Grand Jury Indicts Meat Company Owners in Nebraska
by News Desk | Sep 03, 2012
A federal grand jury in Nebraska has indicted Paul Rosberg, 61, and Kelly
Rosberg, 44, on six counts stemming from the sale of misbranded and/or
non-inspected meat and meat products to Omaha Public Schools. Both men are from
Wausa, NE.
If convicted, each man could be sentenced to 3 to 5 years in federal
prison, and fined from $10,000 to $250,000 on each of the six counts along with
requirements that any release be supervised and fees paid.
Count one charges the pair with conspiracy with the intent to defraud. The
second count charges Kelly Rosberg with selling 2,600 pounds of ground beef that
was labeled as inspected by the USDA when it was not.
Count three charges both men with selling on or about Sept. 19, 2011 ground
beef that was not inspected by USDA. In other words, the indictment is for both
the mislabeling ground beef as inspected when it was not and for actually
selling beef outside the required USDA inspection.
The fourth count against both men is for representing the 2,600 pounds of
beet was USDA inspected, Counts five and six are against Paul Rosberg for making
a false statements on or about Nov. 3, 2011 to USDA's Food Safety and Inspection
Service.
Deborah Gilg, U.S. District Attorney for Nebraska, announced the
indictments.
The pair were caught by a joint investigation of USDA's Office of Program
Evaluation, Enforcement and Review (OPEER) and the Inspector General (IG).
Information development by investigators led to the issuance of a search
warrant for Nebraska's Finest Meats, which led to the confiscation of records,
labels, equipment and other evidence in the case.
Nebraska's Finest Meats has suspended operations.
Omaha Public Schools, with about 50,000 K-12 students, are Nebraska's
largest.
© Food Safety News
Great news that the federal grand jury in Nebraska indicted these folks
that might have poisoned our children.
Good job USDA et al, after the fact, that our children, once again, were
exposed to misbranded and/or non-inspected meat and meat products, which could
include mad cow type disease i.e. typical or atypical BSE, and or other deadly
pathogens, but you got your man.
but, who will watch the children now ?
WHO WILL FOLLOW THE CHILDREN FOR CJD SYMPTOMS (aka mad cow disease) FOR THE
NEXT 50 YEARS ?
> > > Ackerman says downed cattle are 50 times more likely to have
mad cow disease (also known as Bovine Spongiform Encephalopathy, or BSE) than
ambulatory cattle that are suspected of having BSE. Of the 20 confirmed cases of
mad cow disease in North America since 1993, at least 16 have involved downer
cattle, he said. < < <
WHO WILL FOLLOW THE CHILDREN FOR CJD SYMPTOMS (aka mad cow disease) FOR THE
NEXT 50 YEARS ???
don’t kid yourself like the USDA et al do about sporadic CJD. USDA will not
tell the mothers this ;
Thursday, August 12, 2010
Seven main threats for the future linked to prions
First threat
The TSE road map defining the evolution of European policy for protection
against prion diseases is based on a certain numbers of hypotheses some of which
may turn out to be erroneous. In particular, a form of BSE (called atypical
Bovine Spongiform Encephalopathy), recently identified by systematic testing in
aged cattle without clinical signs, may be the origin of classical BSE and thus
potentially constitute a reservoir, which may be impossible to eradicate if a
sporadic origin is confirmed. ***Also, a link is suspected between atypical BSE
and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These
atypical BSE cases constitute an unforeseen first threat that could sharply
modify the European approach to prion diseases.
Second threat
snip...
EFSA Journal 2011 The European Response to BSE: A Success Story
This is an interesting editorial about the Mad Cow Disease debacle, and
it's ramifications that will continue to play out for decades to come ;
Monday, October 10, 2011
EFSA Journal 2011 The European Response to BSE: A Success Story
snip...
EFSA and the European Centre for Disease Prevention and Control (ECDC)
recently delivered a scientific opinion on any possible epidemiological or
molecular association between TSEs in animals and humans (EFSA Panel on
Biological Hazards (BIOHAZ) and ECDC, 2011). This opinion confirmed Classical
BSE prions as the only TSE agents demonstrated to be zoonotic so far but the
possibility that a small proportion of human cases so far classified as
"sporadic" CJD are of zoonotic origin could not be excluded. Moreover,
transmission experiments to non-human primates suggest that some TSE agents in
addition to Classical BSE prions in cattle (namely L-type Atypical BSE,
Classical BSE in sheep, transmissible mink encephalopathy (TME) and chronic
wasting disease (CWD) agents) might have zoonotic potential.
snip...
see follow-up here about North America BSE Mad Cow TSE prion risk factors,
and the ever emerging strains of Transmissible Spongiform Encephalopathy in many
species here in the USA, including humans ;
2010-2011
When L-type BSE was inoculated into ovine transgenic mice and Syrian
hamster the resulting molecular fingerprint had changed, either in the first or
a subsequent passage, from L-type into C-type BSE. In addition, non-human
primates are specifically susceptible for atypical BSE as demonstrated by an
approximately 50% shortened incubation time for L-type BSE as compared to
C-type. Considering the current scientific information available, it cannot be
assumed that these different BSE types pose the same human health risks as
C-type BSE or that these risks are mitigated by the same protective measures.
