparently healthy cows next to the bodies of cows with cancer eye. This switch and slaughter of uninspected cows with cancer eye occurred during the FSIS inspector’s lunch breaks, a time during which plant operations were supposed to cease.

 

As a result of this case, the owner and two employees were charged with numerous criminal acts. They have pled guilty to conspiracy under the Federal Meat Inspection Act3 and to distribution of adulterated, misbranded, and uninspected meat. FSIS suspended operations at the plant, which was subsequently sold to a new owner.

 


 

Final defendant pleads guilty in Petaluma slaughterhouse scandal 

 

Rancho Feeding Corp. slaughterhouse in 2014. (PD FILE)

 

BY PAUL PAYNE

 

THE PRESS DEMOCRAT

 

February 18, 2015, 11:27AM

 

Former Rancho Feeding owner to face trial in July   

 

2nd guilty plea in Rancho Feeding slaughterhouse scandal   

   

The co-owner of a now-defunct Petaluma slaughterhouse at the center of last year’s nationwide recall of 8.7 million pounds of beef pleaded guilty Wednesday to being part of a conspiracy to avoid inspections and distribute meat from diseased cows, including those with eye cancer.

 

Jesse “Babe” Amaral Jr., who operated Rancho Feeding Corp., became the fourth and final defendant to admit criminal wrongdoing in a scandal that rocked the custom beef world and drew attention to practices of federal meat regulators.

 

The plea, which exposes Amaral to a maximum of 28 years in prison, brings to a close a dark chapter of Sonoma County agricultural history.

 

SNIP...SEE ;

 


 

see history on this case ;

 

Tuesday, August 19, 2014

 

Former Rancho Employees Charged With 11 Felonies processed meat from 101 condemned cattle, including 79 with “cancer eye''

 


 


 

*** Because typical clinical signs of BSE cannot always be observed in nonambulatory disabled cattle, and because evidence has indicated these cattle are more likely to have BSE than apparently healthy cattle, FDA is designating material from nonambulatory disabled cattle as prohibited cattle materials.

 


 


 


 

> > > Ackerman says downed cattle are 50 times more likely to have mad cow disease (also known as Bovine Spongiform Encephalopathy, or BSE) than ambulatory cattle that are suspected of having BSE. Of the 20 confirmed cases of mad cow disease in North America since 1993, at least 16 have involved downer cattle, he said. < < <

 


 

Surveillance is more than a numbers game, Detwiler says: "It depends on the population you're testing and how good your rate of return is." The U.S. focuses on the highest-risk animals: neurologically ill and nonambulatory ("downer") cows, in which most BSE cases occur. The U.S. has about 200,000 downer cows every year, "and if you test 12,500 out of that population, you should be able to detect it at that rate of one per million," Detwiler states. Moreover, Europe has a different reason for testing. Whereas the U.S. simply wants to see if BSE has arrived, European nations know they have it and test "to pull more animals out of the food chain," she explains.

 


 


 


 

 >>> The USDA says there's no indication that the slaughtered cattle showed signs of BSE.

 

ha, ha, ha !

 

what a hoot.

 

our fine federal friends think we are all idiots. well, most of the lay public is oblivious to the science behind the TSE prion aka mad cow type disease.

 

SO, every time you here the FSIS, the USDA, the APHIS, or the FDA say that, ‘’NO REPORTED ILLNESS TO DATE’’, and or ‘’USDA says there's no indication that the slaughtered cattle showed signs of BSE’’, they are in fact calling every one of us idiots.

 

sadly, most of us go with the flow...and the industry keeps feeding us SRMs, and the government keeps telling us it’s all sporadic CJD.

 

and the world is happy $$$

 

the incubation period for the BSE CJD TSE prion is in YEARS TO DECADES. SUB-CLINICAL INFECTION IS A MAJOR RISK FACTOR !!!

 

Evidence of subclinical prion disease in aged mice following exposure to bovine spongiform encephalopathy

 

Karen L. Brown and Neil A. Mabbott

 

+ Author Affiliations The Roslin Institute and Royal (Dick) School of Veterinary Sciences, University of Edinburgh, Edinburgh, UK Correspondence Neil A. Mabbott neil.mabbott@roslin.ed.ac.uk Received 5 September 2013. Accepted 6 October 2013.

 

Abstract

 

The occurrence of variant Creutzfeldt–Jakob (vCJD) disease in humans was almost certainly the result of consumption of food contaminated with bovine spongiform encephalopathy (BSE) prions. Despite probable widespread exposure of the UK population to BSE-contaminated food in the 1980s, vCJD has been identified predominantly in young individuals, and there have been fewer cases of clinical disease than anticipated. The reasons for this are uncertain. Following peripheral exposure, many prions replicate within the lymphoid tissues before infecting the central nervous system. We have shown that the effects of host age on the microarchitecture of the spleen significantly impair susceptibility to mouse-adapted prions after peripheral exposure. The transmission of prions between different mammalian species is considered to be limited by the ‘species barrier’, which is dependent on several factors, including an intact immune system. Thus, cross-species prion transmission may be much less efficient in aged individuals. To test this hypothesis, we compared prion pathogenesis in groups of young (6–8 weeks old) and aged (600 days old) mice injected with primary BSE brain homogenate. We showed that prion pathogenesis was impaired dramatically in aged mice when compared with young animals. Whereas most young mice succumbed to clinical prion disease, all aged mice failed to develop clinical disease during their lifespans.

 

*** However, the demonstration that prion accumulation was detected in the lymphoid tissues of some aged mice after injection with primary BSE brain homogenate, in the absence of clinical signs of prion disease, has important implications for human health.

 


 

*** In some cases, the incubation period may be as long as 50 years

 


 

At a hearing in Parliament last Wednesday, the Science and Technology Committee was told that vCJD continued to pose a “significant” risk to UK public health and that more than one in every 2000 people could be silent carriers of the disease.

 

*** vCJD can have an incubation period of over 30 years.

 

Monday, February 03, 2014

 

CREUTZFELDT-JAKOB DISEASE T.S.E. PRION U.K. UPDATE As at 3rd February 2014

 


 

***In addition, non-human primates are specifically susceptible for atypical BSE as demonstrated by an approximately 50% shortened incubation time for L-type BSE as compared to C-type. Considering the current scientific information available, it cannot be assumed that these different BSE types pose the same human health risks as C-type BSE or that these risks are mitigated by the same protective measures.

 


 


 

 how many downer cattle has your child eaten via the NSLP ???

 

Thursday, November 28, 2013

 

Department of Justice Former Suppliers of Beef to National School Lunch Program Settle Allegations of Improper Practices and Mistreating Cows

 


 

seems USDA NSLP et al thought that it would be alright, to feed our children all across the USA, via the NSLP, DEAD STOCK DOWNER COWS, the most high risk cattle for mad cow type disease, and other dangerous pathogens, and they did this for 4 years, that was documented, then hid what they did by having a recall, one of the largest recalls ever, and they made this recall and masked the reason for the recall due to animal abuse (I do not condone animal abuse), not for the reason of the potential for these animals to have mad cow BSE type disease (or other dangerous and deadly pathogens). these TSE prion disease can lay dormant for 5, 10, 20 years, or longer, WHO WILL WATCH OUR CHILDREN FOR THE NEXT 5 DECADES FOR CJD ???

 

Saturday, September 21, 2013

 

Westland/Hallmark: 2008 Beef Recall A Case Study by The Food Industry Center January 2010 THE FLIM-FLAM REPORT

 


 

DID YOUR CHILD CONSUME SOME OF THESE DEAD STOCK DOWNER COWS, THE MOST HIGH RISK FOR MAD COW DISEASE ??? this recall was not for the welfare of the animals. ...tss you can check and see here ; (link now dead, does not work...tss)

 


 

try this link ;

 


 

Sunday, November 13, 2011

 

*** California BSE mad cow beef recall, QFC, CJD, and dead stock downer livestock

 


 

-------- Original Message --------

 

Subject: re-USDA's surveillance plan for BSE aka mad cow disease

 

Date: Mon, 02 May 2005 16:59:07 -0500

 

From: "Terry S. Singeltary Sr."

 

To: paffairs@oig.hhs.gov, HHSTips@oig.hhs.gov, contactOIG@hhsc.state.tx.us

 

Greetings Honorable Paul Feeney, Keith Arnold, and William Busbyet al at OIG, ...............

 

snip...

 

There will be several more emails of my research to follow. I respectfully request a full inquiry into the cover-up of TSEs in the United States of America over the past 30 years. I would be happy to testify...

 

Thank you, I am sincerely, Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518 xxx xxx xxxx

 

Date: June 14, 2005 at 1:46 pm PST In

 

Reply to: Re: Transcript Ag. Secretary Mike Johanns and Dr. John Clifford, Regarding further analysis of BSE Inconclusive Test Results posted by TSS on June 13, 2005 at 7:33 pm:

 

Secretary of Agriculture Ann M. Veneman resigns Nov 15 2004, three days later inclusive Mad Cow is announced. June 7th 2005 Bill Hawks Under Secretary for Marketing and Regulatory Programs resigns. Three days later same mad cow found in November turns out to be positive. Both resignation are unexpected. just pondering... TSS

 

MAD COW IN TEXAS NOVEMBER 2004. ...TSS

 

-------- Original Message --------

 

Director, Public Information Carla Everett ceverett@tahc.state.tx.us

 

Subject: Re: BSE 'INCONCLUSIVE' COW from TEXAS ???

 

Date: Mon, 22 Nov 2004 17:12:15 –0600

 

From: "Terry S. Singeltary Sr."

 

To: Carla Everett References: <[log in to unmask]> <[log in to unmask] us>

 

Greetings Carla,still hear a rumor;

 

Texas single beef cow not born in Canada no beef entered the food chain?

 

and i see the TEXAS department of animal health is ramping up forsomething, but they forgot a url for update?I HAVE NO ACTUAL CONFIRMATION YET...can you confirm???

 

terry

 

-------- Original Message --------

 

Subject: Re: BSE 'INCONCLUSIVE' COW from TEXAS ???

 

Date: Fri, 19 Nov 2004 11:38:21 –0600

 

From: Carla Everett

 

To: "Terry S. Singeltary Sr." References: <[log in to unmask]>

 

The USDA has made a statement, and we are referring all callers to the USDA web site. We have no information about the animal being in Texas. Carla At 09:44 AM 11/19/2004, you wrote:>Greetings Carla,>>i am getting unsubstantiated claims of this BSE 'inconclusive' cow is from>TEXAS. can you comment on this either way please?>>thank you,>Terry S. Singeltary Sr.>>

 

-------- Original Message --------

 

Subject: Re: BSE 'INCONCLUSIVE' COW from TEXAS ???

 

Date: Mon, 22 Nov 2004 18:33:20 -0600 From: Carla Everett

 

To: "Terry S. Singeltary Sr."

 

References: ...sniptss

 

our computer department was working on a place holder we could post USDA's announcement of any results. There are no results to be announced tonight by NVSL, so we are back in a waiting mode and will post the USDA announcement when we hear something. At 06:05 PM 11/22/2004,

 

you wrote:

 

>why was the announcement on your TAHC site removed?

 

>>Bovine Spongiform Encephalopathy:

 

>November 22: Press Release title here

 

>>star image More BSE information

 

>>>>terry

 

>>Carla Everett wrote:

 

>>>no confirmation on the U.S.' inconclusive test...

 

>>no confirmation on location of animal.>>>>>>

 

==========================

 

-------- Original Message --------

 

Director, Public Information Carla Everett ceverett@tahc.state.tx.us

 

Subject: Re: BSE 'INCONCLUSIVE' COW from TEXAS ???

 

Date: Mon, 22 Nov 2004 17:12:15 –0600

 

From: "Terry S. Singeltary Sr."

 

To: Carla Everett References: <[log in to unmask]> <[log in to unmask] us>

 

Greetings Carla,still hear a rumor;

 

Texas single beef cow not born in Canada no beef entered the food chain?

 

and i see the TEXAS department of animal health is ramping up forsomething, but they forgot a url for update?I HAVE NO ACTUAL CONFIRMATION YET...can you confirm???

 

terry

 

==============================

 


 


 

USDA did not test possible mad cows

 

By Steve Mitchell

 

United Press International

 

Published 6/8/2004 9:30 PM

 

WASHINGTON, June 8 (UPI) -- The U.S. Department of Agriculture claims ittested 500 cows with signs of a brain disorder for mad cow disease last year, but agency documents obtained by United Press International show the agency tested only half that number.

 


 


 

‘’These 9,200 cases were different because brain tissue samples were preserved with formalin, which makes them suitable for only one type of test--immunohistochemistry, or IHC."

 

THIS WAS DONE FOR A REASON!

 

THE IHC test has been proven to be the LEAST LIKELY to detect BSE/TSE in the bovine, and these were probably from the most high risk cattle pool, the ones the USDA et al, SHOULD have been testing. ...TSS

 

TEXAS 2ND MAD COW THAT WAS COVERED UP, AFTER AN ACT OF CONGRESS, AND CALLS FROM TSE PRION SCIENTIST AROUND THE GLOBE, THIS 2ND MAD COW IN TEXAS WAS CONFIRMED

 

THE USDA MAD COW FOLLIES POSITIVE TEST COVER UP

 

JOHANNS SECRET POSTIVE MAD COW TEST THAT WERE IGNORED

 

OIG AND THE HONORABLE FONG CONFIRMS TEXAS MAD AFTER AN ACT OF CONGRESS 7 MONTHS LATER

 

TEXAS MAD COW

 

THEY DID FINALLY TEST AFTER SITTING 7+ MONTHS ON A SHELF WHILE GW BORE THE BSE MRR POLICY, i.e. legal trading of all strains of TSE. now understand, i confirmed this case 7 months earlier to the TAHC, and then, only after i contacted the Honorable Phyllis Fong and after an act of Congress, this animal was finally confirmed ;

 

During the course of the investigation, USDA removed and tested a total of 67 animals of interest from the farm where the index animal's herd originated. All of these animals tested negative for BSE. 200 adult animals of interest were determined to have left the index farm. Of these 200, APHIS officials determined that 143 had gone to slaughter, two were found alive (one was determined not to be of interest because of its age and the other tested negative), 34 are presumed dead, one is known dead and 20 have been classified as untraceable. In addition to the adult animals, APHIS was looking for two calves born to the index animal. Due to record keeping and identification issues, APHIS had to trace 213 calves. Of these 213 calves, 208 entered feeding and slaughter channels, four are presumed to have entered feeding and slaughter channels and one calf was untraceable.

 


 

Executive Summary

 

In June 2005, an inconclusive bovine spongiform encephalopathy (BSE) sample from November 2004, that had originally been classified as negative on the immunohistochemistry test, was confirmed positive on SAF immunoblot (Western blot). The U.S. Department of Agriculture (USDA) identified the herd of origin for the index cow in Texas; that identification was confirmed by DNA analysis. USDA, in close cooperation with the Texas Animal Health Commission (TAHC), established an incident command post (ICP) and began response activities according to USDA’s BSE Response Plan of September 2004. Response personnel removed at-risk cattle and cattle of interest (COI) from the index herd, euthanized them, and tested them for BSE; all were negative. USDA and the State extensively traced all at-risk cattle and COI that left the index herd. The majority of these animals entered rendering and/or slaughter channels well before the investigation began. USDA’s response to the Texas finding was thorough and effective.

 

snip...

 

Trace Herd 3 The owner of Trace Herd 3 was identified as possibly having received an animal of interest. The herd was placed under hold order on 7/27/05. The herd inventory was conducted on 7/28/05. The animal of interest was not present within the herd, and the hold order was released on 7/28/05. The person who thought he sold the animal to the owner of Trace Herd 3 had no records and could not remember who else he might have sold the cow to. Additionally, a search of GDB for all cattle sold through the markets by that individual did not result in a match to the animal of interest. The animal of interest traced to this herd was classified as untraceable because all leads were exhausted.

 

Trace Herd 4 The owner of Trace Herd 4 was identified as having received one of the COI through an order buyer. Trace Herd 4 was placed under hold order on 7/29/05. A complete herd inventory was conducted on 8/22/05 and 8/23/05. There were 233 head of cattle that were examined individually by both State and Federal personnel for all man-made identification and brands. The animal of interest was not present within the herd. Several animals were reported to have died in the herd sometime after they arrived on the premises in April 2005. A final search of GDB records yielded no further results on the eartag of interest at either subsequent market sale or slaughter. With all leads having been exhausted, this animal of interest has been classified as untraceable. The hold order on Trace Herd 4 was released on 8/23/05.

 

Trace Herd 5 The owner of Trace Herd 5 was identified as having received two COI and was placed under hold order on 8/1/05. Trace Herd 5 is made up of 67 head of cattle in multiple pastures. During the course of the herd inventory, the owner located records that indicated that one of the COI, a known birth cohort, had been sold to Trace Herd 8 where she was subsequently found alive. Upon completion of the herd inventory, the other animal of interest was not found within the herd. A GDB search of all recorded herd tests conducted on Trace Herd 5 and all market sales by the owner failed to locate the identification tag of the animal of interest and she was subsequently classified as untraceable due to all leads having been exhausted. The hold order on Trace Herd 5 was released on 8/8/05.

 

Trace Herd 6 The owner of Trace Herd 6 was identified as possibly having received an animal of interest and was placed under hold order on 8/1/05. This herd is made up of 58 head of cattle on two pastures. A herd inventory was conducted and the animal of interest was not present within the herd. The owner of Trace Herd 6 had very limited records and was unable to provide further information on where the cow might have gone after he purchased her from the livestock market. A search of GDB for all cattle sold through the markets by that individual did not result in a match to the animal of interest. Additionally, many of the animals presented for sale by the owner of the herd had been re-tagged at the market effectually losing the traceability of the history of that animal prior to re-tagging. The animal of interest traced to this herd was classified as untraceable due to all leads having been exhausted. The hold order on Trace Herd 6 was released on 8/3/05.

 

Trace Herd 7 The owner of Trace Herd 7 was identified as having received an animal of interest and was placed under hold order on 8/1/05. Trace Herd 7 contains 487 head of cattle on multiple pastures in multiple parts of the State, including a unit kept on an island. The island location is a particularly rough place to keep cattle and the owner claimed to have lost 22 head on the island in 2004 due to liver flukes. Upon completion of the herd inventory, the animal of interest was not found present within Trace Herd 7. A GDB search of all recorded herd tests conducted on Trace Herd 7 and all market sales by the owner failed to locate the identification tag of the animal of interest. The cow was subsequently classified as untraceable. It is quite possible though that she may have died within the herd, especially if she belonged to the island unit. The hold order on Trace Herd 7 was released on 8/8/05.

 


 

THE SECRET MAD COW POSITIVE TEST, THAT WAS COVERED UP

 

Owner and Corporation Plead Guilty to Defrauding Bovine Spongiform Encephalopathy (BSE) Surveillance Program

 

An Arizona meat processing company and its owner pled guilty in February 2007 to charges of theft of Government funds, mail fraud, and wire fraud. The owner and his company defrauded the BSE Surveillance Program when they falsified BSE Surveillance Data Collection Forms and then submitted payment requests to USDA for the services. In addition to the targeted sample population (those cattle that were more than 30 months old or had other risk factors for BSE), the owner submitted to USDA, or caused to be submitted, BSE obex (brain stem) samples from healthy USDA-inspected cattle. As a result, the owner fraudulently received approximately $390,000. Sentencing is scheduled for May 2007.

 

snip...

 

4 USDA OIG SEMIANNUAL REPORT TO CONGRESS FY 2007 1st Half

 


 

PAUL BROWN COMMENT TO ME ON THIS ISSUE

 

Tuesday, September 12, 2006 11:10 AM

 

"Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency."

 

end...tss

 

Saturday, May 26, 2012

 

Are USDA assurances on mad cow case 'gross oversimplification'?

 

SNIP...

 

What irks many scientists is the USDA’s April 25 statement that the rare disease is “not generally associated with an animal consuming infected feed.”

 

The USDA’s conclusion is a “gross oversimplification,” said Dr. Paul Brown, one of the world’s experts on this type of disease who retired recently from the National Institutes of Health. "(The agency) has no foundation on which to base that statement.”

 

“We can’t say it’s not feed related,” agreed Dr. Linda Detwiler, an official with the USDA during the Clinton Administration now at Mississippi State.

 

In the May 1 email to me, USDA’s Cole backed off a bit. “No one knows the origins of atypical cases of BSE,” she said

 

The argument about feed is critical because if feed is the cause, not a spontaneous mutation, the California cow could be part of a larger outbreak.

 

SNIP...

 


 

Saturday, August 4, 2012

 

*** Final Feed Investigation Summary - California BSE Case - July 2012

 


 

in the url that follows, I have posted

 

SRM breaches first, as late as 2011.

 

then

 

MAD COW FEED BAN BREACHES AND TONNAGES OF MAD COW FEED IN COMMERCE up until 2007, when they ceased posting them.

 

then,

 

MAD COW SURVEILLANCE BREACHES.

 

Friday, May 18, 2012

 

Update from APHIS Regarding a Detection of Bovine Spongiform Encephalopathy (BSE) in the United States Friday May 18, 2012

 


 

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

 


 


 


 

Thursday, February 10, 2011

 

TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHY REPORT UPDATE CANADA FEBRUARY 2011 and how to hide mad cow disease in Canada Current as of: 2011-01-31

 


 

Friday, February 18, 2011

 

UNITED STATES OF AMERICA VS GALEN J. NIEHUES FAKED MAD COW FEED TEST ON 92 BSE INSPECTION REPORTS FOR APPROXIMATELY 100 CATTLE OPERATIONS ''PLEADS GUILTY"

 


 

Wednesday, December 22, 2010

 

Manitoba veterinarian has been fined $10,000 for falsifying certification documents for U.S. bound cattle and what about mad cow disease ?

 


 

USDA ET AL SECRET TEST THEY USE ON HOW NOT TO FIND MAD COW DISEASE IN USA

 

Tuesday, November 02, 2010

 

*** BSE - ATYPICAL LESION DISTRIBUTION (RBSE 92-21367) statutory (obex only) diagnostic criteria CVL 1992

 


 

Tuesday, February 17, 2015

 

Could we spot the next BSE?, asks BVA President

 


 

> Could we spot the next BSE?

 

we have not spotted all the cases the first time around. with Nations like the United States and Canada, organizations like the USDA, OIE, and WTO et al, it was never about ‘spotting’ all the BSE TSE prion cases, it was more about how not to find them. the triple BSE mad cow firewall, was and still is, nothing but ink on paper. ...please see facts ;

 

Saturday, February 14, 2015

 

Canadian Food Inspection Agency Confirms Bovine Spongiform Encephalopathy (BSE) in Alberta

 


 

Wednesday, February 18, 2015

 

OIE Bovine spongiform encephalopathy ,Canada

 


 

Tuesday, February 10, 2015

 

Alberta Canada First case of chronic wasting disease found in farm elk since 2002

 


 

Monday, October 10, 2011

 

EFSA Journal 2011 The European Response to BSE: A Success Story

 

snip...

 

EFSA and the European Centre for Disease Prevention and Control (ECDC) recently delivered a scientific opinion on any possible epidemiological or molecular association between TSEs in animals and humans (EFSA Panel on Biological Hazards (BIOHAZ) and ECDC, 2011). This opinion confirmed Classical BSE prions as the only TSE agents demonstrated to be zoonotic so far

 

*** but the possibility that a small proportion of human cases so far classified as "sporadic" CJD are of zoonotic origin could not be excluded.

 

*** Moreover, transmission experiments to non-human primates suggest that some TSE agents in addition to Classical BSE prions in cattle (namely L-type Atypical BSE, Classical BSE in sheep, transmissible mink encephalopathy (TME) and chronic wasting disease (CWD) agents) might have zoonotic potential.

 

snip...

 


 


 

Thursday, August 12, 2010

 

Seven main threats for the future linked to prions

 

First threat

 

The TSE road map defining the evolution of European policy for protection against prion diseases is based on a certain numbers of hypotheses some of which may turn out to be erroneous. In particular, a form of BSE (called atypical Bovine Spongiform Encephalopathy), recently identified by systematic testing in aged cattle without clinical signs, may be the origin of classical BSE and thus potentially constitute a reservoir, which may be impossible to eradicate if a sporadic origin is confirmed.