This study will contribute to a correct definition of specified risk
material (SRM) in atypical BSE. The incumbent of this position will develop new
and transfer existing, ultra-sensitive methods for the detection of atypical BSE
in tissue of experimentally infected cattle.
Saturday, May 26, 2012
Are USDA assurances on mad cow case 'gross oversimplification'?
SNIP...
What irks many scientists is the USDA’s April 25 statement that the rare
disease is “not generally associated with an animal consuming infected
feed.”
The USDA’s conclusion is a “gross oversimplification,” said Dr. Paul Brown,
one of the world’s experts on this type of disease who retired recently from the
National Institutes of Health. "(The agency) has no foundation on which to base
that statement.”
“We can’t say it’s not feed related,” agreed Dr. Linda Detwiler, an
official with the USDA during the Clinton Administration now at Mississippi
State.
In the May 1 email to me, USDA’s Cole backed off a bit. “No one knows the
origins of atypical cases of BSE,” she said
The argument about feed is critical because if feed is the cause, not a
spontaneous mutation, the California cow could be part of a larger outbreak.
SNIP...
==============================================
Saturday, August 4, 2012
Final Feed Investigation Summary - California BSE Case - July 2012
=============================================
SUMMARY REPORT CALIFORNIA BOVINE SPONGIFORM ENCEPHALOPATHY CASE
INVESTIGATION JULY 2012
Summary Report BSE 2012
Executive Summary
Saturday, August 4, 2012
Update from APHIS Regarding Release of the Final Report on the BSE
Epidemiological Investigation
WHO WILL FOLLOW THE CHILDREN FOR CJD SYMPTOMS (aka mad cow disease) FOR THE
NEXT 50 YEARS ???
Saturday, May 2, 2009
U.S. GOVERNMENT SUES WESTLAND/HALLMARK MEAT OVER USDA CERTIFIED DEADSTOCK
DOWNER COW SCHOOL LUNCH PROGRAM
OUR SCHOOL CHILDREN ALL ACROSS THE USA WERE FED THE MOST HIGH RISK CATTLE
FOR MAD COW DISEASE FOR 4 YEARS I.E. DEAD STOCK DOWNER CATTLE VIA THE USDA AND
THE NSLP.
WHO WILL WATCH OUR CHILDREN FOR THE NEXT 5+ DECADES ???
DID YOUR CHILD CONSUME SOME OF THESE DEAD STOCK DOWNER COWS, THE MOST HIGH
RISK FOR MAD COW DISEASE ???
you can check and see here ;
the article in question was an opinion article _written_ by Dr. Richard
Raymond former Undersecretary for Food Safety, U.S. Department of Agriculture
(2005-2008), and published on Bill Marlers Food Safety News feed.
Sunday, August 26, 2012
Detection of PrPSc in peripheral tissues of clinically affected cattle
after oral challenge with BSE
in the url that follows, I have posted
SRM breaches first, as late as 2011.
then
MAD COW FEED BAN BREACHES AND TONNAGES OF MAD COW FEED IN COMMERCE up until
2007, when they ceased posting them.
then,
MAD COW SURVEILLANCE BREACHES.
Friday, May 18, 2012
Update from APHIS Regarding a Detection of Bovine Spongiform Encephalopathy
(BSE) in the United States Friday May 18, 2012
2011 Monday, September 26, 2011
L-BSE BASE prion and atypical sporadic CJD
Tuesday, June 26, 2012
Creutzfeldt Jakob Disease Human TSE report update North America, Canada,
Mexico, and USDA PRION UNIT as of May 18, 2012
type determination pending Creutzfeldt Jakob Disease (tdpCJD), is on the
rise in Canada and the USA
Monday, July 23, 2012
The National Prion Disease Pathology Surveillance Center July 2012
after-the-fact litigation, on a slow incubating disease, one that is 100%
fatal, once clinical, is like robbing the dead, and their families.
this is why corporate America will not let science investigate sporadic CJD
strains $$$
what will history show us from a deja vu incident like what happened in the
UK with their mad cow debacle?
as the decades pass, will our children, and adults, be as lucky as the UK
victims of corporate greed were, with their genetic susceptibility and exposer
to their strain of BSE aka mad cow disease ?
OR, will North America population be more susceptible to the ever growing
list of different strains of different TSE prion disease (aka mad cow type
disease), typical and atypical strains growing, in many different species, all
of which have been fed back to food producing animals and humans in North
America, including our children, thanks to the continued disregard, and
after-the-fact findings, by the USDA et al ?
tune in 10, 20, –, 50 years from now, if were still here.
...end...tss
3. I asked the slaughterhouses workers in detail to explain the routine
slaughtering practices and was astonished to learn that sheep’s brains were
generally left inside the skull whereas cattle brains were routinely removed to
be added to our "meat products" – meat pies, pates, tinned items and stock
cubes. I therefore feared that this almost indestructible infective agent was
being swallowed by all beef eaters in the UK in large doses which would
inevitably infect genetically susceptible people.
Sunday, May 18, 2008
BSE, CJD, and Baby foods (the great debate 1999 to 2005)
deja vu
Wednesday, March 14, 2012
PINK SLIME, MRM's, BSE AKA MAD COW DISEASE, AND THE USDA NSLP
layperson
Terry S. Singeltary SR. P.O. Box 42 Bacliff, Texas USA 77518
flounder9@verizon.net