 

*** Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans.

 

*** These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.

 

Second threat

 

snip...

 


 

 

Comment from Terry Singeltary Sr.


Comment

Docket No. APHIS-2014-0107 Bovine Spongiform Encephalopathy; Importation of Animals and Animal Products Singeltary Submission ;

I believe that there is more risk to the world from Transmissible Spongiform Encephalopathy TSE prion aka mad cow type disease now, coming from the United States and all of North America, than there is risk coming to the USA and North America, from other Countries. I am NOT saying I dont think there is any risk for the BSE type TSE prion coming from other Countries, I am just saying that in 2015, why is the APHIS/USDA/FSIS/FDA still ignoring these present mad cow risk factors in North America like they are not here?

North America has more strains of TSE prion disease, in more species (excluding zoo animals in the early BSE days, and excluding the Feline TSE and or Canine TSE, because they dont look, and yes, there has been documented evidence and scientific studies, and DEFRA Hound study, that shows the canine spongiform encephalopathy is very possible, if it has not already happened, just not documented), then any other Country in the world. Mink TME, Deer Elk cervid CWD (multiple strains), cBSE cattle, atypical L-type BSE cattle, atypical H-type BSE cattle, atyical HG type BSE cow (the only cow documented in the world to date with this strain), typical sheep goat Scrapie (multiple strains), and the atypical Nor-98 Scrapie, which has been linked to sporadic CJD, Nor-98 atypical Scrapie has spread from coast to coast. sporadic CJD on the rise, with different strains mounting, victims becoming younger, with the latest nvCJD human mad cow case being documented in Texas again, this case, NOT LINKED TO EUROPEAN TRAVEL CDC.

typical BSE can propagate as nvCJD and or sporadic CJD (Collinge et al), and sporadic CJD has now been linked to atypical BSE, Scrapie and atypical Scrapie, and scientist are very concerned with CWD TSE prion in the Cervid populations. in my opinion, the BSE MRR policy, which overtook the BSE GBR risk assessments for each country, and then made BSE confirmed countries legal to trade mad cow disease, which was all brought forth AFTER that fateful day December 23, 2003, when the USA lost its gold card i.e. BSE FREE status, thats the day it all started. once the BSE MRR policy was shoved down every countries throat by USDA inc and the OIE, then the legal trading of Scrapie was validated to be a legal trading commodity, also shoved through by the USDA inc and the OIE, the world then lost 30 years of attempted eradication of the BSE TSE prion disease typical and atypical strains, and the BSE TSE Prion aka mad cow type disease was thus made a legal trading commodity, like it or not. its all about money now folks, trade, to hell with human health with a slow incubating disease, that is 100% fatal once clinical, and forget the fact of exposure, sub-clinical infection, and friendly fire there from i.e. iatrogenic TSE prion disease, the pass it forward mode of the TSE PRION aka mad cow type disease. its all going to be sporadic CJD or sporadic ffi, or sporadic gss, or now the infamous VPSPr. ...problem solved $$$

the USDA/APHIS/FSIS/FDA triple mad cow BSE firewall, well, that was nothing but ink on paper.

for this very reason I believe the BSE MRR policy is a total failure, and that this policy should be immediately withdrawn, and set back in place the BSE GBR Risk Assessments, with the BSE GBR risk assessments set up to monitor all TSE PRION disease in all species of animals, and that the BSE GBR risk assessments be made stronger than before.

lets start with the recent notice that beef from Ireland will be coming to America.

Ireland confirmed around 1655 cases of mad cow disease. with the highest year confirming about 333 cases in 2002, with numbers of BSE confirmed cases dropping from that point on, to a documentation of 1 confirmed case in 2013, to date. a drastic decrease in the feeding of cows to cows i.e. the ruminant mad cow feed ban, and the enforcement of that ban, has drastically reduced the number of BSE cases in Europe, minus a few BABs or BARBs. a far cry from the USDA FDA triple BSE firewall, which was nothing more than ink on paper, where in 2007, in one week recall alone, some 10 MILLION POUNDS OF BANNED POTENTIAL MAD COW FEED WENT OUT INTO COMMERCE IN THE USA. this is 10 years post feed ban. in my honest opinion, due to the blatant cover up of BSE TSE prion aka mad cow disease in the USA, we still have no clue as to the true number of cases of BSE mad cow disease in the USA or North America as a whole. ...just saying.

Number of reported cases of bovine spongiform encephalopathy (BSE) in farmed cattle worldwide* (excluding the United Kingdom)

Country/Year

snip...please see attached pdf file, with references of breaches in the USA triple BSE mad cow firewalls, and recent science on the TSE prion disease. ...TSS

Attachments

 (1)

Docket No. APHIS-2014-0107 Bovine Spongiform Encephalopathy; Importation of Animals and Animal Products Singeltary Submission

View Attachment:


 

 

Sunday, January 11, 2015

 

Docket No. APHIS-2014-0107 Bovine Spongiform Encephalopathy; Importation of Animals and Animal Products Singeltary Submission

 


 

Discussion: The C, L and H type BSE cases in Canada exhibit molecular characteristics similar to those described for classical and atypical BSE cases from Europe and Japan. *** This supports the theory that the importation of BSE contaminated feedstuff is the source of C-type BSE in Canada. *** It also suggests a similar cause or source for atypical BSE in these countries. ***

 

see page 176 of 201 pages...tss

 


 

*** Singeltary reply ; Molecular, Biochemical and Genetic Characteristics of BSE in Canada Singeltary reply ;

 


 

ruminant feed ban for cervids in the United States ? 31 Jan 2015 at 20:14 GMT http://www.plosone.org/annotation/listThread.action?root=85351

 

Saturday, January 24, 2015

 

Bovine Spongiform Encephalopathy: Atypical Pros and Cons

 


 

Saturday, January 31, 2015

 

RAPID ADVICE 17-2014 : Evaluation of the risk for public health of casings in countries with a “negligible risk status for BSE” and on the risk of modification of the list of specified risk materials (SRM) with regard to BSE

 


 

CANADA SEE STEADY INCREASE OF THE SPORADIC CJD’S AND THE VPSPR’S (sporadic CJD’s). ...tss

 

PLEASE NOTE, type determination pending Creutzfeldt Jakob Disease (tdpCJD) in Canada is also on a steady increase.

 

please see ;

 

> 3. Final classification of 50 cases from 2009, 2010, 2011 and 2012 is pending.

 

CJD Deaths Reported by CJDSS1, 1994-20122

 

As of May 31, 2012

 

Deaths of Definite and Probable CJD

 

Year Sporadic Iatrogenic Familial GSS FFI vCJD Total

 

1994 2 0 0 1 0 0 3

 

1995 3 0 0 0 0 0 3

 

1996 13 0 0 0 0 0 13

 

1997 16 0 1 1 0 0 18

 

1998 22 1 0 1 0 0 24

 

1999 26 2 2 1 0 0 31

 

2000 32 0 0 3 0 0 35

 

2001 27 0 2 1 0 0 30

 

2002 31 0 2 2 0 1 36

 

2003 27 1 1 0 0 0 29

 

2004 42 0 1 0 0 0 43

 

2005 42 0 0 2 0 0 44

 

2006 39 0 1 3 1 0 44

 

2007 35 0 0 4 0 0 39

 

2008 48 0 1 0 0 0 49

 

2009 48 0 3 2 0 0 53

 

2010 34 0 3 0 0 0 37

 

2011 37 0 2 1 0 1 41

 

2012 1 0 0 0 0 0 1

 

Total 525 4 19 22 1 2 573

 

1. CJDSS began in 1998

 

2. Data before 1998 are retrospective and partial, data from 1998 to 2008 are complete, and data for 2009 - 2012 are provisional

 

3. Final classification of 50 cases from 2009, 2010, 2011 and 2012 is pending.

 

CJD Deaths Reported by CJDSS1, 1994-20122

 

As of May 31, 2012

 


 

SEE DECEMBER 2012 CANADA

 


 

Subject: *** Becky Lockhart 46, Utah’s first female House speaker, dies diagnosed with the extremely rare Creutzfeldt-Jakob disease aka mad cow type disease

 

what is CJD ? just ask USDA inc., and the OIE, they are still feeding the public and the media industry fed junk science that is 30 years old.

 

why doesn’t some of you try reading the facts, instead of rubber stamping everything the USDA inc says.

 

sporadic CJD has now been linked to BSE aka mad cow disease, Scrapie, and there is much concern now for CWD and risk factor for humans.

 

My sincere condolences to the family and friends of the House Speaker Becky Lockhart. I am deeply saddened hear this.

 

with that said, with great respect, I must ask each and every one of you Politicians that are so deeply saddened to hear of this needless death of the Honorable House Speaker Becky Lockhart, really, cry me a friggen river. I am seriously going to ask you all this...I have been diplomatic for about 17 years and it has got no where. people are still dying. so, are you all stupid or what??? how many more need to die ??? how much is global trade of beef and other meat products that are not tested for the TSE prion disease, how much and how many bodies is this market worth?

 

Saturday, January 17, 2015

 

*** Becky Lockhart 46, Utah’s first female House speaker, dies diagnosed with the extremely rare Creutzfeldt-Jakob disease

 


 

Thursday, January 15, 2015

 

41-year-old Navy Commander with sporadic Creutzfeldt–Jakob disease CJD TSE Prion: Case Report

 


 


 

TSS

Thursday, March 6, 2014

TEXAS RECALL LIST MASSIVE FROM DEAD STOCK DOWNER CANCER COWS OFFAL from Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8, 2014 shipped to Texas, Florida, and Illinois UPDATE FEBRUARY 14, 2014

TEXAS RECALL LIST MASSIVE FROM DEAD STOCK DOWNER CANCER COWS

OFFAL from Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8, 2014 shipped to Texas, Florida, and Illinois UPDATE FEBRUARY 14, 2014


howdy neighbors,


 
REMEMBER, dead stock downer cows are the most high risk cattle for BSE TSE prion mad cow type disease. I suppose the CANCER is just an added bonus $$$

 
TEXAS PART IN THIS DEAD STOCK CANCER DOWNER IS MASSIVE, and some how, I knew it would be. ...a great deal in the Galveston Bay area, some right in my neighborhood, Dallas, and just about all over Texas. good luck. ...TSS
 
 
 
Thursday, February 27, 2014
 
BEEF, CANCER, PRIONS, AND OTHER DANGEROUS AND DEADLY PATHOGENS, APPARENTLY, IT'S WHAT'S FOR DINNER
 
 
 
15 Amigo 100 N. 2nd Alvin TX-Texas
 
16 Lucky Stop 5001 Fairway Alvin TX-Texas
 
17 Power Mart 1700 South St Alvin TX-Texas
 
18 Shop Get 1517 W. Sealy Alvin TX-Texas
 
19 Shop N Go 1200 W. Sealy Alvin TX-Texas
 
20 Smart Stop 2022 South St. Alvin TX-Texas
 
21 Stantons Supermarket 219 N Taylor Alvin TX-Texas
 
22 Wee Mart 2802 Fm 2912 Alvin TX-Texas 9
 
5 Baytown Market 2900 Market Bay Town TX-Texas
 
96 Express 4300 Decker Bay Town TX-Texas
 
97 Power Fuel 721 Park Bay Town TX-Texas
 
Page 4 of 27 Retailer Name Street Address City State TEXAS
 
98 Shell 4612 W. Cedar Bayou Lunchberg Bay Town TX-Texas
 
99 Texaco 2107 W. Cedar Bayou Lynchberg Bay Town TX-Texas
 
100 Texaco 6114 Decker Bay Town TX-Texas
 
101 Valero 2 E. Fayle Bay Town TX-Texas
 
102 Bayside Grocers 1599 Fm 136 Bayside TX-Texas
 
103 Circle T 1301 N. Main Baytown TX-Texas
 
104 Jc Conoco 4612 Lynchburg - Cedar Ba Baytown TX-Texas
 
105 Sams Express 8305 Hwy 146 Baytown TX-Texas
 
106 Snack Time 2401 Massy Tompkins Baytown TX-Texas
 
107 Stop N Good 9219 N. Hwy 146 Baytown TX-Texas
 
108 Wb & B Groceries 15219 I-10 E Baytown TX-Texas
 
109 Country Supermarket 8210 Beauxart Garden Beaumont TX-Texas
 
110 Market Basket 3955 Phelan Beaumont TX-Texas
 
111 Market Basket 5960 Hwy 105 Beaumont TX-Texas
 
112 Market Basket 655 Lavaca St Beaumont TX-Texas
 
113 Mercado De Familia 1650 College Beaumont TX-Texas
 
114 Target 5850 Eastex Freeway Beaumont TX-Texas
 
115 Quick Stop 7525 Hwy 105 Beaumont TX-Texas
 
142 Bernard Grocery 4127 Fm 2611 Brazoria TX-Texas
 
143 Corner Stop 409 S. Brook Brazoria TX-Texas
 
144 Stewart's Food Store 102 Hwy 332 Brazoria TX-Texas
 
145
 
*** 253 American N Oriental 2100 Avenue J Dickinson TX-Texas
 
*** 254 Chevron 750 Fm 517 W. Dickinson TX-Texas
 
*** 255 Quick Pick 902 Fm 517 Dickinson TX-Texas
 
*** 256 Shell 151 Fm 646 W. Dickinson TX-Texas
 
*** 257 Ziegler's Foods 2308 Fm 517 Rd East Dickinson TX-Texas
 
407 Big Mama Supermarket 12355 Fondren Rd Houston TX-Texas
 
408 Bravo Ranch 2877 S. Richey Houston TX-Texas
 
409 Cherry Foodmart 2405 Kelly Street Houston TX-Texas
 
410 Chevron 10001 Fuqua Houston TX-Texas
 
411 Chevron 12222 Fm 529 Houston TX-Texas
 
412 Chevron 13051 Fm 529 Houston TX-Texas
 
Page 13 of 27 Retailer Name Street Address City State TEXAS
 
413 Chevron 14647 Wood Forest Houston TX-Texas
 
414 Chevron 3306 Navigation Houston TX-Texas
 
415 Chevron 9900 Galveston Houston TX-Texas
 
416 Circle S 9122 Winfern Houston TX-Texas
 
417 Cj Food 5175 Little York Houston TX-Texas
 
418 Coastal Mart 11611 Alief Clodine Houston TX-Texas
 
419 Conoco 10403 I10 East Houston TX-Texas
 
420 Conoco 11250 Fm 529 Houston TX-Texas
 
421 Conoco 14110 Telge Rd Houston TX-Texas
 
422 Conoco 17355 Fm 529 Houston TX-Texas
 
423 Conoco 3604 Oates Houston TX-Texas
 
424 Conoco 8360 Winfern Houston TX-Texas
 
425 Diamond Food 2331 Little York Houston TX-Texas
 
426 Dollar Town 9990 Kleckley Dr Houston TX-Texas
 
427 Express 7355 Alabonson Houston TX-Texas
 
428 Exxon 1003 Studemont Houston TX-Texas
 
429 Exxon 7010 N. Hwy 146 Houston TX-Texas
 
430 Ez Mart 2233 Fm 1960 Houston TX-Texas
 
431 Family Discount NEw 8805 Homestead Houston TX-Texas
 
432 Food Fair 1420 Fm 1960 W Houston TX-Texas
 
433 Food Fair 6470 W Little York Houston TX-Texas
 
434 Food Mart 5202 Canal St Houston TX-Texas
 
435 Food Way 10949 S. Sam Houston Park Houston TX-Texas
 
436 Food World 549 Greens Road Houston TX-Texas
 
437 Foodarama 10810 S. Post Oak Houston TX-Texas
 
438 Foodarama 11021 Fuqua Houston TX-Texas
 
439 Foodarama 11502 Wilcrest Houston TX-Texas
 
440 Foodarama 15915 S. Post Oak Houston TX-Texas
 
441 Foodarama 1805 Ella Blvd Houston TX-Texas
 
442 Foodarama 4425 W Fuqua Houston TX-Texas
 
443 Foodarama 4805 Old Galvestn Rd Houston TX-Texas
 
444 Foodarama 5665 Beechnut Houston TX-Texas
 
445 Foodarama 7320 Antoine Houston TX-Texas
 
446 Foodland 9001 Jensen Houston TX-Texas
 
447 Fuel Depot 2134 Bingle Houston TX-Texas
 
Page 14 of 27 Retailer Name Street Address City State TEXAS
 
448 Fuel Express 11402 N. Houston Rosslyn Houston TX-Texas
 
449 G Mart 10017 W. Montgomery Houston TX-Texas
 
450 Get N Go 7051 Monroe Houston TX-Texas
 
451 Gs Intl Embassy 6 7405 Cayuga St. Houston TX-Texas
 
452 Handi Stop 3543 Oak Forest Houston TX-Texas
 
453 Houston Baptist 7490 Beechnut Houston TX-Texas
 
454 King Point 10640 King Point Houston TX-Texas
 
455 Lees Mart 7901 Long Point Houston TX-Texas
 
456 Los Paisanos 4402 Sherwood Ln Houston TX-Texas
 
457 Lucky Food 2920 White Oak Houston TX-Texas
 
458 Lyons Super Market 3317 Lyons Houston TX-Texas
 
459 Metro 869 Dairy Ashford Houston TX-Texas
 
460 On The Way 6120 N, Fry Houston TX-Texas
 
461 Orbits Convenience Store 1501 Bay Area Blvd Houston TX-Texas
 
462 Phillips 66 15632 West Park Houston TX-Texas
 
463 Phillips 66 7028 Lawndale Houston TX-Texas
 
464 Pig & Save 7710 W. Little York Houston TX-Texas
 
465 Pinemont 7700 Pinemont Houston TX-Texas
 
466 Pop's Supermarket 6371 Windswept Lane Houston TX-Texas
 
467 Pyburns 12675 Fondren Rd Houston TX-Texas
 
468 Pyburns 13824 Almeda Houston TX-Texas
 
469 Quick Mart 252 E.Crosstimber Houston TX-Texas
 
470 Raceway 12411 Kuykendahl Houston TX-Texas
 
471 Rancho Grande 2598 A Fm 1960 E Houston TX-Texas
 
472 Sav A Step 13030 Wood Forest Houston TX-Texas
 
473 Sellers 1050 Federal Rd Houston TX-Texas
 
474 Sellers 10901 Market St Rd Houston TX-Texas
 
475 Sellers 1202 Uvalde Houston TX-Texas
 
476 Sellers 1523 Little York Houston TX-Texas
 
477 Sellers 3337 Telephone Houston TX-Texas
 
478 Sellers 402 Edgebrook Houston TX-Texas
 
479 Sellers 5900 Renwick Houston TX-Texas
 
480 Sellers 601 N. Cesar Chavez Houston TX-Texas
 
481 Sellers 8011 Elvera Houston TX-Texas
 
482 Sellers 8620 Stella Link Houston TX-Texas
 
Page 15 of 27 Retailer Name Street Address City State TEXAS
 
483 Sellers 9494 Hammerly Houston TX-Texas
 
484 Shell 11967 Aldine Westfield Houston TX-Texas
 
485 Shell 11997 Main Houston TX-Texas
 
486 Shell 1300 League Line Houston TX-Texas
 
487 Shell 1315 Blalock Houston TX-Texas
 
488 Shell 1827 Riley Fuzzell Houston TX-Texas
 
489 Shell 5602 Southwest Fwy Houston TX-Texas
 
490 Shell 8108 East Fwy Houston TX-Texas
 
491 Shell 9875 N . Houston Rosslyn Houston TX-Texas
 
492 Sing On Supermarket 3905 Cavalcade Houston TX-Texas
 
493 Sky Gas 15405 Tomball Houston TX-Texas
 
494 Sol Supermarket 7200 Lyons Houston TX-Texas
 
495 Speedex 211 Crosstimber Houston TX-Texas
 
496 Speedy Mart 1708 N. Wayside Houston TX-Texas
 
497 Step N Go 8602 Richmond Houston TX-Texas
 
498 Stop N Buy 7228 W. Gulf Bank Houston TX-Texas
 
499 Stop N Joy 10304 Harwin Houston TX-Texas
 
500 Sunmart 10910 Airline Houston TX-Texas
 
501 Sunny 10240 Bauman Houston TX-Texas
 
502 Sunrise 1301 Federal Houston TX-Texas
 
503 Super K 1326 Dairy Ashford Houston TX-Texas
 
504 Texaco 11602 N. Houston Rosslyn Houston TX-Texas
 
505 Texaco 9343 Clay Houston TX-Texas
 
506 Tip Top 15211 Wood Forest Houston TX-Texas
 
507 Ts All Season 620 Little York Houston TX-Texas
 
508 Twee's Food 3401 Holman St Houston TX-Texas
 
509 Valero 10201 Veterans Memorial Houston TX-Texas
 
510 Valero 9299 Richmond Houston TX-Texas
 
511 Watkins 10510 Cullen Houston TX-Texas
 
512 West Wind 13630 Richmond Houston TX-Texas
 
513 Zips 9922 North Freeway Houston TX-Texas
 
514 529 Market 13051 Fm 529 Road Houston TX-Texas
 
515 Citgo 14929 Tomball Parkway Houston TX-Texas
 
516 Jacks Grocery 5798 N Sam Houston Pkwy E Houston TX-Texas
 
517 Speedy Stop 2302 Northpark Drive Houston TX-Texas
 
Page 16 of 27 Retailer Name Street Address City State TEXAS
 
518 Speedy Stop 4502 Fm 1960 W Houston TX-Texas
 
519 El Ahorro 11132 Aldine Westfield Houston TX-Texas
 
520 El Ahorro 3107 Blalock Houston TX-Texas
 
521 Food City 5230 Aldine Mail Rd Houston TX-Texas
 
526 Exxon 11105 Fm 1960 Humble TX-Texas
 
527 Gas & More 301 St Humble TX-Texas
 
528 Super K 5103 Fm 1960 Humble TX-Texas
 
529 1960 Market 7807 Fm 1960 Bypass Rd W Humble TX-Texas
 
530 Wilson Mart 3103 Wilson Road Humble TX-Texas
 
*** 586 Corner Food 2402 W. Main League City TX-Texas
 
*** 804 Arlan's 4614 E Nasa Rd 1 Seabrook TX-Texas
 
*** 805 One Stop 4024 Nasa Pkwy Seabrook TX-Texas
 
*** 806 Red's Stop 4826 Todville Seabrook TX-Texas
 
*** 807 Super Save 1714 2nd Seabrook TX-Texas
 
*** 859 Amburn Food 8150 Fm 1764 W Texas City TX-Texas
 
*** 860 Citgo 5904 Texas Ave Texas City TX-Texas
 
*** 861 Food King 915 6th St N Texas City TX-Texas
 
*** 862 Food Rite 5320 Fm 1765 Texas City TX-Texas
 
*** 863 M & M Food 1830 25th Ave N. Texas City TX-Texas
 
*** 864 Mainland Express 8500 Fm 1764 W Texas City TX-Texas
 
*** 865 Shop In Drive 2717 25th Ave N Texas City TX-Texas
 
*** 866 Step In 1321 Texas Ave Texas City TX-Texas
 
*** 867 Tiger Express 1025 5 th Street Ave. North Texas City TX-Texas
 
Page 26 of 27 Retailer Name Street Address City State TEXAS
 
*** 868 Timewise 402 Hwy 146 Texas City TX-Texas
 
snip... see more of the stores in Texas and the USA and other countries that received these dead stock downer cancer cows, the most high risk cattle for BSE TSE prion disease aka mad cow disease and CANCER. ...
 
 
 
 
Sent: Friday, February 14, 2014 5:11 PM
Subject: [BSE-L] OFFAL from Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8, 2014 shipped to Texas, Florida, and Illinois UPDATE FEBRUARY 14, 2014
 
OFFAL from Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8, 2014 shipped to Texas, Florida, and Illinois UPDATE FEBRUARY 14, 2014
 
UPDATE !
 
they are still working on the retail list for the out of state recipients of this recall. ...see ;
 
kind regards, terry
 
 
Sent: Wednesday, February 12, 2014 2:24 PM
Subject: RE: OFFAL from Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8, 2014 shipped to Texas, Florida, and Illinois
 
Dear  Mr. Singletary :
 
Thank you for writing back.
 
Offal is another word for organ meats or parts of the animal other than meat. In this case Offal would include the cheeks, tripa, lips, sweetbreads, tripe and other parts of the animal. 20-60lb boxes of these parts were shipped to distributers and retailers in California, Texas, Illinois and Florida. At this time we only have the Retail Distribution List for California but we are working on the lists for the other states.
 
Sincerely,

Meat and Poultry Hotline Webmaster
To speak to a Food Safety Specialist,
call the Hotline at 1-888-674-6854
e-mail:
mphotline@fsis.usda.gov
Food safety information is also available 24/7, by going to “Ask Karen,” our automated virtual representative at www.askkaren.gov. You may type your food safety question directly into the automated virtual representative feature.
 
cid:image012.jpg@01CD22D8.09FABCC0
Check your steps at http://www.foodsafety.gov/keep/
 
From: Terry S. Singeltary Sr. [mailto:flounder9@verizon.net]
Sent: Wednesday, February 12, 2014 3:07 PM
To: Meat and Poultry Hotline - FSIS
Subject: Re: OFFAL from Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8, 2014 shipped to Texas, Florida, and Illinois
 
greetings again Meat and Poultry Hotline,
 
thank you for your reply.
 
however, you did not answer my question.
 
what about the OFFAL that went to Texas, Illinois, Florida, and in California ???
 
the offal from Rancho Feeding Corporation products that went to Texas, Florida, Illinois, and California, are NOT subject to recall ???
 
it will all be fed out ???
 
is this what you are saying ???
 
my letter specifically ask ;
 
I am trying to locate the recalls of the OFFAL that was included in this recall, per ;
 
 
The recall affects all beef processed at Rancho between Jan. 1, 2013 and Jan. 7, 2014, a USDA spokesman said. The carcasses and other parts, commonly referred to as offal, were shipped to retailers and distributors in California, Florida, Illinois and Texas.
 
 
 
 
 
THIS information was not listed anywhere in either of the recalls ???
 
was it used in any feed of any kind, including pet food ???
 
if not, where was the offal intended, and was all of it recalled ???
 
where may I find this information about the recall of the offal, that went to Texas, Florida, Illinois, and California ???
 
 
thank you,
 
kind regards,
terry
 
 
Sent: Wednesday, February 12, 2014 1:55 PM
Subject: RE: OFFAL from Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8, 2014 shipped to Texas, Florida, and Illinois
 
Dear   Mr. Singeltary :
 
Thank you for writing the USDA's Meat and Poultry Hotline.
 
The following Rancho Feeding Corporation products are subject to recall:
  • "Beef Carcasses” (wholesale and custom sales only)
snip...end...tss
 
 
*** LATEST UPDATE ON RECALL FOR OUT OF STATE RECIPIENTS ;
 
 
RETAIL CONSIGNEES FOR FSIS RECALL 013-2014
 
FSIS has reason to believe that the following retail location(s) received meat products that have been recalled by Rancho Feeding Corporation. This list may not include all retail locations that have received the recalled product or may include retail locations that did not actually receive the recalled product. Therefore, it is important that you use the product-specific identification information, available at http://www.fsis.usda.gov/wps/portal/fsis/topics/recalls-and-public-health-alerts/recall-case-archive/archive/2014/recall-013-2014-release in addition to this list of retail stores, to check meat products in your possession to see if they have been recalled.
 
 
Store list begins on page 2.
 
Page 1 of 1
 
Retail List for Recall Number:
 
013-2014, Various beef products
 
List Current As Of:
 
13-Feb-14
 
Retailer Name
 
Location
 
1 Pastoral Plate Pick-up locations in San Francisco Bay area
 
Nationwide, State-Wide, or Area-Wide Distribution
 
Page 1 of 2
 
Retail List for Recall Number:
 
013-2014, Various beef products
 
List Current As Of:
 
13-Feb-14
 
Retailer Name Street Address City State
 
1 La Corona Market 805 Washington St. Calistoga CA-California
 
2 North Coast Opportunities 14832 Lakeshore Drive Clearlake CA-California
 
3 Long Meats 81 E 28th Eugene CA-California
 
4 Food for Thought 6550 Railroad Ave Forestville CA-California
 
5 Fulton Pentecostal Church 3380 Fulton Rd Fulton CA-California
 
6 River Of Life Church 15035 Armstrong Woods Rd. Guerneville CA-California
 
7 Woodland Meats 735 College Ave Kentfield CA-California
 
8 Community Care 8050 A Lake Street Lower Lake CA-California
 
9 Brown's Valley Market 3263 Browns Valley Rd. Napa CA-California
 
10 La Morenita 2436 Jefferson Napa CA-California
 
11 Vallergas Market 2139 1st St. Napa CA-California
 
12 Apple Market 155 San Marin Dr. Novato CA-California
 
13 Los Mexicanos 1244 High St. Oakland CA-California
 
14 Los Primos 4095 Foothill Blvd. Oakland CA-California
 
15 Mi Ranchito 3326 Foothill Blvd. Oakland CA-California
 
16 Buds Meats 7750 Petaluma Hill Rd. Penngrove CA-California
 
17 G & G 701 Sonoma Mtn. Pkwy Petaluma CA-California
 
18 Petaluma Mkt. 210 Western Ave. Petaluma CA-California 19 Gartners 7450 Killingsworth St. Portland CA-California
 
20 Calvary Fellowship of RP 6800 Hunter Ave Suite A Rohnert Park CA-California
 
21 St Elizabeth Seton 4595 Synder Lane Rohnert Park CA-California
 
22 La Luna Market 1153 Rutherford Rd. Rutherford CA-California
 
23 Sunny Hills Services 300 Sunny Hills Dr San Anselmo CA-California
 
24 Drews Bros. Meats 1706 Church St San Francisco CA-California
 
25 La Esmeralda 1330 Market St. San Pablo CA-California
 
26 Azteca Market 802 4th St. San Rafael CA-California
 
27 Carniceria Coalcoman 1415 Maple Ave. Santa Rosa CA-California CALIFORNIA Page 2 of 2 Retailer Name Street Address City State CALIFORNIA
 
28 Carniceria Contreras 1401 Todd Rd. Santa Rosa CA-California
 
29 Carolina Wild 5380 Aero Dr. Santa Rosa CA-California
 
30 Extended Child Care 1745 Copperhill Pkwy, Suite 5 Santa Rosa CA-California
 
31 G & G 1211 W. College Ave. Santa Rosa CA-California
 
32 Greater Power House 777 Hearn Ave Santa Rosa CA-California
 
33 Vertical Call 912 DeTurk Ave Santa Rosa CA-California
 
34 Willowside Meats 3421 Guernville Rd. Santa Rosa CA-California
 
35 California Human Development Corporation 3315 Airway Dr Santa Rosa CA-California
 
36 Christ Tabernacle 1363 Fulton Rd Santa Rosa CA-California
 
37 Christian Family Fellowship 1160 A Hopper Street Santa Rosa CA-California
 
38 Kid Street Center Learning Center 709 Davis Street Santa Rosa CA-California
 
39 Promise Center 791 Lombardi Court Santa Rosa CA-California
 
40 Redwood Empire Food Bank 3990 Brickway Blvd. Santa Rosa CA-California
 
41 Redwood Gospel Mission 101 Sixth Street Santa Rosa CA-California
 
42 Santa Rosa Apostolic Church 1236 Grand Avenue Santa Rosa CA-California
 
43 St Eugene's 2323 Montgomery Drive Santa Rosa CA-California
 
44 St Rose 398 Tenth St Santa Rosa CA-California
 
45 Han's Sausage 717 SW 148th Street, Suite B Seattle CA-California
 
46 Community & Family Service Agency 477 Petaluma Ave Sebastopol CA-California
 
47 Sonoma Market 500 W. Napa Sonoma CA-California
 
48 Azteca Market 789 Main St. St. Helena CA-California
 
49 Butcher Boys 4710 East Fourth Plain Blvd Vancouver CA-California
 
50 Windsor Service Alliance 8987 Windsor Rd Windsor CA-California
 
Page 1 of 1
 
Retail List for Recall Number: 013-2014, Various beef products
 
List Current As Of:
 
13-Feb-14
 
Florida
 
Retailer Name Street Address City State
 
1 Fajitas Meats and Fish 3921 10th Avenue North Lake Worth FL-Florida
 
 
previously this week ;
 
 
 
Tuesday, February 11, 2014
 
*** California Firm Recalls Various Meat Products Produced Without the Benefit of Full Inspection Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8, 2014
 
 
 
Wednesday, February 12, 2014
 
*** USDA/APHIS NOTICE: Final Rule Regarding Imports and BSE Effective March 4, 2014
 
 
 
Friday, February 14, 2014
 
OFFAL from Class I Recall 002-2014 and 013-2014 Health Risk: High Jan 13, 2014 and Feb 8, 2014 shipped to Texas, Florida, and Illinois UPDATE FEBRUARY 14, 2014
 
 
 
ABOUT THAT USDA INC FDA TRIPLE MAD COW FIREWALL ...HA, HA, HA, jokes you, it was nothing but ink on paper...
 
 
*** FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OIA UPDATE DECEMBER 2013 UPDATE ***
 
OAI 2012-2013
 
OAI (Official Action Indicated) when inspectors find significant objectionable conditions or practices and believe that regulatory sanctions are warranted to address the establishment’s lack of compliance with the regulation.
 
*** An example of an OAI classification would be findings of manufacturing procedures insufficient to ensure that ruminant feed is not contaminated with prohibited material.
 
Inspectors will promptly re-inspect facilities classified OAI after regulatory sanctions have been applied to determine whether the corrective actions are adequate to address the objectionable conditions.
 
ATL-DO 1035703 Newberry Feed & Farm Ctr, Inc. 2431 Vincent St. Newberry SC 29108-0714 OPR DR, FL, FR, TH HP 9/9/2013 OAI Y
 
DET-DO 1824979 Hubbard Feeds, Inc. 135 Main, P.O. Box 156 Shipshewana IN 46565-0156 OPR DR, FL, OF DP 8/29/2013 OAI Y
 
ATL-DO 3001460882 Talley Farms Feed Mill Inc 6309 Talley Rd Stanfield NC 28163-7617 OPR FL, TH NP 7/17/2013 OAI N
 
NYK-DO 3010260624 Sherry Sammons 612 Stoner Trail Rd Fonda NY 12068-5007 OPR FR, OF NP 7/16/2013 OAI Y
 
DEN-DO 3008575486 Rocky Ford Pet Foods 21693 Highway 50 East Rocky Ford CO 81067 OPR RE, TH HP 2/27/2013 OAI N
 
CHI-DO 3007091297 Rancho Cantera 2866 N Sunnyside Rd Kent IL 61044-9605 OPR FR, OF HP 11/26/2012 OAI Y
 
DEN-DO 1713202 Weld County Bi Products, Inc. 1138 N 11th Ave Greeley CO 80631-9501 OPR RE, TH HP 10/12/2012 OAI N
 
Ruminant Feed Inspections Firms Inventory (excel format)
 
 
 
PLEASE NOTE, the VAI violations were so numerous, and unorganized in dates posted, as in numerical order, you will have to sift through them for yourselves. ...tss
 
snip...see full text ;
 
Sunday, December 15, 2013
 
FDA PART 589 -- SUBSTANCES PROHIBITED FROM USE IN ANIMAL FOOD OR FEED VIOLATIONS OFFICIAL ACTION INDICATED OIA UPDATE DECEMBER 2013 UPDATE
 
 
 
ONE SUCH INCIDENT CAN CAUSE 10,000,000 MILLION POUNDS OF SUSPECT BANNED MAD COW PROTEIN GOING INTO COMMERCE TO BE FED OUT ;
 
2007
 
10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. BLOOD LACED MBM IN COMMERCE USA 2007
 
Date: March 21, 2007 at 2:27 pm PST
 
RECALLS AND FIELD CORRECTIONS: VETERINARY MEDICINES -- CLASS II
 
PRODUCT
 
Bulk cattle feed made with recalled Darling's 85% Blood Meal, Flash Dried, Recall # V-024-2007
 
CODE
 
Cattle feed delivered between 01/12/2007 and 01/26/2007
 
RECALLING FIRM/MANUFACTURER
 
Pfeiffer, Arno, Inc, Greenbush, WI. by conversation on February 5, 2007.
 
Firm initiated recall is ongoing.
 
REASON
 
Blood meal used to make cattle feed was recalled because it was cross- contaminated with prohibited bovine meat and bone meal that had been manufactured on common equipment and labeling did not bear cautionary BSE statement.
 
VOLUME OF PRODUCT IN COMMERCE
 
42,090 lbs.
 
DISTRIBUTION
 
WI
 
___________________________________
 
PRODUCT
 
Custom dairy premix products:
 
MNM ALL PURPOSE Pellet, HILLSIDE/CDL Prot- Buffer Meal, LEE, M.-CLOSE UP PX Pellet, HIGH DESERT/ GHC LACT Meal, TATARKA, M CUST PROT Meal, SUNRIDGE/CDL PROTEIN Blend, LOURENZO, K PVM DAIRY Meal, DOUBLE B DAIRY/GHC LAC Mineral, WEST PIONT/GHC CLOSEUP Mineral, WEST POINT/GHC LACT Meal, JENKS, J/COMPASS PROTEIN Meal, COPPINI - 8# SPECIAL DAIRY Mix, GULICK, L-LACT Meal (Bulk), TRIPLE J - PROTEIN/LACTATION, ROCK CREEK/GHC MILK Mineral, BETTENCOURT/GHC S.SIDE MK-MN, BETTENCOURT #1/GHC MILK MINR, V&C DAIRY/GHC LACT Meal, VEENSTRA, F/GHC LACT Meal, SMUTNY, A- BYPASS ML W/SMARTA, Recall # V-025-2007
 
CODE
 
The firm does not utilize a code - only shipping documentation with commodity and weights identified.
 
RECALLING FIRM/MANUFACTURER
 
Rangen, Inc, Buhl, ID, by letters on February 13 and 14, 2007. Firm initiated recall is complete.
 
REASON
 
Products manufactured from bulk feed containing blood meal that was cross contaminated with prohibited meat and bone meal and the labeling did not bear cautionary BSE statement.
 
VOLUME OF PRODUCT IN COMMERCE
 
9,997,976 lbs.
 
DISTRIBUTION
 
ID and NV
 
END OF ENFORCEMENT REPORT FOR MARCH 21, 2007
 
 
 
Saturday, August 4, 2012
 
Final Feed Investigation Summary - California BSE Case - July 2012
 
 
 
Saturday, December 15, 2012
 
Bovine spongiform encephalopathy: the effect of oral exposure dose on attack rate and incubation period in cattle -- an update 5 December 2012
 
 
 
Sunday, February 2, 2014
 
The Presence of Disease-Associated Prion Protein in Skeletal Muscle of Cattle Infected with Classical Bovine Spongiform Encephalopathy
 
NOTE Pathology
 
 
 
Thursday, February 20, 2014
 
*** Unnecessary precautions BSE MAD COW DISEASE Dr. William James FSIS VS Dr. Linda Detwiler 2014
 
 
 
WHAT about the sporadic CJD TSE proteins ?
 
WE now know that some cases of sporadic CJD are linked to atypical BSE and atypical Scrapie, so why are not MORE concerned about the sporadic CJD, and all it’s sub-types $$$
 
Creutzfeldt-Jakob Disease CJD cases rising North America updated report August 2013
 
*** Creutzfeldt-Jakob Disease CJD cases rising North America with Canada seeing an extreme increase of 48% between 2008 and 2010 ***
 
 
 
Sunday, October 13, 2013
 
*** CJD TSE Prion Disease Cases in Texas by Year, 2003-2012
 
 
 
Sunday, August 09, 2009
 
CJD...Straight talk with...James Ironside...and...Terry Singeltary... 2009
 
 
 
 
 
Friday, January 10, 2014
 
vpspr, sgss, sffi, TSE, an iatrogenic by-product of gss, ffi, familial type prion disease, what it ???
 
 
 
Sunday, January 19, 2014
 
National Prion Disease Pathology Surveillance Center Cases Examined1 as of January 8, 2014
 
 
 
Wednesday, December 11, 2013
 
*** Detection of Infectivity in Blood of Persons with Variant and Sporadic Creutzfeldt-Jakob Disease ***
 
 
 
Friday, February 14, 2014
 
Creutzfeldt-Jakob disease (CJD) biannual update (February 2014), with briefing on novel human prion disease National CJD Research and Surveillance Unit NCJDRSU
 
 
 
Monday, February 24, 2014
 
*** Sporadic Fatal Insomnia in an Adolescent
 
 
 
I suppose one of the most disturbing studies I have ever read, was the one of Gibbs et al, way back, with electrodes that caused CJD, again, and again.
 
I am not posting this to scare folks, so be it if it does, but I am posting this for you to see what you are dealing with. ...this study still amazes me. read it more than once.
 
please see ;
 
1: J Neurol Neurosurg Psychiatry 1994 Jun;57(6):757-8
 
*** Transmission of Creutzfeldt-Jakob disease to a chimpanzee by electrodes contaminated during neurosurgery.
 
Gibbs CJ Jr, Asher DM, Kobrine A, Amyx HL, Sulima MP, Gajdusek DC.
 
Laboratory of Central Nervous System Studies, National Institute of
 
Neurological Disorders and Stroke, National Institutes of Health,
 
Bethesda, MD 20892.
 
*** Stereotactic multicontact electrodes used to probe the cerebral cortex of a middle aged woman with progressive dementia were previously implicated in the accidental transmission of Creutzfeldt-Jakob disease (CJD) to two younger patients. The diagnoses of CJD have been confirmed for all three cases. More than two years after their last use in humans, after three cleanings and repeated sterilisation in ethanol and formaldehyde vapour, the electrodes were implanted in the cortex of a chimpanzee. Eighteen months later the animal became ill with CJD. This finding serves to re-emphasise the potential danger posed by reuse of instruments contaminated with the agents of spongiform encephalopathies, even after scrupulous attempts to clean them.
 
PMID: 8006664 [PubMed - indexed for MEDLINE]
 
 
 
New studies on the heat resistance of hamster-adapted scrapie agent: Threshold survival after ashing at 600°C suggests an inorganic template of replication
 
 
 
Prion Infected Meat-and-Bone Meal Is Still Infectious after Biodiesel Production
 
 
 
Detection of protease-resistant cervid prion protein in water from a CWD-endemic area
 
 
 
A Quantitative Assessment of the Amount of Prion Diverted to Category 1 Materials and Wastewater During Processing
 
 
 
Rapid assessment of bovine spongiform encephalopathy prion inactivation by heat treatment in yellow grease produced in the industrial manufacturing process of meat and bone meals
 
 
 
PPo4-4:
 
Survival and Limited Spread of TSE Infectivity after Burial
 
 
 
*** The potential impact of prion diseases on human health was greatly magnified by the recognition that interspecies transfer of BSE to humans by beef ingestion resulted in vCJD. While changes in animal feed constituents and slaughter practices appear to have curtailed vCJD, there is concern that CWD of free-ranging deer and elk in the U.S. might also cross the species barrier. Thus, consuming venison could be a source of human prion disease. Whether BSE and CWD represent interspecies scrapie transfer or are newly arisen prion diseases is unknown. Therefore, the possibility of transmission of prion disease through other food animals cannot be ruled out. There is evidence that vCJD can be transmitted through blood transfusion. There is likely a pool of unknown size of asymptomatic individuals infected with vCJD, and there may be asymptomatic individuals infected with the CWD equivalent. These circumstances represent a potential threat to blood, blood products, and plasma supplies.
 
 
 
BSE prions propagate as either variant CJD-like or sporadic CJD-like prion strains in transgenic mice expressing human prion protein
 
Variant Creutzfeldt–Jakob disease (vCJD) has been recognized to date only in individuals homozygous for methionine at PRNP codon 129. Here we show that transgenic mice expressing human PrP methionine 129, inoculated with either bovine spongiform encephalopathy (BSE) or variant CJD prions, may develop the neuropathological and molecular phenotype of vCJD, consistent with these diseases being caused by the same prion strain. Surprisingly, however, BSE transmission to these transgenic mice, in addition to producing a vCJD-like phenotype, can also result in a distinct molecular phenotype that is indistinguishable from that of sporadic CJD with PrPSc type 2. These data suggest that more than one BSE-derived prion strain might infect humans; it is therefore possible that some patients with a phenotype consistent with sporadic CJD may have a disease arising from BSE exposure.
 
 
 
EFSA Journal 2011 The European Response to BSE: A Success Story
 
snip...
 
EFSA and the European Centre for Disease Prevention and Control (ECDC) recently delivered a scientific opinion on any possible epidemiological or molecular association between TSEs in animals and humans (EFSA Panel on Biological Hazards (BIOHAZ) and ECDC, 2011).
 
This opinion confirmed Classical BSE prions as the only TSE agents demonstrated to be zoonotic so far but the possibility that ***a small proportion of human cases so far classified as "sporadic" CJD are of zoonotic origin could not be excluded. Moreover, transmission experiments to non-human primates suggest that some TSE agents in addition to Classical BSE prions in cattle (namely L-type Atypical BSE, Classical BSE in sheep, transmissible mink encephalopathy (TME) and chronic wasting disease (CWD) agents) might have zoonotic potential.
 
snip...
 
 
 
 
 
Seven main threats for the future linked to prions First threat The TSE road map defining the evolution of European policy for protection against prion diseases is based on a certain numbers of hypotheses some of which may turn out to be erroneous. In particular, a form of BSE (called atypical Bovine Spongiform Encephalopathy), recently identified by systematic testing in aged cattle without clinical signs, may be the origin of classical BSE and thus potentially constitute a reservoir, which may be impossible to eradicate if a sporadic origin is confirmed.
 
***Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.
 
Second threat
 
snip...
 
 
 
 
 
 
Friday, February 14, 2014
 
Creutzfeldt-Jakob disease (CJD) biannual update (February 2014), with briefing on novel human prion disease National CJD Research and Surveillance Unit NCJDRSU
 
 
 
Thursday, February 20, 2014
 
*** Unnecessary precautions BSE MAD COW DISEASE Dr. William James FSIS VS Dr. Linda Detwiler 2014
 
 
IN A NUT SHELL ;
 
(Adopted by the International Committee of the OIE on 23 May 2006)
 
11. Information published by the OIE is derived from appropriate declarations made by the official Veterinary Services of Member Countries. The OIE is not responsible for inaccurate publication of country disease status based on inaccurate information or changes in epidemiological status or other significant events that were not promptly reported to the Central Bureau,
 
 
 
Thursday, May 30, 2013
 
World Organization for Animal Health (OIE) has upgraded the United States' risk classification for mad cow disease to "negligible" from "controlled", and risk further exposing the globe to the TSE prion mad cow type disease
 
U.S. gets top mad-cow rating from international group and risk further exposing the globe to the TSE prion mad cow type disease
 
 
 
 
 
Tuesday, July 2, 2013
 
APHIS USDA Administrator Message to Stakeholders: Agency Vision and Goals Eliminating ALL remaining BSE barriers to export market
 
 
 
Saturday, July 6, 2013
 
Small Ruminant Nor98 Prions Share Biochemical Features with Human Gerstmann-Sträussler-Scheinker Disease and Variably Protease-Sensitive Prionopathy
 
Research Article
 
 
 
Wednesday, December 4, 2013
 
*** Bovine Spongiform Encephalopathy; Importation of Bovines and Bovine Products; Final Rule Federal Register / Vol. 78 , No. 233 /
 
Wednesday, December 4, 2013
 
 
 
Saturday, November 2, 2013
 
*** APHIS Finalizes Bovine Import Regulations in Line with International Animal Health Standards while enhancing the spread of BSE TSE prion mad cow type disease around the Globe
 
 
 
 
 
 
Gov. C.L. "Butch" Otter of Idaho signs bill that will force consumers to eat dead stock downers and whatever else the industry decides
 
BY signing this bill, Gov. C.L. Butch Otter signed, every consumer out there will loose. the industry is laughing all the way to the bank.
 
I ASSURE ALL CONSUMERS, THE INDUSTRY FLAT OUT DOES NOT CARE WHAT THEY FEED YOU, AS LONG AS IT MAKES MONEY.
 
IF not for these undercover videos, we would have never know that for 4 years, our children all across the nation, from state to state, county to county, and school to school, the NSLP via USDA inc, fed or children the most high risk cattle for mad cow type disease, and other deadly pathogens, they fed them dead stock downer cows, and then hid this 50 year nightmare (incubation for cjd up to 50 years), under the guise of a recall for animal abuse, one of the largest meat recalls in USA history was ordered. I ask again, what about child abuse ?
 
OR, recently again, by another secret video, cattle with cancer, sick and diseases cattle, from a more recent recall. cattle with cancer of the eyes, heads chopped off, no problem, no cancer no more, put them in commerce, and feed them out.
 
 
Thursday, February 27, 2014
 
BEEF, CANCER, PRIONS, AND OTHER DANGEROUS AND DEADLY PATHOGENS, APPARENTLY, IT'S WHAT'S FOR DINNER
 
snip...
 
 
Monday, March 3, 2014
 
Gov. C.L. "Butch" Otter of Idaho signs bill that will force consumers to eat dead stock downers and whatever else the industry decides
 
 
 
 
Thursday, February 27, 2014
 
BEEF, CANCER, PRIONS, AND OTHER DANGEROUS AND DEADLY PATHOGENS, APPARENTLY, IT'S WHAT'S FOR DINNER
 
 
 
 
Wednesday, February 12, 2014
 
*** USDA/APHIS NOTICE: Final Rule Regarding Imports and BSE Effective March 4, 2014
 
 
 
 
I applaud the great efforts of the HSUS on their undercover work on this and other cases, but in my opinion, USDA ET AL are wrong on the science of BSE and risk factor there from from young animals, especially with veal (*** see BSE Inquiry CJD report on veal and BSE), and the department of justice missed the boat on the NSLP deadstock downer cow program, where for 4 years, the NSLP fed deadstock downer cows to our children all across the Nation, the most high risk cattle for mad cow disease and other deadly pathogens, then hid this fact under a recall for animal abuse...these are the sad facts as I have come to know them...who will watch our children for the next 50 years $$$
 
============================================================================================================
 
*** Individuals reported to eat veal on average at least once a year appear to be at 13 TIMES THE RISK of individuals who have never eaten veal.
 
*** There is, however, a very wide confidence interval around this estimate. There is no strong evidence that eating veal less than once per year is associated with increased risk of CJD (p = 0.51).
 
*** The association between venison eating and risk of CJD shows similar pattern, with regular venison eating associated with a 9 FOLD INCREASE IN RISK OF CJD (p = 0.04).
 
*** There is some evidence that risk of CJD INCREASES WITH INCREASING FREQUENCY OF LAMB EATING (p = 0.02). *** In conclusion, an analysis of dietary histories revealed statistical associations between various meats/animal products and INCREASED RISK OF CJD. When some account was taken of possible confounding, the association between VEAL EATING AND RISK OF CJD EMERGED AS THE STRONGEST OF THESE ASSOCIATIONS STATISTICALLY. ...
 
=============================================================================================================
 
CREUTZFELDT JAKOB DISEASE SURVEILLANCE IN THE UNITED KINGDOM THIRD ANNUAL REPORT AUGUST 1994
 
Consumption of venison and veal was much less widespread among both cases and controls. For both of these meats there was evidence of a trend with increasing frequency of consumption being associated with increasing risk of CJD. (not nvCJD, but sporadic CJD...tss) These associations were largely unchanged when attention was restricted to pairs with data obtained from relatives. ...
 
Table 9 presents the results of an analysis of these data.
 
*** There is STRONG evidence of an association between ‘’regular’’ veal eating and risk of CJD (p = .0.01).
 
*** Individuals reported to eat veal on average at least once a year appear to be at 13 TIMES THE RISK of individuals who have never eaten veal.
 
There is, however, a very wide confidence interval around this estimate. There is no strong evidence that eating veal less than once per year is associated with increased risk of CJD (p = 0.51).
 
The association between venison eating and risk of CJD shows similar pattern, with regular venison eating associated with a 9 FOLD INCREASE IN RISK OF CJD (p = 0.04).
 
There is some evidence that risk of CJD INCREASES WITH INCREASING FREQUENCY OF LAMB EATING (p = 0.02).
 
The evidence for such an association between beef eating and CJD is weaker (p = 0.14). When only controls for whom a relative was interviewed are included, this evidence becomes a little STRONGER (p = 0.08).
 
snip...
 
It was found that when veal was included in the model with another exposure, the association between veal and CJD remained statistically significant (p = < 0.05 for all exposures), while the other exposures ceased to be statistically significant (p = > 0.05).
 
snip...
 
In conclusion, an analysis of dietary histories revealed statistical associations between various meats/animal products and INCREASED RISK OF CJD. When some account was taken of possible confounding, the association between VEAL EATING AND RISK OF CJD EMERGED AS THE STRONGEST OF THESE ASSOCIATIONS STATISTICALLY. ...
 
snip...
 
In the study in the USA, a range of foodstuffs were associated with an increased risk of CJD, including liver consumption which was associated with an apparent SIX-FOLD INCREASE IN THE RISK OF CJD. By comparing the data from 3 studies in relation to this particular dietary factor, the risk of liver consumption became non-significant with an odds ratio of 1.2 (PERSONAL COMMUNICATION, PROFESSOR A. HOFMAN. ERASMUS UNIVERSITY, ROTTERDAM). (???...TSS)
 
snip...see full report ;
 
 
 
 
Thursday, October 10, 2013
 
*** CJD REPORT 1994 increased risk for consumption of veal and venison and lamb
 
 
 
Thursday, November 28, 2013
 
Department of Justice Former Suppliers of Beef to National School Lunch Program Settle Allegations of Improper Practices and Mistreating Cows
 
 
 
seems USDA NSLP et al thought that it would be alright, to feed our children all across the USA, via the NSLP, DEAD STOCK DOWNER COWS, the most high risk cattle for mad cow type disease, and other dangerous pathogens, and they did this for 4 years, that was documented, then hid what they did by having a recall, one of the largest recalls ever, and they made this recall and masked the reason for the recall due to animal abuse (I do not condone animal abuse), not for the reason of the potential for these animals to have mad cow BSE type disease (or other dangerous and deadly pathogens). these TSE prion disease can lay dormant for 5, 10, 20 years, or longer, WHO WILL WATCH OUR CHILDREN FOR THE NEXT 5 DECADES FOR CJD ???
 
Saturday, September 21, 2013
 
Westland/Hallmark: 2008 Beef Recall A Case Study by The Food Industry Center January 2010 THE FLIM-FLAM REPORT
 
 
 
DID YOUR CHILD CONSUME SOME OF THESE DEAD STOCK DOWNER COWS, THE MOST HIGH RISK FOR MAD COW DISEASE ??? this recall was not for the welfare of the animals. ...tss you can check and see here ; (link now dead, does not work...tss)
 
 
 
try this link ;
 
 
 
 
Sunday, November 13, 2011
 
*** California BSE mad cow beef recall, QFC, CJD, and dead stock downer livestock
 
 
 
Thursday, February 13, 2014
 
HSUS VS USDA ET AL BAN DOWNER CALVES FOR HUMAN CONSUMPTION (*veal) and potential BSE risk factor there from
 
 
 
*** see today ;
 
 
 
Sunday, February 2, 2014
 
The Presence of Disease-Associated Prion Protein in Skeletal Muscle of Cattle Infected with Classical Bovine Spongiform Encephalopathy
 
NOTE Pathology
 
 
 
 
*** October 2009 O.11.3
 
*** Infectivity in skeletal muscle of BASE-infected cattle
 
Silvia Suardi1, Chiara Vimercati1, Fabio Moda1, Ruggerone Margherita1, Ilaria Campagnani1, Guerino Lombardi2, Daniela Gelmetti2, Martin H. Groschup3, Anne Buschmann3, Cristina Casalone4, Maria Caramelli4, Salvatore Monaco5, Gianluigi Zanusso5, Fabrizio Tagliavini1 1Carlo Besta" Neurological Institute,Italy; 2IZS Brescia, Italy; 33FLI Insel Riems, D, Germany; 4CEA-IZS Torino, Italy; 5University of Verona, Italy
 
Background: BASE is an atypical form of bovine spongiform encephalopathy caused by a prion strain distinct from that of BSE. Upon experimental transmission to cattle, BASE induces a previously unrecognized disease phenotype marked by mental dullness and progressive atrophy of hind limb musculature. Whether affected muscles contain infectivity is unknown. This is a critical issue since the BASE strain is readily transmissible to a variety of hosts including primates, suggesting that humans may be susceptible.
 
Objectives: To investigate the distribution of infectivity in peripheral tissues of cattle experimentally infected with BASE. Methods: Groups of Tg mice expressing bovine PrP (Tgbov XV, n= 7-15/group) were inoculated both i.c. and i.p. with 10% homogenates of a variety of tissues including brain, spleen, cervical lymph node, kidney and skeletal muscle (m. longissimus dorsi) from cattle intracerebrally infected with BASE. No PrPres was detectable in the peripheral tissues used for inoculation either by immunohistochemistry or Western blot.
 
Results: Mice inoculated with BASE-brain homogenates showed clinical signs of disease with incubation and survival times of 175±15 and 207±12 days. Five out of seven mice challenged with skeletal muscle developed a similar neurological disorder, with incubation and survival times of 380±11 and 410±12 days. At present (700 days after inoculation) mice challenged with the other peripheral tissues are still healthy. The neuropathological phenotype and PrPres type of the affected mice inoculated either with brain or muscle were indistinguishable and matched those of Tgbov XV mice infected with natural BASE.
 
Discussion: Our data indicate that the skeletal muscle of cattle experimentally infected with BASE contains significant amount of infectivity, at variance with BSE-affected cattle, raising the issue of intraspecies transmission and the potential risk for humans. Experiments are in progress to assess the presence of infectivity in skeletal muscles of natural BASE.
 
 
 
 
Wednesday, April 25, 2012
 
USA MAD COW DISEASE AND CJD THERE FROM SINGELTARY ET AL 1999 – 2012
 
 
 
SUMMARY REPORT CALIFORNIA BOVINE SPONGIFORM ENCEPHALOPATHY CASE INVESTIGATION JULY 2012
 
Summary Report BSE 2012
 
Executive Summary
 
 
 
Novant Health Forsyth Medical Center Information on potential CJD exposure
 
 
 
2003 Mad Cow Scaremongers
 
Mad Cow Scaremongers by Terry S. Singeltary Sr. a review of the TSE prion agent 2003-2011
 
 
 
Re: vCJD in the USA * BSE in U.S. 15 November 1999 Terry S Singeltary, NA
 
CWD is just a small piece of a very big puzzle. I have seen while deer hunting, deer, squirrels and birds, eating from cattle feed troughs where they feed cattle, the high protein cattle by products, at least up until Aug. 4, 1997. So why would it be so hard to believe that this is how they might become infected with a TSE. Or, even by potentially infected land. It's been well documented that it could be possible, from scrapie.
 
It was proven in Oprah Winfrey's trial, that Cactus Cattle feeders, sent neurologically ill cattle, some with encephalopathy stamped on the dead slips, were picked up and sent to the renders, along with sheep carcasses.
 
 
 
U.S. Scientist should be concerned with a CJD epidemic in the U.S., as well...
 
2 January 2000 Terry S Singeltary
 
The exact same recipe for B.S.E. existed in the U.S. for years and years. In reading over the Qualitative Analysis of BSE Risk Factors-1, this is a 25 page report by the USDA:APHIS:VS. It could have been done in one page. The first page, fourth paragraph says it all;
 
"Similarities exist in the two countries usage of continuous rendering technology and the lack of usage of solvents, however, large differences still remain with other risk factors which greatly reduce the potential risk at the national level."
 
Then, the next 24 pages tries to down-play the high risks of B.S.E. in the U.S., with nothing more than the cattle to sheep ratio count, and the geographical locations of herds and flocks. That's all the evidence they can come up with, in the next 24 pages.
 
Something else I find odd, page 16;
 
"In the United Kingdom there is much concern for a specific continuous rendering technology which uses lower temperatures and accounts for 25 percent of total output. This technology was _originally_ designed and imported from the United States. However, the specific application in the production process is _believed_ to be different in the two countries."
 
A few more factors to consider, page 15;
 
"Figure 26 compares animal protein production for the two countries. The calculations are based on slaughter numbers, fallen stock estimates, and product yield coefficients. This approach is used due to variation of up to 80 percent from different reported sources. At 3.6 million tons, the United States produces 8 times more animal rendered product than the United Kingdom."
 
"The risk of introducing the BSE agent through sheep meat and bone meal is more acute in both relative and absolute terms in the United Kingdom (Figures 27 and 28). Note that sheep meat and bone meal accounts for 14 percent, or 61 thousand tons, in the United Kingdom versus 0.6 percent or 22 thousand tons in the United States. For sheep greater than 1 year, this is less than one-tenth of one percent of the United States supply."
 
"The potential risk of amplification of the BSE agent through cattle meat and bone meal is much greater in the United States where it accounts for 59 percent of total product or almost 5 times more than the total amount of rendered product in the United Kingdom."
 
Considering, it would only take _one_ scrapie infected sheep to contaminate the feed. Considering Scrapie has run rampant in the U.S. for years, as of Aug. 1999, 950 scrapie infected flocks. Also, Considering only one quarter spoonful of scrapie infected material is lethal to a cow. Considering all this, the sheep to cow ration is meaningless. As I said, it's 24 pages of B.S.e.
 
To be continued...
 
Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA
 
Competing interests: None declared
 
 
 
Letters
 
JAMA. 2001;285(6):733-734. doi: 10.1001/jama.285.6.733
 
Diagnosis and Reporting of Creutzfeldt-Jakob Disease
 
Terry S. Singeltary, Sr Bacliff, Tex
 
Since this article does not have an abstract, we have provided the first 150 words of the full text.
 
KEYWORDS: creutzfeldt-jakob disease, diagnosis. To the Editor: In their Research Letter, Dr Gibbons and colleagues1 reported that the annual US death rate due to Creutzfeldt-Jakob disease (CJD) has been stable since 1985. These estimates, however, are based only on reported cases, and do not include misdiagnosed or preclinical cases. It seems to me that misdiagnosis alone would drastically change these figures. An unknown number of persons with a diagnosis of Alzheimer disease in fact may have CJD, although only a small number of these patients receive the postmortem examination necessary to make this diagnosis. Furthermore, only a few states have made CJD reportable. Human and animal transmissible spongiform encephalopathies should be reportable nationwide and internationally.
 
References 1. Gibbons RV, Holman RC, Belay ED, Schonberger LB. Creutzfeldt-Jakob disease in the United States: 1979-1998. JAMA. 2000;284:2322-2323.
 
 
 
Published March 26, 2003
 
RE-Monitoring the occurrence of emerging forms of Creutzfeldt-Jakob disease in the United States
 
Terry S. Singeltary, retired (medically)
 
I lost my mother to hvCJD (Heidenhain Variant CJD). I would like to comment on the CDC's attempts to monitor the occurrence of emerging forms of CJD. Asante, Collinge et al [1] have reported that BSE transmission to the 129-methionine genotype can lead to an alternate phenotype that is indistinguishable from type 2 PrPSc, the commonest sporadic CJD. However, CJD and all human TSEs are not reportable nationally. CJD and all human TSEs must be made reportable in every state and internationally. I hope that the CDC does not continue to expect us to still believe that the 85%+ of all CJD cases which are sporadic are all spontaneous, without route/source. We have many TSEs in the USA in both animal and man. CWD in deer/elk is spreading rapidly and CWD does transmit to mink, ferret, cattle, and squirrel monkey by intracerebral inoculation. With the known incubation periods in other TSEs, oral transmission studies of CWD may take much longer. Every victim/family of CJD/TSEs should be asked about route and source of this agent. To prolong this will only spread the agent and needlessly expose others. In light of the findings of Asante and Collinge et al, there should be drastic measures to safeguard the medical and surgical arena from sporadic CJDs and all human TSEs. I only ponder how many sporadic CJDs in the USA are type 2 PrPSc?
 
Published March 26, 2003
 
 
 
14th ICID International Scientific Exchange Brochure - Final Abstract Number: ISE.114
 
Session: International Scientific Exchange
 
Transmissible Spongiform encephalopathy (TSE) animal and human TSE in North America update October 2009
 
T. Singeltary Bacliff, TX, USA
 
Background: An update on atypical BSE and other TSE in North America. Please remember, the typical U.K. c-BSE, the atypical l-BSE (BASE), and h-BSE have all been documented in North America, along with the typical scrapie's, and atypical Nor-98 Scrapie, and to date, 2 different strains of CWD, and also TME. All these TSE in different species have been rendered and fed to food producing animals for humans and animals in North America (TSE in cats and dogs ?), and that the trading of these TSEs via animals and products via the USA and Canada has been immense over the years, decades.
 
Methods: 12 years independent research of available data
 
Results: I propose that the current diagnostic criteria for human TSEs only enhances and helps the spreading of human TSE from the continued belief of the UKBSEnvCJD only theory in 2009. With all the science to date refuting it, to continue to validate this old myth, will only spread this TSE agent through a multitude of potential routes and sources i.e. consumption, medical i.e., surgical, blood, dental, endoscopy, optical, nutritional supplements, cosmetics etc.
 
Conclusion: I would like to submit a review of past CJD surveillance in the USA, and the urgent need to make all human TSE in the USA a reportable disease, in every state, of every age group, and to make this mandatory immediately without further delay. The ramifications of not doing so will only allow this agent to spread further in the medical, dental, surgical arena's. Restricting the reporting of CJD and or any human TSE is NOT scientific. Iatrogenic CJD knows NO age group, TSE knows no boundaries. I propose as with Aguzzi, Asante, Collinge, Caughey, Deslys, Dormont, Gibbs, Gajdusek, Ironside, Manuelidis, Marsh, et al and many more, that the world of TSE Transmissible Spongiform Encephalopathy is far from an exact science, but there is enough proven science to date that this myth should be put to rest once and for all, and that we move forward with a new classification for human and animal TSE that would properly identify the infected species, the source species, and then the route.
 
 
 
The Lancet Infectious Diseases, Volume 3, Issue 8, Page 463, August 2003 doi:10.1016/S1473-3099(03)00715-1Cite or Link Using DOI
 
Tracking spongiform encephalopathies in North America
 
Original
 
Xavier Bosch
 
“My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my mom to hvCJD (Heidenhain variant CJD) and have been searching for answers ever since. What I have found is that we have not been told the truth. CWD in deer and elk is a small portion of a much bigger problem.” 49-year—old Singeltary is one of a number of people who have remained largely unsatisfied after being told that a close relative died from a rapidly progressive dementia compatible with spontaneous Creutzfeldt—Jakob ...
 
 
 
 
 
SEE FULL TEXT ;
 
-------- Original Message --------
 
Subject: Tracking spongiform encephalopathies in North America LANCET INFECTIOUS DISEASE Volume 3, Number 8 01 August 2003
 
Date: Tue, 29 Jul 2003 17:35:30 –0500
 
From: "Terry S. Singeltary Sr." Reply-To: Bovine Spongiform Encephalopathy
 
To: BSE-L@uni-karlsruhe.de
 
Volume 3, Number 8 01 August 2003
 
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Tracking spongiform encephalopathies in North America
 
Xavier Bosch
 
My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my mom to hvCJD (Heidenhain variant CJD) and have been searching for answers ever since. What I have found is that we have not been told the truth. CWD in deer and elk is a small portion of a much bigger problem.
 
49-year-old Singeltary is one of a number of people who have remained largely unsatisfied after being told that a close relative died from a rapidly progressive dementia compatible with spontaneous Creutzfeldt-Jakob disease (CJD). So he decided to gather hundreds of documents on transmissible spongiform encephalopathies (TSE) and realised that if Britons could get variant CJD from bovine spongiform encephalopathy (BSE), Americans might get a similar disorder from chronic wasting disease (CWD)the relative of mad cow disease seen among deer and elk in the USA. Although his feverish search did not lead him to the smoking gun linking CWD to a similar disease in North American people, it did uncover a largely disappointing situation.
 
Singeltary was greatly demoralised at the few attempts to monitor the occurrence of CJD and CWD in the USA. Only a few states have made CJD reportable. Human and animal TSEs should be reportable nationwide and internationally, he complained in a letter to the Journal of the American Medical Association (JAMA 2003; 285: 733). I hope that the CDC does not continue to expect us to still believe that the 85% plus of all CJD cases which are sporadic are all spontaneous, without route or source.
 
Until recently, CWD was thought to be confined to the wild in a small region in Colorado. But since early 2002, it has been reported in other areas, including Wisconsin, South Dakota, and the Canadian province of Saskatchewan. Indeed, the occurrence of CWD in states that were not endemic previously increased concern about a widespread outbreak and possible transmission to people and cattle.
 
To date, experimental studies have proven that the CWD agent can be transmitted to cattle by intracerebral inoculation and that it can cross the mucous membranes of the digestive tract to initiate infection in lymphoid tissue before invasion of the central nervous system. Yet the plausibility of CWD spreading to people has remained elusive.
 
Part of the problem seems to stem from the US surveillance system. CJD is only reported in those areas known to be endemic foci of CWD. Moreover, US authorities have been criticised for not having performed enough prionic tests in farm deer and elk.
 
Although in November last year the US Food and Drug Administration issued a directive to state public-health and agriculture officials prohibiting material from CWD-positive animals from being used as an ingredient in feed for any animal species, epidemiological control and research in the USA has been quite different from the situation in the UK and Europe regarding BSE.
 
Getting data on TSEs in the USA from the government is like pulling teeth, Singeltary argues. You get it when they want you to have it, and only what they want you to have.
 
Norman Foster, director of the Cognitive Disorders Clinic at the University of Michigan (Ann Arbor, MI, USA), says that current surveillance of prion disease in people in the USA is inadequate to detect whether CWD is occurring in human beings; adding that, the cases that we know about are reassuring, because they do not suggest the appearance of a new variant of CJD in the USA or atypical features in patients that might be exposed to CWD. However, until we establish a system that identifies and analyses a high proportion of suspected prion disease cases we will not know for sure. The USA should develop a system modelled on that established in the UK, he points out.
 
Ali Samii, a neurologist at Seattle VA Medical Center who recently reported the cases of three hunterstwo of whom were friendswho died from pathologically confirmed CJD, says that at present there are insufficient data to claim transmission of CWD into humans; adding that [only] by asking [the questions of venison consumption and deer/elk hunting] in every case can we collect suspect cases and look into the plausibility of transmission further. Samii argues that by making both doctors and hunters more aware of the possibility of prions spreading through eating venison, doctors treating hunters with dementia can consider a possible prion disease, and doctors treating CJD patients will know to ask whether they ate venison.
 
CDC spokesman Ermias Belay says that the CDC will not be investigating the [Samii] cases because there is no evidence that the men ate CWD-infected meat. He notes that although the likelihood of CWD jumping the species barrier to infect humans cannot be ruled out 100% and that [we] cannot be 100% sure that CWD does not exist in humans& the data seeking evidence of CWD transmission to humans have been very limited.
 
 
 
LANCET SINGELTARY ET AL CWD TSE PRION NORTH AMERICA
 
 
 
Singeltary submission to PLOS ;
 
No competing interests declared.
 
see full text ;
 
 
 
Owens, Julie
 
From: Terry S. Singeltary Sr. [flounder9@verizon.net]
 
Sent: Monday, July 24, 2006 1:09 PM
 
To: FSIS RegulationsComments
 
Subject: [Docket No. FSIS-2006-0011] FSIS Harvard Risk Assessment of Bovine Spongiform Encephalopathy (BSE) Page 1 of 98
 
 
 
FSIS, USDA, REPLY TO SINGELTARY
 
 
 
Sunday, August 09, 2009
 
CJD...Straight talk with...James Ironside...and...Terry Singeltary... 2009
 
 
 
Tuesday, August 18, 2009
 
* BSE-The Untold Story - joe gibbs and singeltary 1999 - 2009
 
 
 
 
Wednesday, May 16, 2012
 
*** Alzheimer’s disease and Transmissible Spongiform Encephalopathy prion disease, Iatrogenic, what if ?
 
Proposal ID: 29403
 
 
 
CJD QUESTIONNAIRE USA
 
 
 
 
 
CJD VOICE
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
August 5, 2001
 
 
 
 
 
layperson
 
wasted days and wasted nights
 
Terry S. Singeltary Sr. P.O. Box 42 Bacliff, Texas USA 77518 flounder9@verizon.net